Lifestyle factors are common contributors to resistant hypertension:

• Elderly patients, black patients, and those with CKD tend to be particularly salt sensitive.
• In an estimated 10% of adults with hypertension in the United States, the hypertension is due primarily to alcohol consump-tion.
• For every 10% increase in body weight, systolic BP increases by 6.5 mm Hg.

What's your dx?
@contactzero_bot

ها صدك

The patient is an old, thin woman with abdominal pain

أول ما شفت الأشعة ضربتلي صفنة داحاول أفتهم شديصير. من جهة هي تبدو كأنها gallstones ومكانها هو نفسه مكان المرارة بس من جهة ثانية شكلها عبالك حبوب كهوة وما يرهم يكون gallstones... مع ذلك سألت المُرافقة إذا الحجية عدها ألم ببطنها أو حصوة بالمرارة... المُرافقة افتهمت السالفة (على ما يبدو هالأشعة محيرة كم شخص قبلي وسائليها نفس الاسئلة) ف كالتلي أنّ الحجية عايفة السبحة بجيبها وما تقبل تعوفها ولهذا طالعة هيج.

الظاهر الردهة كلها طلعت تعرف بالسالفة، بحيث لما انداريت لكيت المريضة اللي كبالنا هم دتضحك وتكلي "والله الحجية عدها سوالف"

As for the abdominal pain, I forgot to even ask about it or take a proper hx tbh... I have seen enough medicine for the day

They are used to rule out stable angina. Theyshouldn't be used in low-risk patients because they have a high false-positive probability.
They induce ischemia by various ways and then detect it by ECG, echocardiography, or radionuclide imaging.

Stress tests can be divided into:

• Exercise stress test, most commonly using a treadmill.

• Pharmacological stress tests. They are used in patients who are unable to exercise or the exercise stress test is contraindicated (like in LBBB). They are further divided based on the drug being used into:

A. Positive inotropic/chronotropic drugs like dobutamine which is a B1-receptor agonist and simulates the heart physiology during exercise (in terms of increasing the heart contractility & rhythm).

B. Vasodilators like adenosine & regadenoson (adenosine analogue). They make heart ischemia manifest via a mechanism called coronary steal.

The idea is fairly simple: Adenosie and the likes of it specifically activate the A2A adenosine receptor in the coronary vessels which causes coronary vasodilation. In terms of blood flow, normal coronary vessels are up to 400% more responsive to the vasodilatory effect of adenosine than stenotic vessels. Therefore, they dilate & the blood flow increases in them. Meanwhile, atherosclerotic coronary vessels cannot dilate properly (because they are already maximally dilated) and blood flow is stolen from these narrowed coronary vessels to normal coronary vessels. This induces symptoms of ischemia, and perfusion defects appear in cardiac nuclear imaging or as ST-segment changes on the ECG.

Syphilis (and afterwards, TB) is often nicknamed "the great imitator/masquerader" because of its protean nature. It has nonspecific symptoms that can be confused with many other diseases which make it hard to diagnose and easily misdiagnosed.

📝 Quick note
اغلب المرضى أهل CKD on dialysis يجون للطوارئ
ونسويلهم ECG
من ضمن الشغلات المهمة لازم تنتبة الها هي

Bradycardia Bradycardia Bradycardia

لا تستعجل وتنطيهم atropine بل عاملهم معاملة hyperK وانطي Calcium gluconate

👉 The only ECG sign of severe and life threatening hyperK may simply be bradycardia
👉 Bradycardia is hyperK in dialysis patients until proven otherwise

Hyperkalemia is often called "the syphilis of ECG" because it can present in so many forms; bradyarrhythmia, tachyarrhythmia, AV-block, BB block, and many others...

طبعًا الشكر موصول لدكتور يوسف نسائية على هالمعلومة القيمة

An ECG report takes the form of a description followed by an interpretation.

The description should always be given in the same sequence:
1. Rate & Rhythm
2. Conduction intervals
3. Cardiac axis
4. A description of the QRS complexes
5. A description of the ST segments and T waves.

The interpretation of an ECG indicates whether the record is normal or abnormal: if abnormal, the underlying pathology needs to be identified. One of the main problems of ECG reporting is that there is quite a lot of normal variation in the ECG which are consistent with perfectly normal hearts. Recognizing the limits of normality is one of the main difficulties of ECG interpretation.