Robin Monotti + Cory Morningstar
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VITAMIN D3 "Vitamin D3 is a secosteroid hormone produced in the skin in amounts estimated up to 25,000 international units (IUs) a day by the action of UVB radiation on 7-dehydrocholesterol. Vitamin D deficiency is common due to both lack of adequate sun exposure to the skin, and because vitamin D is present in very few food sources. Deficiency is strongly linked to increased risk for a multitude of diseases, several of which have historically been shown to improve dramatically with either adequate UVB exposure to the skin, or to oral or topical supplementation with vitamin D. These diseases include asthma, psoriasis, rheumatoid arthritis, rickets and tuberculosis. All patients in our hospital have been routinely screened on admission for vitamin D deficiency since July 2011, and offered supplementation to either correct or prevent deficiency. During this time, we have admitted over 4700 patients, the vast majority of whom agreed to supplementation with either 5000 or 10,000 IUs/day. Due to disease concerns, a few agreed to larger amounts, ranging from 20,000 to 50,000 IUs/day. There have been no cases of vitamin D3 induced hypercalcemia or any adverse events attributable to vitamin D3 supplementation in any patient...In summary, long-term supplementation with vitamin D3 in doses ranging from 5000 to 50,000 IUs/day appears to be safe."
https://www.sciencedirect.com/science/article/abs/pii/S0960076018306228
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Vera Sharav, Holocaust survivor, founder of the Alliance for Human Research Protection and biomedical activist, in her testimony to the German Corona Committee, compares the mandating of face masks to the yellow star imposed on Jews under the Third Reich:
https://youtu.be/r02j7HaKYe8
"We do know that at least ninety-five per-cent of people who are recorded as dying of COVID19 had other serious medical conditions. Claiming that COVID19 was the primary/recordable cause of death in all of these cases is just ridiculous. Beyond ridiculous."
https://drmalcolmkendrick.org/2021/03/20/covid19-hidden-figures-and-ooda/
WE HAVE ALMOST WON THIS:
"Covid rules on groups gathering outdoors 'not policeable' ahead of lockdown easing: Chairman of Metropolitan Police Federation says his members have no desire to carry out the law to the letter"
https://www.telegraph.co.uk/news/2021/02/28/covid-rules-groups-gathering-outdoors-not-policeable-ahead-lockdown/
BRISTOL KILL THE BILL PROTESTS. PLEASE ADD ANY VIDEO LINKS AND FOOTAGE IN THIS THREAD. I WOULD LIKE TO GET MORE INFO ON WHAT HAPPENED/WHAT IS HAPPENING/WHICH GROUP CALLED THE PROTEST ETC. NO NAMES PLEASE REMEMBER WE HAVE TO ASSUME THIS CHANNEL IS MONITORED.
London - Rally for Freedom - 20/03/2021
https://youtu.be/Er1RbeRk03E
Russia makes its move: it unequivocally opposes vaccine passports for travel. Says it will certainly lead to forced vaccination
https://youtu.be/VtGyXDMMP3E
Uploaded a few inconvenient facts on my Instagram today. Take a look, this is just one of four:

https://www.instagram.com/p/CMt8nvwsbPV/?igshid=1w1azcrovdemz
I think SARSCoV2 persists in the body, Nicola Bidoli calls it parasitic. I can think of two ways in which it can persist:
1 It continues replicating in the human bacterial microbiome, but when immune defenses are working well other bacteria in the microbiome keep it in check.
2 It is a retrovirus, or parts of it are. It persists in human cells but significant replication only occurs when immune defenses are down.
I think that both options are possible, as well as the possibility that both are true.
What do you think? How do you tell a retrovirus from a non retrovirus in terms of its microbiological properties and composition?
We already know there is research proving that SARSCoV2 replicates in the bacterial microbiome (Brogna et al, 2020). Of course what defends in you in both cases are:
1 Vitamin D 2,000 IUs per day minimum, 4,000 IUs for adults of normal size
2 Macrolides, bacteriostatics, antibiotics at the signs of high fever (over 38.8,°C approx)
MELATONIN: "Under physiological conditions, circulating melatonin (N-acetyl-5-methoxytryptamine) is mainly released from the pineal gland. However, its production has also been demonstrated in several extrapineal organs, including immune system cells, CNS, bone marrow, skin and likely other tissues [21]. The therapeutic value of melatonin in mental disorders and neurodegenerative diseases, cardiovascular diseases, cancer, gastrointestinal pathologies or infectious diseases has been highlighted in numerous scientific reports [22,23,24]. The multiple actions of melatonin have been attributed to its potent antioxidant activity, modifying energy metabolism as well as its ability to modulate the immune response and neuroinflammation among others [25,26,27,28]. In this respect, the immunomodulatory actions of melatonin at acute high doses may counteract the response to sepsis [29,30]. However, many melatonin actions are also mediated through its interaction with two high-affinity G-protein-coupled receptors, named MT1 and MT2, whose physiological functions and pharmacological properties are well documented [28].
In previous studies, we have demonstrated that melatonin provides a protective effect in brain ischemia that, at least in part, depends on the nicotinic receptor (nAChR) activation, the overexpression of HO-1 and the reduction of oxidative stress [31]. Moreover, melatonin may regulate autophagic flux via α7 nAChRs [32], and activation of these receptors in microglia has a potent anti-inflammatory role [33,34]. Here, we show that melatonin reduces oxidative stress and NLRP3 triggering due to activation of autophagic flux."
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7767051/