Forwarded from Review team | Internal medicine (Wafaa Sodagar)
تلبيةً لطلب العديد من متابعي فريق Review Team
نُعلن عن إعادة رفع الفيديوهات المسجلة لدورة اختبار SMLE للعام الماضي 2021
*SMLE course 2021*
والتي سيتم إتاحة مشاهدتها على قناة اليوتيوب الخاصة بالفريق من خلال روابط خاصة سوف ترسل على قناة التليجرام، سيتم فتح كل رابط لمدة ٤٨ ساعه ثم إعادة إغلاقه
🔴خلال شهر August 1-16
أما بالنسبة للكورس الجديد لهذا العام سيُعلن عنه في سبتمبر 2022 بإذن الله
مع تمنياتنا لكم بالتوفيق والنجاح ✨
نُعلن عن إعادة رفع الفيديوهات المسجلة لدورة اختبار SMLE للعام الماضي 2021
*SMLE course 2021*
والتي سيتم إتاحة مشاهدتها على قناة اليوتيوب الخاصة بالفريق من خلال روابط خاصة سوف ترسل على قناة التليجرام، سيتم فتح كل رابط لمدة ٤٨ ساعه ثم إعادة إغلاقه
🔴خلال شهر August 1-16
أما بالنسبة للكورس الجديد لهذا العام سيُعلن عنه في سبتمبر 2022 بإذن الله
مع تمنياتنا لكم بالتوفيق والنجاح ✨
Review team | Internal medicine
تلبيةً لطلب العديد من متابعي فريق Review Team نُعلن عن إعادة رفع الفيديوهات المسجلة لدورة اختبار SMLE للعام الماضي 2021 *SMLE course 2021* والتي سيتم إتاحة مشاهدتها على قناة اليوتيوب الخاصة بالفريق من خلال روابط خاصة سوف ترسل على قناة التليجرام، سيتم فتح…
هذا كورس أم القرى الله يجزاهم ألف خير ❤️
A male patient who is a known alcoholic for many years presents with abdominal pain, vomiting and hematemesis. He was recently managed as a case of perforated peptic ulcer. On exam, there is epigastric? tenderness. Liver enzymes are normal. Diagnosis is
A- Acute pancreatitis
B- Chronic pancreatitis
C- Varices
D- Mallory-weiss
Abdulrahman: The answer is D, due to his history of alcohol use, abdominal pain, and repeated vomiting which lead to the hematemesis. If the answer was varices, he would at least have some signs and symptoms of liver failure or will present first with hematemesis.
A- Acute pancreatitis
B- Chronic pancreatitis
C- Varices
D- Mallory-weiss
Abdulrahman: The answer is D, due to his history of alcohol use, abdominal pain, and repeated vomiting which lead to the hematemesis. If the answer was varices, he would at least have some signs and symptoms of liver failure or will present first with hematemesis.
indications for oxygen home therapy COPD-32
A- 6.3
B- 7.9
C- 7.3
D- 8.3
Better recall: Which of these is an indication for home oxygen therapy in COPD?
A- 1 reading of Po2 <7.8pka
B- 2 readings of Po2 <7.3
C- 1 reading of Po2 <8
D- 2 readings of Po2<6.3
Abdulrahman: The answer is two readings Po2 <7.3. Here are the other indications for long term home oxygen therapy in COPD patient:
1- PaO2 in ABG (out of time of acute attack) less than 55
2- PaO2 55-59 + one of the following: dependent edema (Cor pulmonale), polycythemia (Hct > 56%), pulmonary hypertension, Nocturnal hypoxemia
A- 6.3
B- 7.9
C- 7.3
D- 8.3
Better recall: Which of these is an indication for home oxygen therapy in COPD?
A- 1 reading of Po2 <7.8pka
B- 2 readings of Po2 <7.3
C- 1 reading of Po2 <8
D- 2 readings of Po2<6.3
Abdulrahman: The answer is two readings Po2 <7.3. Here are the other indications for long term home oxygen therapy in COPD patient:
1- PaO2 in ABG (out of time of acute attack) less than 55
2- PaO2 55-59 + one of the following: dependent edema (Cor pulmonale), polycythemia (Hct > 56%), pulmonary hypertension, Nocturnal hypoxemia
had prosthetic valve disease since 10 days. Underwent dental procedure and developed infective endocarditis. What's the organism caused infective endocarditis?
A- Staph aureus
B- Staph epidermis
C- Strept viridians
Abdulrahman: The answer is C. Answers vary between references as to what are the organisms that cause infective endocarditis. I’ve decided on what is found Davidson’s and Harrison’s books (approved references by SCFHCS):
A- Staph aureus
B- Staph epidermis
C- Strept viridians
Abdulrahman: The answer is C. Answers vary between references as to what are the organisms that cause infective endocarditis. I’ve decided on what is found Davidson’s and Harrison’s books (approved references by SCFHCS):
Child has inflammation in hemiscrotum upon exploration there was viable testes but inflammation in epididymis and cord and swelling what the dx:
A- epididymitis
B- incarcerated inguinal hernia
C- testicular torsion
D- not remembered
Another recall: unilateral testicular painful swelling since 1 day, on exploration red scrotum viable but cord edematous:
A- Torsion
B- Incarcerated inguinal hernia
C- Testicular appendages torsion
D- epididymitis
Abdulrahman: The answer is epididymoorchitis/epididymitis.
- Epididymitis: Inflamed scrotum and edema
- Appendiceal testis torsion: Vertical testicle and blue dot sign
- Testicular torsion: Horizontal testicle
- Incarcerated inguinal hernia: Inflamed and red inguinal area with a visible mass extending to the scrotum
A- epididymitis
B- incarcerated inguinal hernia
C- testicular torsion
D- not remembered
Another recall: unilateral testicular painful swelling since 1 day, on exploration red scrotum viable but cord edematous:
A- Torsion
B- Incarcerated inguinal hernia
C- Testicular appendages torsion
D- epididymitis
Abdulrahman: The answer is epididymoorchitis/epididymitis.
- Epididymitis: Inflamed scrotum and edema
- Appendiceal testis torsion: Vertical testicle and blue dot sign
- Testicular torsion: Horizontal testicle
- Incarcerated inguinal hernia: Inflamed and red inguinal area with a visible mass extending to the scrotum
22 month infant with LEFT inflamed red hemi scrotum, on examination there was red firm irreducible painful scrotal swelling which is extended to to left inguinal region. LEFT testis couldn't be palpated. What is the diagnosis?
A. Torsion appendix testis.
B. testicular torsion
C. epididymo orchitis.
D. incarcerated inguinal hernia
Abdulrahman: The answer is D.
A. Torsion appendix testis.
B. testicular torsion
C. epididymo orchitis.
D. incarcerated inguinal hernia
Abdulrahman: The answer is D.
8 years old Female pt brought by her mother to the emergency department with nausea and vomiting after ingestion of 2 packages of paracetamol, each package contains 20 tables, this is happened before 24 hours after fight with her mother. On examination pt looks jaundiced has right hypochondrial tenderness, Bp=90/60mmhg HR=86, no labs provided. What is the stage of paracetamol toxicity?
A-1
B-2
C-3
D-4
Abdulrahman: The answer is B. The patient ingested a toxic amount of paracetamol before 24 hours, has RUQ pain, and early signs of abnormal LFT (jaundiced). Stage 3 will present with much more severe symptoms (encephalopathy, renal failure, acidosis, coagulopathy).
A-1
B-2
C-3
D-4
Abdulrahman: The answer is B. The patient ingested a toxic amount of paracetamol before 24 hours, has RUQ pain, and early signs of abnormal LFT (jaundiced). Stage 3 will present with much more severe symptoms (encephalopathy, renal failure, acidosis, coagulopathy).
A 2 year old child presented with fever jaundice and a (third thing). Family history: his brother had neonatal jaundice and required multiple blood transfusions. They are from the Mediterranean. temp 38, RBC 4, Hbg 9,) Reticulocyte(3%) Bleeding. Which medication should be avoided in the management?
A- Aspirin
B- amoxicillin
C- ibuprofen
D- acetaminophen
Abdulrahman: The answer is A since the patient most likely has G6PD deficiency (jaundice, family history, and from a Mediterranean region). People always get this question mixed up. Just because the patient is from a Mediterranean area, does not mean he has Mediterranean fever.
- Avoid aspirin in G6PD deficiency.
- Avoid Amoxicillin in familial Mediterranean fever. Macrolides are a better choice if it is in the choices.
A- Aspirin
B- amoxicillin
C- ibuprofen
D- acetaminophen
Abdulrahman: The answer is A since the patient most likely has G6PD deficiency (jaundice, family history, and from a Mediterranean region). People always get this question mixed up. Just because the patient is from a Mediterranean area, does not mean he has Mediterranean fever.
- Avoid aspirin in G6PD deficiency.
- Avoid Amoxicillin in familial Mediterranean fever. Macrolides are a better choice if it is in the choices.
Female patient just delivered a baby weighing 3000g. Which of the following is a risk factor for developing postpartum hemorrhage?
A- Fetal Macrosomia
B- Precipitous labour
C- Grand parity
Better recall: 35 y.o , G4p3L2 , medically free, presented in labor which take 16 hrs to deliver a 3 kg baby boy healthy, after delivering the placenta the patient had large amount of bleeding , what could be the cause?
A- grand parity
B- long duration of labor
C- macrosomic baby
D- multiple gestation
Abdulrahman: The answer is long duration of labor. Let’s discuss the choices:
- Grand multiparity: patients who have had ≥5 births (live or stillborn) at ≥20 weeks of gestation.
- Long duration of labor: Prolonged labor requires that, in the face of regular uterine contractions, the cervical dilation is <6 cm for a duration of >20 h in a primipara or >14 h in a multipara. in this case, it is 16 hrs in a multipara women, which is abnormal.
- Marcoscomic baby: 3000 g (which is normal). Macrosomia is >4000-4500g
- Multiple gestation: The patient is delivering one baby only. There is no mentioning of twins.
A- Fetal Macrosomia
B- Precipitous labour
C- Grand parity
Better recall: 35 y.o , G4p3L2 , medically free, presented in labor which take 16 hrs to deliver a 3 kg baby boy healthy, after delivering the placenta the patient had large amount of bleeding , what could be the cause?
A- grand parity
B- long duration of labor
C- macrosomic baby
D- multiple gestation
Abdulrahman: The answer is long duration of labor. Let’s discuss the choices:
- Grand multiparity: patients who have had ≥5 births (live or stillborn) at ≥20 weeks of gestation.
- Long duration of labor: Prolonged labor requires that, in the face of regular uterine contractions, the cervical dilation is <6 cm for a duration of >20 h in a primipara or >14 h in a multipara. in this case, it is 16 hrs in a multipara women, which is abnormal.
- Marcoscomic baby: 3000 g (which is normal). Macrosomia is >4000-4500g
- Multiple gestation: The patient is delivering one baby only. There is no mentioning of twins.
man had recurrent episodes of arthritis in his big toe, he diagnosed with gout and he is k/c of psoriasis. He is compliant to gout medications but with no improvement and high uric acid level. Now he presented bilateral ankle arthritis, the diagnosis is?
A- Pseudogout arthritis
B- Gout arthritis
C- Psoriasisarthritis ( not sure)
Better recall: patient who had right big toe pain and because of that “written like this” he was diagnosed with gout and treated with allopurinol. He’s a known case of psoriasis. He presented now with right big toe pain, right ankle pain and bilateral knee pain and there was no improvement with allopurinol. Labs shows high uric acid What’s the diagnosis?
A- active pseudo-gout arthritis
B- active gout arthritis
C- psoriasis arthritis
D- Osteoarthritis
Abdulrahman: I think the answer is psoriatic arthritis since he almost fulfills the criteria for psoriatic arthritis (CASPAR) and might be missing more important info in the question. Even though he has been diagnosed with gout, he has not been responding to his medications. He has an established inflammatory articular disease + history of psoriasis (1 point) + dactylitis (1 point). We are missing 1 point to confirm the diagnosis. The symmetrical joint pain (bilateral knee pain) is what is throwing me off also. I hope the full question will be much clearer for you.
Update: Another point of view from an excellent person: Psoriasis is a risk factor for secondary hyperuricemia. The clinical presentation for arthritis involvement goes towards gout + it is a recurrent episode, so it is common to see multiple joints. Based on the recall, the Q mentioned that he took allopurinol during the active episode so I believe this is why he didn’t improve because it doesn’t have an effect unlike NSAID or steroid rapid relief. As you can see in the pic from uptodate I feel the scenario fits it as a recurrent episode for an active gout. Although I don’t agree with bilateral knees. But the ankle involvement is common in recurrent episodes of gout.
A- Pseudogout arthritis
B- Gout arthritis
C- Psoriasisarthritis ( not sure)
Better recall: patient who had right big toe pain and because of that “written like this” he was diagnosed with gout and treated with allopurinol. He’s a known case of psoriasis. He presented now with right big toe pain, right ankle pain and bilateral knee pain and there was no improvement with allopurinol. Labs shows high uric acid What’s the diagnosis?
A- active pseudo-gout arthritis
B- active gout arthritis
C- psoriasis arthritis
D- Osteoarthritis
Abdulrahman: I think the answer is psoriatic arthritis since he almost fulfills the criteria for psoriatic arthritis (CASPAR) and might be missing more important info in the question. Even though he has been diagnosed with gout, he has not been responding to his medications. He has an established inflammatory articular disease + history of psoriasis (1 point) + dactylitis (1 point). We are missing 1 point to confirm the diagnosis. The symmetrical joint pain (bilateral knee pain) is what is throwing me off also. I hope the full question will be much clearer for you.
Update: Another point of view from an excellent person: Psoriasis is a risk factor for secondary hyperuricemia. The clinical presentation for arthritis involvement goes towards gout + it is a recurrent episode, so it is common to see multiple joints. Based on the recall, the Q mentioned that he took allopurinol during the active episode so I believe this is why he didn’t improve because it doesn’t have an effect unlike NSAID or steroid rapid relief. As you can see in the pic from uptodate I feel the scenario fits it as a recurrent episode for an active gout. Although I don’t agree with bilateral knees. But the ankle involvement is common in recurrent episodes of gout.
Male patient complains of episode of hematemesis. normal past medical history Labs show mild anemia ,All over labs are normal except elevated urea level in blood?
A-Mallory Weiss syndrome
B-erosive gastritis
C-PUD
Better recall: 40 years old adult male medically free presented to ER C/O several episodes of hematemesis for the first time, takes no medication, no family history of similar attacks, normal abdominal examination no guarding no tenderness.
Hgb: 9
Plt: 250
Alk: slightly decrease
All other tests were normal
What is the diagnosis:
A-Erosive gastritis
B-eptic ulcer disease
C-Mallory Weiss syndrome
D-Esophageal varices
Another recall: 30 year old male, presented to ER with several episodes of hematemesis.
No similar previous episodes, not on any medications, no alcohol intake, no smoking, no liver disease/jaundice.
Labs:
Normal ALT
Low ALP
BUN 14 (high)
(Nothing else of significance based on my memory)
Abdulrahman: Even though we have a lot of info in this question, it is still vague. I will go with Erosive gastritis, but you need to check for other clues when you get the full question:
- Varices: No history of liver disease/jaundice (normal labs) or alcohol intake
- Erosive gastritis and PUD: Clinical signs and symptoms may be similar. High urea could be a sign of H-pylori. if the anemia is a B12 anemia, then consider erosive gastritis.
- Mallory-Wiss syndrome: will present with multiple episodes of vomiting then the hematemesis will occur.
A-Mallory Weiss syndrome
B-erosive gastritis
C-PUD
Better recall: 40 years old adult male medically free presented to ER C/O several episodes of hematemesis for the first time, takes no medication, no family history of similar attacks, normal abdominal examination no guarding no tenderness.
Hgb: 9
Plt: 250
Alk: slightly decrease
All other tests were normal
What is the diagnosis:
A-Erosive gastritis
B-eptic ulcer disease
C-Mallory Weiss syndrome
D-Esophageal varices
Another recall: 30 year old male, presented to ER with several episodes of hematemesis.
No similar previous episodes, not on any medications, no alcohol intake, no smoking, no liver disease/jaundice.
Labs:
Normal ALT
Low ALP
BUN 14 (high)
(Nothing else of significance based on my memory)
Abdulrahman: Even though we have a lot of info in this question, it is still vague. I will go with Erosive gastritis, but you need to check for other clues when you get the full question:
- Varices: No history of liver disease/jaundice (normal labs) or alcohol intake
- Erosive gastritis and PUD: Clinical signs and symptoms may be similar. High urea could be a sign of H-pylori. if the anemia is a B12 anemia, then consider erosive gastritis.
- Mallory-Wiss syndrome: will present with multiple episodes of vomiting then the hematemesis will occur.
39 y / o Patient with symptoms of SLE, arthritis and malar rash, what medication should be started ?
A- Hydroxychloroquine and Mycophenolate mofetil
B- Hydroxychloroquine and Methotrexate and Steroid
C- Hydroxychloroquine
D- Methotrexate
Abdulrahman: The answer is Hydroxychloroquine and steroids. Arthritis in patients with SLE usually responds to NSAIDs or hydroxychloroquine. Patients with arthralgia may benefit from acetaminophen. Glucocorticoids may be added to control arthritis in patients whose symptoms have not resolved while on antimalarials and/or NSAIDs.
- Mild-moderate disease (skin and joints involvement): NSAIDs + HCQ (if failed low dose steroids + MTX)
- Moderate - severe (CNS, cardiac, renal): High dose steroids + cyclosporine
Here is a different but similar question from Umm Al-Qura:
A- Hydroxychloroquine and Mycophenolate mofetil
B- Hydroxychloroquine and Methotrexate and Steroid
C- Hydroxychloroquine
D- Methotrexate
Abdulrahman: The answer is Hydroxychloroquine and steroids. Arthritis in patients with SLE usually responds to NSAIDs or hydroxychloroquine. Patients with arthralgia may benefit from acetaminophen. Glucocorticoids may be added to control arthritis in patients whose symptoms have not resolved while on antimalarials and/or NSAIDs.
- Mild-moderate disease (skin and joints involvement): NSAIDs + HCQ (if failed low dose steroids + MTX)
- Moderate - severe (CNS, cardiac, renal): High dose steroids + cyclosporine
Here is a different but similar question from Umm Al-Qura: