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دتكفرني كمية الأشياء اللي أكتبها هنا، وراها بفترة لما انسئل عنها أنساها...

ليهسة أتذكر بالباطنية مال الرابع بسشنات النيورو، انسألت على الـ UMN lesions وما عرفت الجواب... واني قبلها بسنة بالضبط شارحها هنا :")

المهم...

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Within 10 years of developing overt symptoms of diabetic autonomic neuropathy, 30–50% of patients are dead, many from sudden cardiorespiratory arrest. Patients with postural hypotension (a drop in systolic pressure of 30 mmHg or more on standing from the supine position) have the highest subsequent mortality.

Basic principles of potassium homeostasis:

This brings me to "Hypokalemia in DKA"

Every patient in DKA is potassium-depleted but the plasma concentration of potassium gives very little indication of the total body deficit. Plasma potassium may even be raised initially due to disproportionate loss of water, and displacement of potassium from the intracellular compartment by H+ ions.

Soon after treatment is started, there is likely to be a precipitous fall in the plasma potassium due to dilution of extracellular potassium by administration of intravenous fluids, the movement of potassium into cells induced by insulin, and the continuing renal loss of potassium.

Me when a DKA patient presents with hyperkalemia but actually has hypokalemia: