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Let's talk about Eternal life
A senolytic is among a class of small molecules under basic research to determine if they can selectively induce death of senescent cells and improve health in humans. A goal of this research is to discover or develop agents to delay, prevent, alleviate, or reverse age-related diseases.A related concept is "senostatic", which means to suppress senescence.
A senolytic is among a class of small molecules under basic research to determine if they can selectively induce death of senescent cells and improve health in humans. A goal of this research is to discover or develop agents to delay, prevent, alleviate, or reverse age-related diseases.A related concept is "senostatic", which means to suppress senescence.
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Forwarded from magic is fun
Azot (ahuizotl) is a gene named after the multi-armed Aztec mythological creature that protected lakes from fishing boats. This gene is involved in the removal of aged and damaged cells in the body to maintain healthy tissues during development and adulthood[1]. Lack of this gene leads to increased morphologic deformities and susceptibility to random mutations and accelerates degenerative processes in body tissues.
Usually there are two copies of this gene in a cell. The researchers were able to add a third copy of the Azot gene to the cell of Drosophila melanogaster. As a result, the rejection of aged cells improved and the average lifespan of individuals increased by 50-60% compared to normal.
The azot gene consists of a single exon encoding an evolutionarily conserved cytoplasmic protein with four EF arms.
https://www.sciencedaily.com/releases/2015/01/150115134624.htm
Usually there are two copies of this gene in a cell. The researchers were able to add a third copy of the Azot gene to the cell of Drosophila melanogaster. As a result, the rejection of aged cells improved and the average lifespan of individuals increased by 50-60% compared to normal.
The azot gene consists of a single exon encoding an evolutionarily conserved cytoplasmic protein with four EF arms.
https://www.sciencedaily.com/releases/2015/01/150115134624.htm
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