Slide 13
❗ D :- rheumatic heart disease
❗ c.f :- Aschoff
body in acute rheumatic
carditis.

Slide 14
❗ D :- Rheumatic myocarditis
❗c.f :- Presence of Aschoff bodies.

Slide 15
❗ D: Rheumatic
heart disaese
❗ c.f : Aschoff
bodies with binucleated macrophage.

Slide 16
1.
❗ D :- Dilated cardiomyopathy
❗ c.f :-
Dilation of all chambers
بخلاف الdilation الذي بسبب ال hypertension يحدث
dilation only in 2 chambers

2.
❗D :- hypertrophic cardiomyopathy
❗c.f :- hypertrophy of all cardiac muscle .
بخلاف الذي يحصل بسبب hypertension يكون ققي في
1 chamber

3.
❗D :- restrictive cardiomyopathy
❗c.f :-
loss of cardiac compliance.

Slide 17
❗ D :- sample of hypertrophic
cardiacmyopathy.
❗ c.f :-
Irregular ( abnormal distorted ) cardiac myocyte

Slide 18
❗D :- Dilated cardiomyopathy

❗ c.f :-
- small myocyte and between them more C.T

Slide 19
❗D :- types of vegetation
❗ c.f :-
1. RHD :
- small vegetation ( warty ) at site of closure .
- normal valve tissue .
2. IE :
- large vegetation
- destructive valve tissue
3. NBTE :
- mass of thrombus ( small bland ) attached to the valve
4. LSE
- similar to NBTE but on both sides .

Slide 20
❗ D :- infective endocarditis.
❗ c.f :-severe inflammation with vegetation in the edge of leaflets and upper and lower.

Slide 21
❗ D: infective endocarditis
❗c.f : bacterial colonies + fibrin
deposition + inflammatory cells

Slide 22
Similar slide21

Slide 23
Similar slide 21

Slide 24
❗ D: NBTE

❗ c.f : slight or no inflammation with fibrin no bacteria

Slide 25
❗ D: NBTE

❗ c.f : slight or no inflammation with fibrin no bacteria

Slide 26
❗ D: Acute myocarditis
❗ c.f :-
- Neutrophilic infiltrate in
between myocyte
-not MI because here
muscles and nucleus are intact
and viable no necrosis
 In MI cells are dead
“ necrosis “ no nucleus
not MI.
طبعا الصورة هي تشرح أنواع الmyocarditis الأربعة،
A: lymphocytic myocarditis.
B: Hypersenstivity myocarditis.
C: Giant cell Myocarditis.
D: Chagas myocarditis.
ولكل واحدة خواصها،
Features:
A: edema and lymphcyte infilterate.
B: esinophils.
C:Giant cells and extensive damage.
D: Myofuber distended with tryponsoma with Ch. inf. infilterate.

Slide 27
❗D :- cardiac myxoma
❗ c.f :-
- benign tumor arise from C.T
- myxoid material + blood vessels

Slide 28
❗D:- Rheumatic valvular
endocarditis
❗c.f :- Presence of Aschoff
bodies
in addition to the above mentioned charcteristics about RVD, the slide here exhibit a clear gross Aschof Body.
in addition to the above mentioned charcteristics about RVD, the slide here exhibit a clear gross Aschof Body.

Slide
❗D :- pericarditis
❗c.f :-
- inflammation of pericardium
- adhesive scaring
- excessive production of serious fluid

Slide 5
❗ D: Clubbing fingersIt occurs in cyanotic heart ,lung disease and liver disease .
❗c.f :- spoon-like fingers

Slide 7
❗ D:- steps of MI
❗ c.f :-
1. Normal myocytes .
2. Cuagulative necrosis " no nucleus of myocytes " with neutrophiles .
3. Cleaning of all dead myomcyte by macrophage.
4. Granulation tissue and formation of blood vessel
5. Fibrosis " by fibroblast "

مقرر معمل الCardiac من إعداد الزين ابراهيم سعيد،
له منا جزيل الشكر وبالغ الإمتنان ع جهده وتفانيه،

#سلايدات
#معمل_باثو
#مجموعة_الباثو
#اللجنة_العلمية

💢ملاحظات💢
حول بكتيريا Enterobacteriaceae
تقسم الى مجموعتين

⭕️المجموعه الاولى:-

⬅️المخمره للاكتوز
Lactose fermenter
مثل:-
🔸Escherichia
🔸Klebsiella
🔸Entrobacter
🔸Citrobacter
ملاحظات مهمه جدا تساعد في التشخيص حول هذه المجموعة:-
1⃣-كلها مخمرة للسكر وتعطي مستعمرات ورديه داخل MacConkey agar
2⃣جميعها تعطي نتيجه A/A داخل وسط TSI او KIA
3⃣جميعها oxidase negative
4⃣الوحيدة منها Indole positive هي E.coli
5⃣ -الوحيدة منها التي تعطي H2S positive هي Citrobacter
6⃣-كلها متحركه ماعدا Klebsiella
7⃣-كلها Citrate positive ماعدا E.coli
8⃣-كلها منتجة للــ Gas