🖊 Acute Mastoiditis | SOMedEd
🦻 Acute mastoiditis is the most common suppurative complication of acute otitis media.
It presents with fever, postauricular tenderness/swelling, a bulging tympanic membrane, and outward displacement of the ear.
📌 Consider mastoiditis in any child with acute otitis media who develops tenderness behind the ear.
🦻 Acute mastoiditis is the most common suppurative complication of acute otitis media.
It presents with fever, postauricular tenderness/swelling, a bulging tympanic membrane, and outward displacement of the ear.
📌 Consider mastoiditis in any child with acute otitis media who develops tenderness behind the ear.
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Forwarded from Black Lion | School of Medicine 💉💊
Is vitamin toxicity more likely with water-soluble or fat-soluble vitamins?
Anonymous Quiz
31%
Water-soluble vitamins
69%
Fat-soluble vitamins
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Children aged 6–24 months are predisposed to OME due to:
Anonymous Quiz
9%
Immature cochlear development
10%
Increased mastoid air cell formation
76%
Narrow poorly draining eustachian tubes
5%
Reduced middle ear vascular supply
Pediatrics Questions Channel 💉💊
SOMedEd
Initial management of uncomplicated OME is:
Anonymous Quiz
24%
Urgent IV antibiotics
8%
Immediate myringotomy
66%
Observation with follow-up
2%
Systemic corticosteroids
Key differentiating feature of peritonsillar abscess from tonsillitis:
Anonymous Quiz
1%
Cough
61%
Trismus
6%
Rhinorrhea
31%
Bilateral tonsillar swelling
A child with “hot potato voice”, drooling, uvular deviation, and trismus most likely has:
Anonymous Quiz
6%
Acute tonsillitis
41%
Peritonsillar abscess
41%
Epiglottitis
12%
Retropharyngeal abscess
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🧠 Eustachian Tube in Young Children | SOMedEd
In young children, the Eustachian tube is short, narrow, and more horizontal. This causes poor drainage and easy blockage, especially after viral URIs or AOM. Fluid then gets trapped in the middle ear, leading to OME.
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Which of the following best describes sickle cell trait?
Anonymous Quiz
19%
Severe hemolytic anemia with low HbA
8%
Markedly decreased RBC indices and reticulocytosis
14%
Always associated with splenomegaly
59%
Usually asymptomatic with HbA > HbS on electrophoresis
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Vesicoureteral reflux (VUR)
Anonymous Quiz
17%
Retrograde flow of urine from bladder to ureter due to urethral obstruction
70%
Retrograde flow of urine from bladder to ureter due to congenital UVJ defect
6%
Antegrade flow of urine from kidney to bladder due to infection
6%
Incomplete bladder emptying due to neurogenic bladder
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Pediatrics Questions Channel 💉💊
Vesicoureteral reflux (VUR)
Most specific for diagnosing vesicoureteral reflux?
Anonymous Quiz
5%
Urinalysis
12%
Renal ultrasound
79%
Voiding cystourethrogram
4%
CT abdomen
Pediatrics Questions Channel 💉💊
Most specific for diagnosing vesicoureteral reflux?
A child with suspected VUR undergoes imaging. Which finding confirms the diagnosis?
Anonymous Quiz
16%
Bilateral renal enlargement on ultrasound
80%
Reflux of contrast into ureters during voiding
1%
Proteinuria on dipstick
3%
Reduced glomerular filtration rate
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Infant with 3 - 5 weeks of age presents with nonbilious projectile vomiting after feeds. Which acid–base disturbance is most likely?
Anonymous Quiz
10%
Hyperchloremic metabolic acidosis
79%
Hypochloremic hypokalemic metabolic alkalosis
6%
Respiratory acidosis
5%
High anion gap metabolic acidosis
👍2
A patient with hematuria after infection and low C3 most likely has which condition?
Anonymous Quiz
8%
IgA nephropathy
9%
Focal segmental glomerulosclerosis
78%
Poststreptococcal glomerulonephritis
5%
Minimal change disease
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First-line treatment for central precocious puberty is:
Anonymous Quiz
20%
Dopamine agonists
50%
GnRH agonists
26%
Aromatase inhibitors
4%
Glucocorticoids
Central precocious puberty results from early activation of the:
Anonymous Quiz
71%
Hypothalamic-pituitary-gonadal axis
21%
Hypothalamic-pituitary-adrenal axis
2%
Renin-angiotensin system
5%
Hypothalamic-pituitary-thyroid axis
Early estrogen/testosterone exposure in central precocious puberty leads to:
Anonymous Quiz
13%
Delayed bone age
73%
Premature epiphyseal closure
7%
Decreased bone maturation
7%
Prolonged linear growth
Type I Von Gierke disease is caused by deficiency of:
Anonymous Quiz
18%
Branching enzyme
71%
Glucose-6-phosphatase
9%
Debranching enzyme
3%
Acid maltase
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A child with doll-like facies, hepatomegaly, and fasting hypoglycemia most likely has:
Anonymous Quiz
7%
McArdle disease
55%
Von Gierke disease
24%
Pompe disease
14%
Fabry disease
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