We need another protest as they really do not get it.
https://twitter.com/PoliticsForAlI/status/1373573454159568897?s=19
https://twitter.com/PoliticsForAlI/status/1373573454159568897?s=19
Twitter
Politics For All
🚨BREAKING: Mary Ramsay, from Public Health England, says measures like masks and travel restrictions are likely to continue “certainly for a few years” before we “gradually go back to a more normal situation” Via @BBCNews
Frontiers | Editorial: Antidepressant Prescriptions in Children and Adolescents | Psychiatry
https://www.frontiersin.org/articles/10.3389/fpsyt.2020.600283/full
https://www.frontiersin.org/articles/10.3389/fpsyt.2020.600283/full
Frontiers
Editorial: Antidepressant Prescriptions in Children and Adolescents
The use of antidepressants in children and adolescents has a troubled history, for almost all principles of good evidence-based medicine were violated or compromised. It is a history characterised by systematically biased research, financial conflicts of…
BRISTOL KILL THE BILL PROTESTS. PLEASE ADD ANY VIDEO LINKS AND FOOTAGE IN THIS THREAD. I WOULD LIKE TO GET MORE INFO ON WHAT HAPPENED/WHAT IS HAPPENING/WHICH GROUP CALLED THE PROTEST ETC. NO NAMES PLEASE REMEMBER WE HAVE TO ASSUME THIS CHANNEL IS MONITORED.
London - Rally for Freedom - 20/03/2021
https://youtu.be/Er1RbeRk03E
https://youtu.be/Er1RbeRk03E
Russia makes its move: it unequivocally opposes vaccine passports for travel. Says it will certainly lead to forced vaccination
https://youtu.be/VtGyXDMMP3E
https://youtu.be/VtGyXDMMP3E
Uploaded a few inconvenient facts on my Instagram today. Take a look, this is just one of four:
https://www.instagram.com/p/CMt8nvwsbPV/?igshid=1w1azcrovdemz
https://www.instagram.com/p/CMt8nvwsbPV/?igshid=1w1azcrovdemz
Take a look at this thread. Confirms most of my research:
Thread by @BidoliNicola on Thread Reader App – Thread Reader App
https://threadreaderapp.com/thread/1361669981302317057.html
Thread by @BidoliNicola on Thread Reader App – Thread Reader App
https://threadreaderapp.com/thread/1361669981302317057.html
Threadreaderapp
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I think SARSCoV2 persists in the body, Nicola Bidoli calls it parasitic. I can think of two ways in which it can persist:
1 It continues replicating in the human bacterial microbiome, but when immune defenses are working well other bacteria in the microbiome keep it in check.
2 It is a retrovirus, or parts of it are. It persists in human cells but significant replication only occurs when immune defenses are down.
I think that both options are possible, as well as the possibility that both are true.
What do you think? How do you tell a retrovirus from a non retrovirus in terms of its microbiological properties and composition?
We already know there is research proving that SARSCoV2 replicates in the bacterial microbiome (Brogna et al, 2020). Of course what defends in you in both cases are:
1 Vitamin D 2,000 IUs per day minimum, 4,000 IUs for adults of normal size
2 Macrolides, bacteriostatics, antibiotics at the signs of high fever (over 38.8,°C approx)
1 It continues replicating in the human bacterial microbiome, but when immune defenses are working well other bacteria in the microbiome keep it in check.
2 It is a retrovirus, or parts of it are. It persists in human cells but significant replication only occurs when immune defenses are down.
I think that both options are possible, as well as the possibility that both are true.
What do you think? How do you tell a retrovirus from a non retrovirus in terms of its microbiological properties and composition?
We already know there is research proving that SARSCoV2 replicates in the bacterial microbiome (Brogna et al, 2020). Of course what defends in you in both cases are:
1 Vitamin D 2,000 IUs per day minimum, 4,000 IUs for adults of normal size
2 Macrolides, bacteriostatics, antibiotics at the signs of high fever (over 38.8,°C approx)
MELATONIN: "Under physiological conditions, circulating melatonin (N-acetyl-5-methoxytryptamine) is mainly released from the pineal gland. However, its production has also been demonstrated in several extrapineal organs, including immune system cells, CNS, bone marrow, skin and likely other tissues [21]. The therapeutic value of melatonin in mental disorders and neurodegenerative diseases, cardiovascular diseases, cancer, gastrointestinal pathologies or infectious diseases has been highlighted in numerous scientific reports [22,23,24]. The multiple actions of melatonin have been attributed to its potent antioxidant activity, modifying energy metabolism as well as its ability to modulate the immune response and neuroinflammation among others [25,26,27,28]. In this respect, the immunomodulatory actions of melatonin at acute high doses may counteract the response to sepsis [29,30]. However, many melatonin actions are also mediated through its interaction with two high-affinity G-protein-coupled receptors, named MT1 and MT2, whose physiological functions and pharmacological properties are well documented [28].
In previous studies, we have demonstrated that melatonin provides a protective effect in brain ischemia that, at least in part, depends on the nicotinic receptor (nAChR) activation, the overexpression of HO-1 and the reduction of oxidative stress [31]. Moreover, melatonin may regulate autophagic flux via α7 nAChRs [32], and activation of these receptors in microglia has a potent anti-inflammatory role [33,34]. Here, we show that melatonin reduces oxidative stress and NLRP3 triggering due to activation of autophagic flux."
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7767051/
In previous studies, we have demonstrated that melatonin provides a protective effect in brain ischemia that, at least in part, depends on the nicotinic receptor (nAChR) activation, the overexpression of HO-1 and the reduction of oxidative stress [31]. Moreover, melatonin may regulate autophagic flux via α7 nAChRs [32], and activation of these receptors in microglia has a potent anti-inflammatory role [33,34]. Here, we show that melatonin reduces oxidative stress and NLRP3 triggering due to activation of autophagic flux."
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7767051/
PubMed Central (PMC)
Melatonin Reduces NLRP3 Inflammasome Activation by Increasing α7 nAChR-Mediated Autophagic Flux
Microglia controls the immune system response in the brain. Specifically, the activation and dysregulation of the NLRP3 inflammasome is responsible for the initiation of the inflammatory process through IL-1β and IL-18 release. In this work, we ...
The Bad News (see Vera Sharav)
https://twitter.com/AdamWagner1/status/1374003219005911042?s=19
https://twitter.com/AdamWagner1/status/1374003219005911042?s=19
Twitter
Adam Wagner
"8.—(1) No person may, without a reasonable excuse— (a) leave England to travel to a destination outside the United Kingdom, or (b) travel to, or be present at, an embarkation point for the purpose of travelling from there to a destination outside the United…
Children across Italy protest over covid school closures
https://www.wantedinrome.com/news/children-across-italy-protest-over-covid-school-closures.html
https://www.wantedinrome.com/news/children-across-italy-protest-over-covid-school-closures.html
Wanted in Rome
Children across Italy protest over covid school closures
Thousands of parents, children and teachers took to the streets in cities across Italy to protest the closure of schools under Italy's covid-19 restrictions.