🔴Haemorrhoids (Piles)
▪︎Enlargement & Protrusion Of Normal Anal Cushions Into The Anal Canal (Symptomatic Anal Cushions Arise From Anal Venous Plexuses)
▪︎Due To Shearing Forces Acting On The Anus Leads To Trauma To Anal Mucosa & Downward Displacement Of Anal Cushions With Fragmentation & Loss Elasticity Of Anal Supporting Structures Leads To Anal Cushions Protrusion & No Retraction After Defecation
🔹Risk Factors For Haemorrhoids
1. Chronic Constipation & Straining
2. Pregnancy & Multiple Births
3. Obesity
4. Portal Hypertension
5. Diet Low in Fibers
6. Repeated Anal Infections
7. Anal Sphincter HyperTonia
8. Sitting in Toilet For Long Time
🔰Types Of Haemorrhoids
🔶A. Primary Internal Haemorrhoids
▪︎Symptomatic Anal Cushions Arise From Superior (Deep Or Internal) Haemorrhoidal Plexus Above The Dentate Line (Pectinate Line Or Upper Anal Canal)
▪︎Covered By Columnar Epithelium Of The Upper Anal Canal That Lacks Pain Receptors (Painless)
▪︎Present in 3 & 7 & 11 O'clock Position Inside The Anal Canal (Left Lateral/Right Posterior/Right Anterior in Lithotomy)
▪︎Not Visible From Outside
▪︎Idiopathic Aetiology
🔹Clinical Features Of Internal Haemorrhoids
1. Bright-Red Painless Rectal Bleeding After Defecation & Separated From The Stool (Seen On Paper After Wiping Or in The Pan)
2. Anal Discomfort & Irritation (Itching) Due To Mucous Discharge
3. Lump Prolapse During Defecation
4. Acute Thrombosis Of Prolapsed Haemorrhoids Results in Severe Anal Pain With Anal Sphincter Spasm & Large Swollen Irreducible Haemorrhoid
🔹Degree Of Internal Haemorrhoids
1. Only Bleeding (No Prolapse)
2. Prolapse Reduced Spontaneously
3. Prolapse Reduced Manually
4. Permanent Prolapse (Irreducible)
🔹Mx Of Internal Haemorrhoids
📍1. Exclusion Other Causes Of Rectal Bleeding (ColoRectal Cancer)
📍2. Normalize The Bowel & Defaectory Habits
▪︎Stool Softeners
▪︎Increase Dietary Fibers & Fluids Intake
▪︎Warm Baths & Local Anesthetics To Relive Pain
▪︎Defecation Only When Natural Desire Present
▪︎Minimize Straining
📍3. ScleroTherapy Injection (5ml 5% SubMucosal Phenol Injection) ➡️ For 1st & 2nd Degree Haemorrhoids Not Improved On Conservative Tx
📍4. Haemorrhoidal Banding (Barron's Banding) ➡️ For More Bulky (Large) Haemorrhoids
📍5. Haemorrhoidal Artery Ligation & Tissue Fixation (THD/HALO/HaemorrhoidoPexy) ➡️ For 2nd & 3rd Degree Haemorrhoids
📍6. Surgical Haemorrhoidectomy For
▪︎3rd & 4th Degree Haemorrhoids
▪︎2nd Degree Haemorrhoids Not Cured By Other Methods
▪︎Fibrosed Haemorrhoids (Thrombosed External Haemorrhoids Undergo Fibrosis)
▪︎Mixed (Internal & External Haemorrhoids) When The External One is Well Defined
▪︎Chronic Rectal Bleeding Causing Anemia
📍7. Postoperative Care
▪︎Two Warm Baths Each Day
▪︎Stool Softeners/Metronidazole/Analgesics
🔹Complications Of Internal Haemorrhoids
🔶A. Preoperative
1. Chronic Bleeding & Anemia
2. Severe Acute Bleeding ➡️ Do Local Compression & Adrenaline Injection & Blood Transfusion Then Ligation & Excision Of The Haemorrhoid
3. Strangulation Then Thrombosis & Gangrene ➡️ Give AB & Analgesics With Hot Baths For 3-4 Days (To Induce Shrinkage) Then Do Haemorrhoidectomy
4. Ulceration/Fibrosis
5. Portal Pyaemia
🔶B. Postoperative
📍1. Early ➡️ Pain/Urine Retention/Reactionary Bleeding (More Common)
📍2. Late
▪︎Secondary Bleeding
▪︎Anal Strictures
▪︎Anal Fissures & Abscess
▪︎Incontinence
#Haemorrhoids
#surgery
https://t.me/Surgery_Practice
▪︎Enlargement & Protrusion Of Normal Anal Cushions Into The Anal Canal (Symptomatic Anal Cushions Arise From Anal Venous Plexuses)
▪︎Due To Shearing Forces Acting On The Anus Leads To Trauma To Anal Mucosa & Downward Displacement Of Anal Cushions With Fragmentation & Loss Elasticity Of Anal Supporting Structures Leads To Anal Cushions Protrusion & No Retraction After Defecation
🔹Risk Factors For Haemorrhoids
1. Chronic Constipation & Straining
2. Pregnancy & Multiple Births
3. Obesity
4. Portal Hypertension
5. Diet Low in Fibers
6. Repeated Anal Infections
7. Anal Sphincter HyperTonia
8. Sitting in Toilet For Long Time
🔰Types Of Haemorrhoids
🔶A. Primary Internal Haemorrhoids
▪︎Symptomatic Anal Cushions Arise From Superior (Deep Or Internal) Haemorrhoidal Plexus Above The Dentate Line (Pectinate Line Or Upper Anal Canal)
▪︎Covered By Columnar Epithelium Of The Upper Anal Canal That Lacks Pain Receptors (Painless)
▪︎Present in 3 & 7 & 11 O'clock Position Inside The Anal Canal (Left Lateral/Right Posterior/Right Anterior in Lithotomy)
▪︎Not Visible From Outside
▪︎Idiopathic Aetiology
🔹Clinical Features Of Internal Haemorrhoids
1. Bright-Red Painless Rectal Bleeding After Defecation & Separated From The Stool (Seen On Paper After Wiping Or in The Pan)
2. Anal Discomfort & Irritation (Itching) Due To Mucous Discharge
3. Lump Prolapse During Defecation
4. Acute Thrombosis Of Prolapsed Haemorrhoids Results in Severe Anal Pain With Anal Sphincter Spasm & Large Swollen Irreducible Haemorrhoid
🔹Degree Of Internal Haemorrhoids
1. Only Bleeding (No Prolapse)
2. Prolapse Reduced Spontaneously
3. Prolapse Reduced Manually
4. Permanent Prolapse (Irreducible)
🔹Mx Of Internal Haemorrhoids
📍1. Exclusion Other Causes Of Rectal Bleeding (ColoRectal Cancer)
📍2. Normalize The Bowel & Defaectory Habits
▪︎Stool Softeners
▪︎Increase Dietary Fibers & Fluids Intake
▪︎Warm Baths & Local Anesthetics To Relive Pain
▪︎Defecation Only When Natural Desire Present
▪︎Minimize Straining
📍3. ScleroTherapy Injection (5ml 5% SubMucosal Phenol Injection) ➡️ For 1st & 2nd Degree Haemorrhoids Not Improved On Conservative Tx
📍4. Haemorrhoidal Banding (Barron's Banding) ➡️ For More Bulky (Large) Haemorrhoids
📍5. Haemorrhoidal Artery Ligation & Tissue Fixation (THD/HALO/HaemorrhoidoPexy) ➡️ For 2nd & 3rd Degree Haemorrhoids
📍6. Surgical Haemorrhoidectomy For
▪︎3rd & 4th Degree Haemorrhoids
▪︎2nd Degree Haemorrhoids Not Cured By Other Methods
▪︎Fibrosed Haemorrhoids (Thrombosed External Haemorrhoids Undergo Fibrosis)
▪︎Mixed (Internal & External Haemorrhoids) When The External One is Well Defined
▪︎Chronic Rectal Bleeding Causing Anemia
📍7. Postoperative Care
▪︎Two Warm Baths Each Day
▪︎Stool Softeners/Metronidazole/Analgesics
🔹Complications Of Internal Haemorrhoids
🔶A. Preoperative
1. Chronic Bleeding & Anemia
2. Severe Acute Bleeding ➡️ Do Local Compression & Adrenaline Injection & Blood Transfusion Then Ligation & Excision Of The Haemorrhoid
3. Strangulation Then Thrombosis & Gangrene ➡️ Give AB & Analgesics With Hot Baths For 3-4 Days (To Induce Shrinkage) Then Do Haemorrhoidectomy
4. Ulceration/Fibrosis
5. Portal Pyaemia
🔶B. Postoperative
📍1. Early ➡️ Pain/Urine Retention/Reactionary Bleeding (More Common)
📍2. Late
▪︎Secondary Bleeding
▪︎Anal Strictures
▪︎Anal Fissures & Abscess
▪︎Incontinence
#Haemorrhoids
#surgery
https://t.me/Surgery_Practice
🔶B. External Haemorrhoids (PeriAnal Hematoma)
▪︎Thrombosis In Veins Of Inferior (Superficial Or External) Haemorrhoidal Plexus Below The Dentate Line (Pectinate Line Or Lower Anal Canal)
▪︎Covered By Squamous Epithelium Of Lower Anal Canal & Skin (Painful)
🔹Clinical Features Of External Haemorrhoids
1. Sudden Onset Of Painful Olive-Shaped Blue Subcutaneous Mass Protrusion At Anal Opening (Anal Verge)
2. Visible From Outside (Mass Can Be Seen Near The Anus)
3. Present After Straining/Coughing/Lifting Heavy Weight
🔹Mx Of External Haemorrhoids
📍1. Incision & Clot Evacuation If Present Within 48h
📍2. Mostly Resolve Spontaneously Or Fibrosed (Called 5 Day Pain Or Self Curing Lesion)
📍3. Haemorrohidectomy (If Symptomatic & Persist)
📍4. Its Fate If Not Treated ➡️
▪︎Form Cutaneous Tag
▪︎Burst & Disappears
▪︎Continue Bleeding
▪︎Suppurate (Become Infected)
🔶C. Secondary Internal Haemorrhoids Due To
1. AnoRectal Carcinoma Or Deformity/HypoTonic Anal Sphincter
2. Abdominal Ascites
3. Uterine Enlargement/Uterine & Ovarian & Bladder Tumour
4. ParaPlegia/Multiple Sclerosis
🔶D. InteroExternal Haemorrhoids (Mixed) ➡️ External Extension Of Internal Haemorrhoids To Involve The Superior & Inferior Haemorrhoidal Plexuses
#Haemorrhoids
#part 2
#surgery
https://t.me/Surgery_Practice
▪︎Thrombosis In Veins Of Inferior (Superficial Or External) Haemorrhoidal Plexus Below The Dentate Line (Pectinate Line Or Lower Anal Canal)
▪︎Covered By Squamous Epithelium Of Lower Anal Canal & Skin (Painful)
🔹Clinical Features Of External Haemorrhoids
1. Sudden Onset Of Painful Olive-Shaped Blue Subcutaneous Mass Protrusion At Anal Opening (Anal Verge)
2. Visible From Outside (Mass Can Be Seen Near The Anus)
3. Present After Straining/Coughing/Lifting Heavy Weight
🔹Mx Of External Haemorrhoids
📍1. Incision & Clot Evacuation If Present Within 48h
📍2. Mostly Resolve Spontaneously Or Fibrosed (Called 5 Day Pain Or Self Curing Lesion)
📍3. Haemorrohidectomy (If Symptomatic & Persist)
📍4. Its Fate If Not Treated ➡️
▪︎Form Cutaneous Tag
▪︎Burst & Disappears
▪︎Continue Bleeding
▪︎Suppurate (Become Infected)
🔶C. Secondary Internal Haemorrhoids Due To
1. AnoRectal Carcinoma Or Deformity/HypoTonic Anal Sphincter
2. Abdominal Ascites
3. Uterine Enlargement/Uterine & Ovarian & Bladder Tumour
4. ParaPlegia/Multiple Sclerosis
🔶D. InteroExternal Haemorrhoids (Mixed) ➡️ External Extension Of Internal Haemorrhoids To Involve The Superior & Inferior Haemorrhoidal Plexuses
#Haemorrhoids
#part 2
#surgery
https://t.me/Surgery_Practice
❤1👍1
The most common site of amoebic liver abscess is in
Anonymous Quiz
26%
The anterior part of dome of Rt. lobe
58%
The posteior part of dome of Rt. lobe
8%
Tha anterior part of dome of Lt. lobe
8%
The posteior part of dome of Lt. lobe
A case of very large amoebic liver abscess with severe toxemia that hasn't responded to conservative ttt for 5 days, what is the next step for management?
Anonymous Quiz
61%
Aspiration
33%
Open drainage surgery
0%
Hepatic resection
6%
Endoscope & biopsy
The most common tumor like condition is ..
Anonymous Quiz
21%
Liver cell adenoma (LCA)
30%
Focal nodular hyperplasia (FNH)
48%
Hemangioma
0%
Biliary cystadenoma
Which of the following is FALSE about hemangioma ?
Anonymous Quiz
45%
Complications are the commonest presentation.
14%
May rupture causing serious bleeding.
21%
Asymptomatic patients need no treatment.
21%
Feeding artery occlusion is done for large tumors.
The gold standard investigation for liver cirrhosis is
Anonymous Quiz
13%
US
74%
Liver biopsy
13%
Liver function tests
0%
Endoscope
Micro nodular cirrhosis is characterized by:
Anonymous Quiz
21%
Regenerating nodules are < 3 Cm
14%
Uniform involvement of liver
21%
None of the above
43%
Both
Macronodular liver cirrhosis often caused by:
Anonymous Quiz
15%
Alcohol
4%
Biliary tract disease
19%
Chronic viral hepatitis
63%
All of the above
Which of the following drugs is used in 1ry prevention of variceal hge?
Anonymous Quiz
15%
Terlipressin
15%
Somatostatin
18%
Octreotide
53%
Propranolol
Regarding the use of non-selective beta-blockers in 1ry prevention of variceal hge, which of the following is FALSE?
Anonymous Quiz
10%
Reduces the possibility of bleeding by 50%
10%
May be associated with increased survival
20%
Cost-effective
60%
Indicated for severely asthmatic patients with cirrhosis
Initial step for management of bleeding esophageal varices
Anonymous Quiz
20%
Urgent endoscopy
7%
Sclerotherapy band ligation
63%
Resuscitation
7%
Terlipressin
3%
Sengstaken tube
Which of the following is FALSE about resuscitation of acute variceal hge?
Anonymous Quiz
27%
Over-correction of hypovolemia is preferred due to the diminished baroreceptor reflexes in cirrhosis
35%
Target blood Hb of 8 g/L is sufficient to prevent rebleeding
19%
Ascitic tap is carried out
15%
Prophylactic antibiotics
4%
IV thiamine is given in cases of alcohol withdrawal
Which of the following is TRUE about Terlipressin?
Anonymous Quiz
16%
Induces splanchnic arterial dilatation
24%
Used in 1ry prevention of variceal hge
20%
Suitable for patients with ischemic heart disease
40%
The only vasoconstrictor proved to ⬇️ mortality
The main role of vasoconstrictive therapy in management of variceal hge is:
Anonymous Quiz
26%
The 1st line ttt & the most effective single therapeutic modality in acute variceal hge
26%
Adjuvant to endoscopic ttt
7%
1ry prevention of variceal hge
41%
1 & 3
Which of the following is FALSE about Sangstaken-Blakemore tube?
Anonymous Quiz
23%
Indicated if endoscopic ttt has failed in management of severe acute variceal hge
15%
Shouldn't be left in place for more than 12 days
38%
Should be placed in close opposition to GEJ to ❌ cephalad varcieal flow to the bleeder
19%
Esophageal balloon is inflated only if gastric balloon failed to stop bleeding alone
4%
Hemostasis is achieved in 90% of cases
Complications of balloon temponade do NOT include:
Anonymous Quiz
19%
Aspiration pneumonia
12%
Esophageal rupture
15%
Mucosal ulceration
54%
Significant ⬆️ risk of developing hepatic encephalopathy
Which of the following is FALSE about management of acute variceal hge?
Anonymous Quiz
22%
The patient should remain NPO til bleeding stops
26%
PPIs is preferred over ranitidine in ⬇️ acid secretion
13%
Prevention of encephalopthy is crucial; specially in cases with large bleed
39%
Sucralfate is used after endoscopic therapy to reduce esophageal ulceration
The 1st ttt option in management of acute rebleed is:
Anonymous Quiz
48%
Endoscopy
21%
TIPS
7%
Balloon temponade
24%
Surgery (Esophageal transection & ligation of the feeding vessels to the bleeding varices)