🧑⚕No one help 😭
Please open Telegram to view this post
VIEW IN TELEGRAM
Forwarded from Yaqob Hamood
Real case :
PH : 7.49
PCO2 : 49.7
PO2 : 37.4
Na : 128
HCO3 : 36
BE: 14.5
What is your interpretation ?
PH : 7.49
PCO2 : 49.7
PO2 : 37.4
Na : 128
HCO3 : 36
BE: 14.5
What is your interpretation ?
Forwarded from Yaqob Hamood
PH : Less than 7.35
CO2 is more than 45 mmHg
HCO3 normal
وكان ال acute respiratory Acidosis تساوي uncompensated respiratory Acidosis
Next will about
Please open Telegram to view this post
VIEW IN TELEGRAM
Forwarded from Yaqob Hamood
Consider a case where the PaCO2 = 23 mm Hg, the pH = 7.54, and the HCO3 = 38 mEq/L ?
internal medicine
Real case : PH : 7.49 PCO2 : 49.7 PO2 : 37.4 Na : 128 HCO3 : 36 BE: 14.5 What is your interpretation ?
This case metabolic alkalosis
You want discussion in which part ?
Anonymous Poll
50%
Continue cardiology Topics
31%
Change to metabolic disorders as Acid base disorder
20%
Change to respiratory
Anyone interested in Acid base disorder
join to my personal channel
t.me/Yaqob_Hamood
I wish it's helpful ...
I'm still learning forever 📖
join to my personal channel
t.me/Yaqob_Hamood
I wish it's helpful ...
I'm still learning forever 📖
Telegram
Yaqob Hamood
Doctor
Forwarded from Yaqob Hamood
A 78-year-old man is brought in from home after a 3-day history of diarrhoea. His Arterial blood gas
shows:
• pH 7.27 (7.35–7.45)
• HCO3 −. 14 mmol/L (22–24)
• PCO2 28 mmol/L (35–45)
• K+ 2.6 mmol/L (3.5–5.0)
• Na+ 134 mmol/L (135–145)
• Cl− 113 mmol/L (95–105)
• Ur 14 mmol/L (2–7)
• Cr 320 mmol/L (50–100)
• lactate 1.5 mmol/L (<2.2).
Which ONE of the following options would BEST explain the clinical scenario?
A. High AG metabolic acidosis secondary to renal failure
B. High AG metabolic acidosis secondary to dehydration and lactic acidosis
C. Normal AG metabolic acidosis secondary to diarrhoea
D. Concurrent normal AG metabolic acidosis and respiratory alkalosis
shows:
• pH 7.27 (7.35–7.45)
• HCO3 −. 14 mmol/L (22–24)
• PCO2 28 mmol/L (35–45)
• K+ 2.6 mmol/L (3.5–5.0)
• Na+ 134 mmol/L (135–145)
• Cl− 113 mmol/L (95–105)
• Ur 14 mmol/L (2–7)
• Cr 320 mmol/L (50–100)
• lactate 1.5 mmol/L (<2.2).
Which ONE of the following options would BEST explain the clinical scenario?
A. High AG metabolic acidosis secondary to renal failure
B. High AG metabolic acidosis secondary to dehydration and lactic acidosis
C. Normal AG metabolic acidosis secondary to diarrhoea
D. Concurrent normal AG metabolic acidosis and respiratory alkalosis
Forwarded from Yaqob Hamood
👨🏻⚕Is mmol/L equal mEq/ L ?
Forwarded from Yaqob Hamood
A 35-year-old female presents with a prolongedseizure. Her initial ABG is shown.
• pH 7.25 (7.35–7.45)
• PCO2 55 mmol/L (35–45)
• PO2 100 mmol/L (80–100)
• HCO3 15 mmol/L (22–24)
• Na+ 135 mmol/L (135–145)
• K+ 4.5 mmol/L (3.5–5)
• Cl- 98 mmol/L (95–105).
Which ONE of the following explains the aboveblood gas?
A. Mixed normal AG metabolic acidosis andrespiratory acidosis
B. High AG metabolic acidosis
C. Mixed high AG metabolic acidosis and metabolicalkalosis
D. Mixed high AG metabolic acidosis andrespiratory acidosis
• pH 7.25 (7.35–7.45)
• PCO2 55 mmol/L (35–45)
• PO2 100 mmol/L (80–100)
• HCO3 15 mmol/L (22–24)
• Na+ 135 mmol/L (135–145)
• K+ 4.5 mmol/L (3.5–5)
• Cl- 98 mmol/L (95–105).
Which ONE of the following explains the aboveblood gas?
A. Mixed normal AG metabolic acidosis andrespiratory acidosis
B. High AG metabolic acidosis
C. Mixed high AG metabolic acidosis and metabolicalkalosis
D. Mixed high AG metabolic acidosis andrespiratory acidosis
in text books and lectures , statement : excess CO2 production in the body leads to acidosis
my confusion : even if tons of CO2 being produced , what matters ??
CO2 reacts with H20 and is converted to H2CO3 and dissociates to H+ & HCO3-
H+ is an acid while bicarbonate is a base , which neutralizes each other .
so what's the problem with excess CO2 ??
isn't it a self-solved issue ??
my confusion : even if tons of CO2 being produced , what matters ??
CO2 reacts with H20 and is converted to H2CO3 and dissociates to H+ & HCO3-
H+ is an acid while bicarbonate is a base , which neutralizes each other .
so what's the problem with excess CO2 ??
isn't it a self-solved issue ??
internal medicine
in text books and lectures , statement : excess CO2 production in the body leads to acidosis my confusion : even if tons of CO2 being produced , what matters ?? CO2 reacts with H20 and is converted to H2CO3 and dissociates to H+ & HCO3- H+ is an acid while…
،this question is from member ?
internal medicine
🧑🏼⚕ My diagnosis is chronic rheumatic heart disease with mitral regurgitation.
Please open Telegram to view this post
VIEW IN TELEGRAM
internal medicine
🧑⚕A. As follows:
✓ Tricuspid regurgitation (TR)
✓ Ventricular septal defect (VSD).
✓ Tricuspid regurgitation (TR)
✓ Ventricular septal defect (VSD).