Forwarded from Internal Medicine By Doha Rawag (Doha Ali Rawaq)
Forwarded from Internal Medicine By Doha Rawag (Doha Ali Rawaq)
Forwarded from Internal Medicine By Doha Rawag (Doha Ali Rawaq)
#OSCE
โฝCVS
๐น๏ธAS
Q1_ What're the BP changes could you see in pt with AS ?
โก๏ธLow BP with narrow pulse pressure
Q2_What're the puse changes could you see in pt with AS ?
1_Slow volume ( pulsus parvus)
2_Slow Rising pulse (pulsus tardus)
3_Pulses bisferiens (AS+AR)
Q3_ What's the type of murmur and it's radiation in pt with AS?
โก๏ธ Ejection systolic murmur radiates to the root of neck
Q4_ How to differentiate between AS radiation and Carotid a atherosclerosis?
โก๏ธPhonoangiography
โฝCVS
๐น๏ธAS
Q1_ What're the BP changes could you see in pt with AS ?
โก๏ธLow BP with narrow pulse pressure
Q2_What're the puse changes could you see in pt with AS ?
1_Slow volume ( pulsus parvus)
2_Slow Rising pulse (pulsus tardus)
3_Pulses bisferiens (AS+AR)
Q3_ What's the type of murmur and it's radiation in pt with AS?
โก๏ธ Ejection systolic murmur radiates to the root of neck
Q4_ How to differentiate between AS radiation and Carotid a atherosclerosis?
โก๏ธPhonoangiography
โค2
Forwarded from Internal Medicine By Doha Rawag (Doha Ali Rawaq)
#OSCE
โฝCVS
๐น๏ธApex beat
Q1_ Prescribe the normal apex beat site and character ?
1_lt 5th intercostal space midclvicular line
2_gentle non sustained
Q2_ Mention 2 manoeuvres in case of impalpable?
1_Turn pt to lt lateral position
2_palpate at Rt side
Q3_Mension 3 abnormal character?
1_ Forceful sustained / AS
2_ Forceful unsustained / AR , MR
3_ Tapping / MS
4_ Diffuse / DCM
5_ Double / HOCM
Q3_What valve is responsible for S1 , S2 ?
1_S1 / Closure of M
2_ S2 / Closure of A
Q4_ What signs you should look for in case with Dextrocardia ?
1_ Cyanosis
2_ Clubbing
3_During measuring the liver span โก๏ธThe liver is at Lt side ( if Dextrocardia with situs inversus )
Q5_ Pt with โฌ๏ธ JVP , LL edema , ascitis what could be the cause ?
Rt side heat failure
โฝCVS
๐น๏ธApex beat
Q1_ Prescribe the normal apex beat site and character ?
1_lt 5th intercostal space midclvicular line
2_gentle non sustained
Q2_ Mention 2 manoeuvres in case of impalpable?
1_Turn pt to lt lateral position
2_palpate at Rt side
Q3_Mension 3 abnormal character?
1_ Forceful sustained / AS
2_ Forceful unsustained / AR , MR
3_ Tapping / MS
4_ Diffuse / DCM
5_ Double / HOCM
Q3_What valve is responsible for S1 , S2 ?
1_S1 / Closure of M
2_ S2 / Closure of A
Q4_ What signs you should look for in case with Dextrocardia ?
1_ Cyanosis
2_ Clubbing
3_During measuring the liver span โก๏ธThe liver is at Lt side ( if Dextrocardia with situs inversus )
Q5_ Pt with โฌ๏ธ JVP , LL edema , ascitis what could be the cause ?
Rt side heat failure
โค2๐1
Forwarded from Internal Medicine By Doha Rawag (Doha Ali Rawaq)
#OSCE
โฝGIT
๐น๏ธAscitis
Q1_ What is the mechanism of ascitis in liver cirrhosis?
1_ โฌ๏ธ Hydrostatic pressure
2_ โฌ๏ธ Osmotic pressure ( Hypoalbumenia)
3_ โฌ๏ธ Aldesterone ( renal Na+ retention)
4_ lymphatic obstruction
Q2_ What is the most common organism which cause SBP ?
E.choli
Q3_What are the causes of absent bowl sounds?
1_ Postsurgical paralytic ileus
2_ Peritonitis
Q4_ What is the cause of Hepatomegally + LLO + โฌ๏ธ JVP?
RHF
โฝGIT
๐น๏ธAscitis
Q1_ What is the mechanism of ascitis in liver cirrhosis?
1_ โฌ๏ธ Hydrostatic pressure
2_ โฌ๏ธ Osmotic pressure ( Hypoalbumenia)
3_ โฌ๏ธ Aldesterone ( renal Na+ retention)
4_ lymphatic obstruction
Q2_ What is the most common organism which cause SBP ?
E.choli
Q3_What are the causes of absent bowl sounds?
1_ Postsurgical paralytic ileus
2_ Peritonitis
Q4_ What is the cause of Hepatomegally + LLO + โฌ๏ธ JVP?
RHF
โค2
Forwarded from Internal Medicine By Doha Rawag (Doha Ali Rawaq)
#OSCE
โฌRespiratory
๐น๏ธ Percussion note
Q1_ What are the types of percussion notes + DD ?
๐
1_ Resonace โก๏ธ Normal
2_ Hyper_resonance โก๏ธ Emphysema, Pneumothorax
3_ Dullnessโก๏ธ Collapse, Consolidation
4_ Stony dullness โก๏ธMassive Pl.Eff
Q2_ Where are the sites of normal dullness over the chest on percussion ?
๐
Percussion over soild structures
1_ Liver/ Upper level of liver dullness at Rt fifth inercostal space midclavicular line
2_ Heart / Dullness over the anterior chest
Q3_ What are the examination findings of pt with lt side lung collapse ?
๐
1_ โฌ๏ธ Chest expansion at lt side of chest
2_ โฌ๏ธ TVF at lt side of chest
3_ Pulled Trachea to lt side
4_ Dullness percussion note
5_ โฌ๏ธ air enty at lt side
Q4_ ูู ุง ูุจู ููุญุต ุงู middle lobe
ููู ูุฏูุฑ ุงู percussion ุ
๐
Over the Rt Axilla
Q5_ Who to do chest expansion by percussion?
๐
Tidal percussion
Q6_ What is the type of percussion in pt with pl.eff ?
๐
Stony dullness.
โฌRespiratory
๐น๏ธ Percussion note
Q1_ What are the types of percussion notes + DD ?
๐
1_ Resonace โก๏ธ Normal
2_ Hyper_resonance โก๏ธ Emphysema, Pneumothorax
3_ Dullnessโก๏ธ Collapse, Consolidation
4_ Stony dullness โก๏ธMassive Pl.Eff
Q2_ Where are the sites of normal dullness over the chest on percussion ?
๐
Percussion over soild structures
1_ Liver/ Upper level of liver dullness at Rt fifth inercostal space midclavicular line
2_ Heart / Dullness over the anterior chest
Q3_ What are the examination findings of pt with lt side lung collapse ?
๐
1_ โฌ๏ธ Chest expansion at lt side of chest
2_ โฌ๏ธ TVF at lt side of chest
3_ Pulled Trachea to lt side
4_ Dullness percussion note
5_ โฌ๏ธ air enty at lt side
Q4_ ูู ุง ูุจู ููุญุต ุงู middle lobe
ููู ูุฏูุฑ ุงู percussion ุ
๐
Over the Rt Axilla
Q5_ Who to do chest expansion by percussion?
๐
Tidal percussion
Q6_ What is the type of percussion in pt with pl.eff ?
๐
Stony dullness.
โค2
Forwarded from Internal Medicine By Doha Rawag (Doha Ali Rawaq)
#OSCE
โฌCNC
๐น๏ธCerebellum
Q1_ What is the definition of Apraxia ?
๐
Apraxia is a neurological disorder characterized by the inability to perform learned (familiar) movements on command, even though the command is understood and there is a willingness to perform the movement.
Q2_ What are the types of apraxia ?
๐
1_ melokinetic (or limbโkinetic)
2_ ideomotor apraxia
3_ ideational apraxia
4_ Conceptual apraxia
5_ Buccofacial apraxia
6_ Constructional apraxia
7_ Oculomotor apraxia
8_ Verbal apraxia
Q3_ How to examine constructional apraxia ?
โฌ๏ธ
By writing words and numbers.
โฌCNC
๐น๏ธCerebellum
Q1_ What is the definition of Apraxia ?
๐
Apraxia is a neurological disorder characterized by the inability to perform learned (familiar) movements on command, even though the command is understood and there is a willingness to perform the movement.
Q2_ What are the types of apraxia ?
๐
1_ melokinetic (or limbโkinetic)
2_ ideomotor apraxia
3_ ideational apraxia
4_ Conceptual apraxia
5_ Buccofacial apraxia
6_ Constructional apraxia
7_ Oculomotor apraxia
8_ Verbal apraxia
Q3_ How to examine constructional apraxia ?
โฌ๏ธ
By writing words and numbers.
๐2
Forwarded from Internal Medicine By Doha Rawag (Doha Ali Rawaq)
ุดุฑุญ ู
ูุตู ููุฃููุงุน ๐ก
1_Limb-kinetic apraxia
๐
Pt is unable to use a finger, arm, or leg to make precise and coordinated movements. Although people with limb-kinetic apraxia may understand how to use a tool, such as a screwdriver, and may have used it in the past, they are now unable to carry out the same movement.
ู ุชูุงู ู ุด ูุงุฏุฑ ูุญุฑู ูุฏูู ููุชุญ ู ูุตูุฑ .
2_Ideomotor apraxia
๐
Pt is unable to follow a verbal command to copy the movements of others or follow suggestions for movements.
ู ุด ูุงุฏุฑ ูููุฏ ุญุฑูุฉ.
3_Conceptual apraxia
๐
similar to ideomotor apraxia. Pt is also unable to perform tasks that involve more than one step.
ู ุด ูุงุฏุฑ ูููุฏ ู ุฌู ูุนุฉ ู ู ุงูุญุฑูุงุช .
4_Ideational apraxia
๐
Pt is unable to plan a particular movement. They may find it hard to follow a sequence of movements, such as getting dressed or bathing.
ู ุด ูุงุฏุฑ ููุจุณ ู ูุงุจุณู
5_Buccofacial apraxia
๐
Pt is unable to make movements with the face and lips on command.
ู ุด ูุงุฏุฑ ูุญุฑู ุนุถูุงุช ูุฌูู
6_Constructional apraxia
๐
Pt is unable to copy, draw, or construct basic diagrams or figures.
ู ุด ูุงุฏุฑ ูููุฏ ุฑุณู ุฉ ุงู ููุชุจ ุญุฑู
7_Oculomotor apraxia
๐
Pt have difficulty making eye movements on command.
ู ุด ูุงุฏุฑ ูุญุฑู ุนุถูุงุช ุนูููู
8_Verbal apraxia
๐
Pt find it challenging to make the movements necessary for speech. They may have problems producing sounds and understanding rhythms of speech.
ู ุด ูุงุฏุฑ ูุญุฑู ุงูุนุถูุงุช ุงูู ุณุคููุฉ ุนูู ุงูุตูุช ุจุดูู ุณููู ุ ููุจุฏุง ุตูุชู ู ุด ูุงุถุญ ู ููุงู ู ู ุด ู ูููู .
1_Limb-kinetic apraxia
๐
Pt is unable to use a finger, arm, or leg to make precise and coordinated movements. Although people with limb-kinetic apraxia may understand how to use a tool, such as a screwdriver, and may have used it in the past, they are now unable to carry out the same movement.
ู ุชูุงู ู ุด ูุงุฏุฑ ูุญุฑู ูุฏูู ููุชุญ ู ูุตูุฑ .
2_Ideomotor apraxia
๐
Pt is unable to follow a verbal command to copy the movements of others or follow suggestions for movements.
ู ุด ูุงุฏุฑ ูููุฏ ุญุฑูุฉ.
3_Conceptual apraxia
๐
similar to ideomotor apraxia. Pt is also unable to perform tasks that involve more than one step.
ู ุด ูุงุฏุฑ ูููุฏ ู ุฌู ูุนุฉ ู ู ุงูุญุฑูุงุช .
4_Ideational apraxia
๐
Pt is unable to plan a particular movement. They may find it hard to follow a sequence of movements, such as getting dressed or bathing.
ู ุด ูุงุฏุฑ ููุจุณ ู ูุงุจุณู
5_Buccofacial apraxia
๐
Pt is unable to make movements with the face and lips on command.
ู ุด ูุงุฏุฑ ูุญุฑู ุนุถูุงุช ูุฌูู
6_Constructional apraxia
๐
Pt is unable to copy, draw, or construct basic diagrams or figures.
ู ุด ูุงุฏุฑ ูููุฏ ุฑุณู ุฉ ุงู ููุชุจ ุญุฑู
7_Oculomotor apraxia
๐
Pt have difficulty making eye movements on command.
ู ุด ูุงุฏุฑ ูุญุฑู ุนุถูุงุช ุนูููู
8_Verbal apraxia
๐
Pt find it challenging to make the movements necessary for speech. They may have problems producing sounds and understanding rhythms of speech.
ู ุด ูุงุฏุฑ ูุญุฑู ุงูุนุถูุงุช ุงูู ุณุคููุฉ ุนูู ุงูุตูุช ุจุดูู ุณููู ุ ููุจุฏุง ุตูุชู ู ุด ูุงุถุญ ู ููุงู ู ู ุด ู ูููู .
โค2๐1
ุดูุช ู
ุฌู
ุนุฉ ููู ุงูู
ุงูุญุงูุงุช ุงููู ุชุฌู ูู ุงู
ุชุญุงู ุงูููููู ุจุงู DD ู ุงู Inv
ุดูููู ุญูููุฏูู ููุจุง ุจุงุฐู ุงููู ๐ธ๐ธ
ุดูููู ุญูููุฏูู ููุจุง ุจุงุฐู ุงููู ๐ธ๐ธ
โค3๐1
Forwarded from Internal Medicine By Doha Rawag (Doha Ali Rawaq)
Most common findings in clinical exam by Doha.pdf
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โค1๐1
Forwarded from Internal Medicine By Doha Rawag
#General
ูู ูุงูุช ุงููุงููุฏููู jaundice
~ูุณุฃูู ุจุนุฏูุง ุนุงู DD of jaundice ุ
A_per_hepatic / Hemolysis
B_Hepatic / hepatitis , liver cirrhosis and its cases , malignancies
C_pos_thepatic / Stone , PBC , PSC , Cancer head of pancreas
~ุจุนุฏูุง ูุณุงูู ุนูู ุงู investigations ุ
๐ธ๏ธper_hepatic
CBC
Blood film
LDH
Heptoglobulin
Bilirubin
ESR , ANA , Anti ds
( ุนูู ุฎุงุทุฑ ุงู acquired causes of hemolytic anemia )
ู ุจุงูู ุงูุชุญุงููู ุงููู ุฎุงุตุฉ ุจูู ููุน ู ู ุงู congenital causes
HB electrophoresis
Osmotic fragility test
G6P level
๐ธ๏ธHepatic , post_hepatic
LFT
Abdominal US
MRCP
ERCP
ูู ูุงูุช ุงููุงููุฏููู jaundice
~ูุณุฃูู ุจุนุฏูุง ุนุงู DD of jaundice ุ
A_per_hepatic / Hemolysis
B_Hepatic / hepatitis , liver cirrhosis and its cases , malignancies
C_pos_thepatic / Stone , PBC , PSC , Cancer head of pancreas
~ุจุนุฏูุง ูุณุงูู ุนูู ุงู investigations ุ
๐ธ๏ธper_hepatic
CBC
Blood film
LDH
Heptoglobulin
Bilirubin
ESR , ANA , Anti ds
( ุนูู ุฎุงุทุฑ ุงู acquired causes of hemolytic anemia )
ู ุจุงูู ุงูุชุญุงููู ุงููู ุฎุงุตุฉ ุจูู ููุน ู ู ุงู congenital causes
HB electrophoresis
Osmotic fragility test
G6P level
๐ธ๏ธHepatic , post_hepatic
LFT
Abdominal US
MRCP
ERCP
๐2
Forwarded from Internal Medicine By Doha Rawag
#General
ูู ูุงูุช ุงููุงููุฏูู Signs of anemia
~ูุณุฃูู ุจุนุฏูุง ุนูู ุฃููุงุน ุงูุงููู ูุง ู ุงู DD ููู ููุนุ
โก๏ธMicrocytic
1_ Iron deficiency anmia
2_ Thalassemia
3_ Sidroplastic anemia
4_ lead poisoning
5_ Anemia of chronic illness
โก๏ธMacrocytic..
1_ Megaloplastic anemia
2_ Liver disease
3_ Alcholism
4_ Renal failure
โก๏ธNormocytic..
1_ Anemia of chronic illness
2_ hemolytic anemia
~ู ูุณุฃูู ุจุนุฏูุง ุนุงู investigations ุ
ุงูู ุดู ูุจุนุช
CBC
๐ธ๏ธูู ุทูุนุช microcytic
ูุจุนุช
Iron study
Upper and lower git endoscopy ( if old age)
Stool analysis
๐ธ๏ธูู ุทูุนุช macrocytic
ูุจุนุช
Folic acid level
Methylmalonic acid
B12 level
๐ธ๏ธูู ุทูุนุช Normocytic
ูููุฑ ูู hemolytic
ูุจุนุช
Blood film
LDH
Heptoglobulin
Bilirubin
ู ูู ุง ุงูุชุญุงููู ูุงุฏู ุชุงูุฏูู ุงููุง ูุนูุงู hemolytic
ูุจุนุช ุชุญุงููู ุจุงุด ูุนุฑู ุงูุณุจุจ๐
ESR CRP ANA Antids...(ุนูู ุฎุงุทุฑ ุงู acquired causes / eg :- SlE )
ู ุจุงููุณุจุฉ ูู congenital causes ูููู ูู ููุน ุนูุฏู ุชุญููู ุฎุงุต ุจูู ุชูููู .
Osmotic fragility test
G6P level
Hemoglobin electrophoresis
ูู ูุงูุช ุงููุงููุฏูู Signs of anemia
~ูุณุฃูู ุจุนุฏูุง ุนูู ุฃููุงุน ุงูุงููู ูุง ู ุงู DD ููู ููุนุ
โก๏ธMicrocytic
1_ Iron deficiency anmia
2_ Thalassemia
3_ Sidroplastic anemia
4_ lead poisoning
5_ Anemia of chronic illness
โก๏ธMacrocytic..
1_ Megaloplastic anemia
2_ Liver disease
3_ Alcholism
4_ Renal failure
โก๏ธNormocytic..
1_ Anemia of chronic illness
2_ hemolytic anemia
~ู ูุณุฃูู ุจุนุฏูุง ุนุงู investigations ุ
ุงูู ุดู ูุจุนุช
CBC
๐ธ๏ธูู ุทูุนุช microcytic
ูุจุนุช
Iron study
Upper and lower git endoscopy ( if old age)
Stool analysis
๐ธ๏ธูู ุทูุนุช macrocytic
ูุจุนุช
Folic acid level
Methylmalonic acid
B12 level
๐ธ๏ธูู ุทูุนุช Normocytic
ูููุฑ ูู hemolytic
ูุจุนุช
Blood film
LDH
Heptoglobulin
Bilirubin
ู ูู ุง ุงูุชุญุงููู ูุงุฏู ุชุงูุฏูู ุงููุง ูุนูุงู hemolytic
ูุจุนุช ุชุญุงููู ุจุงุด ูุนุฑู ุงูุณุจุจ๐
ESR CRP ANA Antids...(ุนูู ุฎุงุทุฑ ุงู acquired causes / eg :- SlE )
ู ุจุงููุณุจุฉ ูู congenital causes ูููู ูู ููุน ุนูุฏู ุชุญููู ุฎุงุต ุจูู ุชูููู .
Osmotic fragility test
G6P level
Hemoglobin electrophoresis
Forwarded from Internal Medicine By Doha Rawag
#General
**Rheumatology
ุจู ุง ุงูู Hand examination ุนูู ุทูู ุงู DD ุจูููู
DD of small joint arthritis ๐ก
ุจุนุฏ ู ุง ุชูู ู ุฎุทูุงุช ุงูุงูุฒุงู ููุดู ุชุณู ู ุงู DD ู ุงููู ูู ุง
1_RA
2_SLE
3_Psoriatic arthritis
4_Osteoarthritis
5_ Gouty arthritis
~ู ูุณุฃูู ููู ุชุนุฑู ุงูุญุงูุฉactive ู ูุง ูุง ุ
Hx / joint pain ,morning stifness, joint swelling
Ex/ Tenderness , Hotness ,joint Swelling , Rhematoid nodules
Inx / CBC ,ESR ,CRP
ู ุทุจุนุงู ููู ุนูู ุญุณุจ ุดู ุดููุช ูู ุงูุงูุฒุงู ููุดู ุ ูู ููู ุชูุฏุฑูุณ ู ููุชูุณ ุจุชูููู ุงูุชู .
~ ู ูุณุฃูู ุจุนุฏูุง ุนุงู investigationsุ
CBC
ESR
CRP
RF
AntiCCP
ANA ,Antids
Joint aspiration
X,ray
**Rheumatology
ุจู ุง ุงูู Hand examination ุนูู ุทูู ุงู DD ุจูููู
DD of small joint arthritis ๐ก
ุจุนุฏ ู ุง ุชูู ู ุฎุทูุงุช ุงูุงูุฒุงู ููุดู ุชุณู ู ุงู DD ู ุงููู ูู ุง
1_RA
2_SLE
3_Psoriatic arthritis
4_Osteoarthritis
5_ Gouty arthritis
~ู ูุณุฃูู ููู ุชุนุฑู ุงูุญุงูุฉactive ู ูุง ูุง ุ
Hx / joint pain ,morning stifness, joint swelling
Ex/ Tenderness , Hotness ,joint Swelling , Rhematoid nodules
Inx / CBC ,ESR ,CRP
ู ุทุจุนุงู ููู ุนูู ุญุณุจ ุดู ุดููุช ูู ุงูุงูุฒุงู ููุดู ุ ูู ููู ุชูุฏุฑูุณ ู ููุชูุณ ุจุชูููู ุงูุชู .
~ ู ูุณุฃูู ุจุนุฏูุง ุนุงู investigationsุ
CBC
ESR
CRP
RF
AntiCCP
ANA ,Antids
Joint aspiration
X,ray
Forwarded from Internal Medicine By Doha Rawag
#General
**Endocrine
ุงููุงููุฏููู ุญุชููู ูุง ุงู ุง signs of hypo or hyperthyroidism
~ุจุนุฏูุง ูุณุฃูู ุนูู ุงู Causes ุ
๐ธ๏ธูู ูุงูุช hypo
1_Autoimmune/ Hashimoto,s
2_Post_thyrodectomy or radiotherapy
3_Iodine deficiency
4_Drugs SE / lithium, Amiodarone
๐ธ๏ธูู ูุงูุช hyper
1_ Graves
2_ Multinodular goiter
3_ Single toxic nodule
4_ Thyroiditis
~ู ุจุนุฏูุง ูุณุฃูู ุนูู investigations ุ
TFT / Free T3 , T4 , TSH
Neck US
Antibodies ุงููู ุฎุงุตุฉ ุจูู ูุงุญุฏ ู ููู
Anti TSH stimulating hormone antibodies
Anti thyroid peroxidase antibodies
**Endocrine
ุงููุงููุฏููู ุญุชููู ูุง ุงู ุง signs of hypo or hyperthyroidism
~ุจุนุฏูุง ูุณุฃูู ุนูู ุงู Causes ุ
๐ธ๏ธูู ูุงูุช hypo
1_Autoimmune/ Hashimoto,s
2_Post_thyrodectomy or radiotherapy
3_Iodine deficiency
4_Drugs SE / lithium, Amiodarone
๐ธ๏ธูู ูุงูุช hyper
1_ Graves
2_ Multinodular goiter
3_ Single toxic nodule
4_ Thyroiditis
~ู ุจุนุฏูุง ูุณุฃูู ุนูู investigations ุ
TFT / Free T3 , T4 , TSH
Neck US
Antibodies ุงููู ุฎุงุตุฉ ุจูู ูุงุญุฏ ู ููู
Anti TSH stimulating hormone antibodies
Anti thyroid peroxidase antibodies
๐1
Forwarded from Internal Medicine By Doha Rawag
#CNS
โก๏ธMotor examination
ุงูุซุฑ ูุงููุฏููู ู ู ูู ุชุฌู UMNL
~ู ุจุนุฏูุง ูุณุฃูู ุนูู ุงู DD
1_Vascular ( ischemic, hemorrhagic )MCC
2_Tumer
3_Trauma
4_Infection
5_Inflammation_MS
~ู ุจุนุฏูุง ูุณุฃูู ุนูู ุงู investigations ุ
Non contrast CT
MRI
ECG
ECHO
Carotid duplex
CBC
ESR___if high / ูุจุนุช ุญุชู ANA , Antids
RBS , HBA1c
Lipid profile
Coagulation profile
LFT
RFT
CSF examination
Oligoclonal IgG
...........................
โก๏ธCerebellar examination
ุงููุงููุฏููู ุญ ุชููู signs of ataxia
~ูุณุฃูู ุจุนุฏูุง ุนูู ุงู DD ุ
A_Congenital
1_spinocerebellar ataxia
2_freidreich ataxia
3_a beta lipoproteinemia
4_ataxic telagectasia
B_Acquired
1_Trauma
2_tumer
3_infection
4_Inflammation/ MS
5_Vascular ( ischemic, hemorrhagic)
6_Alcohol, phenyton toxicity
7_Autoimmune ( SCC , Celiac)
8_Hypothyroidism
9_Vit B deficiency _ especially B12
~ุจุนุฏูุง ูุณุฃูู ุนูู ุงู investigations ุ
ููุณ ุงููู ููู ุชุฒูุฏ ุนูููู ๐
Vit B 12 level
Blood film
TSH
Free T4
โก๏ธ Crainal nerve examination
ูุง ุงู ุง ูุจูู ุงุฏูุฑูู ูููู ู ู ุฑุงุช ุจุณ ูุจู 7 ุจุฑูุญู ุงู 3 ู 4 ู ุน ุจุนุถ
ู ุงูุฏุณูุดู ุนุงูุงุบูุจ ุญุชููู ุนูู facial nerve palsy ู ุดู ุฃุณุจุงุจูุ
๐ธ๏ธCauses of upper facial nerve palsy
1_ Trauma
2_ Tumer
3_ Infection
4_ Inflammation/ MS
5_ Vascular/ Stroke
ุจุงููุณุจุฉ ูู investigations ููุณูู ู ุชุน ุงู UMNL
๐ธ๏ธCauses of lower facial nerve palsy (Unilateral )
1_ Bells palsy
2_ DM
3_ Post acoustic neuroma surgery
4_ Ramsy hunt syndrome
5_ OM
6_Parotid tumer
๐ธ๏ธCauses of lower facial nerve palsy( Bilateral )
2L , 2 syndrome
1_ leprosy
2_ lyme disease
3_ fisherman syndrome (part of GBS)
4_ Heerfords syndrome (part of sarcidosis)
โก๏ธMotor examination
ุงูุซุฑ ูุงููุฏููู ู ู ูู ุชุฌู UMNL
~ู ุจุนุฏูุง ูุณุฃูู ุนูู ุงู DD
1_Vascular ( ischemic, hemorrhagic )MCC
2_Tumer
3_Trauma
4_Infection
5_Inflammation_MS
~ู ุจุนุฏูุง ูุณุฃูู ุนูู ุงู investigations ุ
Non contrast CT
MRI
ECG
ECHO
Carotid duplex
CBC
ESR___if high / ูุจุนุช ุญุชู ANA , Antids
RBS , HBA1c
Lipid profile
Coagulation profile
LFT
RFT
CSF examination
Oligoclonal IgG
...........................
โก๏ธCerebellar examination
ุงููุงููุฏููู ุญ ุชููู signs of ataxia
~ูุณุฃูู ุจุนุฏูุง ุนูู ุงู DD ุ
A_Congenital
1_spinocerebellar ataxia
2_freidreich ataxia
3_a beta lipoproteinemia
4_ataxic telagectasia
B_Acquired
1_Trauma
2_tumer
3_infection
4_Inflammation/ MS
5_Vascular ( ischemic, hemorrhagic)
6_Alcohol, phenyton toxicity
7_Autoimmune ( SCC , Celiac)
8_Hypothyroidism
9_Vit B deficiency _ especially B12
~ุจุนุฏูุง ูุณุฃูู ุนูู ุงู investigations ุ
ููุณ ุงููู ููู ุชุฒูุฏ ุนูููู ๐
Vit B 12 level
Blood film
TSH
Free T4
โก๏ธ Crainal nerve examination
ูุง ุงู ุง ูุจูู ุงุฏูุฑูู ูููู ู ู ุฑุงุช ุจุณ ูุจู 7 ุจุฑูุญู ุงู 3 ู 4 ู ุน ุจุนุถ
ู ุงูุฏุณูุดู ุนุงูุงุบูุจ ุญุชููู ุนูู facial nerve palsy ู ุดู ุฃุณุจุงุจูุ
๐ธ๏ธCauses of upper facial nerve palsy
1_ Trauma
2_ Tumer
3_ Infection
4_ Inflammation/ MS
5_ Vascular/ Stroke
ุจุงููุณุจุฉ ูู investigations ููุณูู ู ุชุน ุงู UMNL
๐ธ๏ธCauses of lower facial nerve palsy (Unilateral )
1_ Bells palsy
2_ DM
3_ Post acoustic neuroma surgery
4_ Ramsy hunt syndrome
5_ OM
6_Parotid tumer
๐ธ๏ธCauses of lower facial nerve palsy( Bilateral )
2L , 2 syndrome
1_ leprosy
2_ lyme disease
3_ fisherman syndrome (part of GBS)
4_ Heerfords syndrome (part of sarcidosis)
๐1
Forwarded from Internal Medicine By Doha Rawag
#Respiratory
ุงูุซุฑ ูุงููุฏููู ุชุฌู
๐ธ๏ธRhonchi
~ู ูุณุงูู ุจุนุฏูุง ุนูู ุงู DD ุ
1_Asthma
2_COPD
3_Pneumonia
...ect
~ุจุนุฏูุง ูุณุฃูู ุนูู ุงู investigations ุ
ุทุจุนุงู ุฒู ู ุง ู ุญูุธูุง ุณูู
ABG ,pusle oxymetry
CXR
PFT
ู ุชุฒูุฏ ุนูููู ๐
Spirometry , peak flow meter
(ุนูู ุฎุงุทุฑ ุงูุงุฒู ุง )
HRCT
(ุนูู ุฎุงุทุฑ ูู ุนูุฏู ุงู ููุฒูู ุง )
CBC
Sputum culture, blood culture, urine culture
(ุนูู ุฎุงุทุฑ ุงููููู ูููุง )
ุงูู ูู ุงูุฎูุงุตุฉ ุจุชุณู ู ุงูุงููุฒุชูููุดู ุนูู ุญุณุจ ุงู DD
..........................................................................
๐ธ๏ธFine crepitation
~ูุณุฃูู ุจุนุฏูุง ุนุงู DD
1_ILD
2_early pneumonia
3_pulmonary edema as complications of HF
~ ู ูุณุฃูู ุจุนุฏูุง ุนุงู investigations ุ
ููุณ ุงูุดู
ABG
PFT
CXR
ู ุชุฒูุฏ ุนูููู ๐
CBC
ESR , CRP
CTD screening / ANA , Antids
HRCT
BAl
Biopsy
( ุนูู ุฎุงุทุฑ ุงู ILD )
Echo
(ุนูู ุฎุงุทุฑ pulmonary edema )
Sputum culture , blood culture
(ุนูู ุฎุงุทุฑ ุงููููู ูููุง )
...........................................................................
๐ธ๏ธCourse crepitation
~ูุณุฃูู ุจุนุฏูุง ุนุงู DD ุ
1_Bronchiactasis
2_chronic bronchitis
3_late pneumonia
~ ู ูุณุฃูู ุจุนุฏูุง ุนุงู investigations ุ
ููุณ ุงูุดู
ABG , pusle oxymetry
PFT
CXR
ู ุชุฒูุฏ ุนูููู ๐
HRCT
Sputum microbiology
Serology
Sacharin test
Ig level
(ุนูู ุฎุงุทุฑ ุงู Bronchiactasis )
CBC
Sputum culture, blood culture
(ุนูู ุฎุงุทุฑ ุงู ูููู ูููุง )
ุญุงุฌุฉ ุชุงููุฉ ูู ุงูุฑูุณุจูุฑุงุชูุฑู ุ ู ุฑุงุช ุงูููุงูุฏููู ุชููู๐
Decreased air entry
Dullness at lower zones
Chest tube under water seal
~ูุณุฃูู ุจุนุฏูุง ุนูู ุงู DD of dulness ุ
1_Pleural effusion
2_Collapse
3_Consolidation
~ู ูุณุฃูู ุจุนุฏูุง ุนูู ุงู most likely Diagnosis ู ุงููู ูู plural effusion.
~ู ูุณุฃูู ุนูู ุงู causes ุ
โก๏ธA_General
CHF
Nephrotic syndrome
Liver cell failure
โก๏ธB_local
Pneumonia
TB
CTD / SLE , RA
Malignancies
Pulmonary infarction
~ุจุนุฏูุง ูุณุฃูู ุนูู ุงู investigations ุ
CXR
CT scan
US ู ุฑุงุช ุชููุฏูู ุจุณ
ABG , pulse oxymetry
Plurocentesis /ุจุงุด ูุญุฏุฏ ุงูููุน exudative ุงู transudative
ู ุนูู ุฃุณุงุณ ุงูุชุญููู ูุฐุง ุจูุจุนุช ุชุญุงููู ุชุงููุฉ ุจุงุด ูุฒุจุท ุงู cause ุ
ูู ูุงู transudative
ุจูุจุนุช
ECG
Echo
Urine for RE
U/Cr/E
LFT
Abdominal US
ูู ูุงู ุงูุณุจุจ exudative
ููุณ ุงูุดู ุจุชุจุนุช ุชุญุงููู ุจุงุด ุชุณุชุจุนุฏ ูู ุงูุงุณุจุงุจ
CBC
Sputum culture, blood culture
Quantiferon test
ESR , CRP
ANA , Anti ds
ุงูุซุฑ ูุงููุฏููู ุชุฌู
๐ธ๏ธRhonchi
~ู ูุณุงูู ุจุนุฏูุง ุนูู ุงู DD ุ
1_Asthma
2_COPD
3_Pneumonia
...ect
~ุจุนุฏูุง ูุณุฃูู ุนูู ุงู investigations ุ
ุทุจุนุงู ุฒู ู ุง ู ุญูุธูุง ุณูู
ABG ,pusle oxymetry
CXR
PFT
ู ุชุฒูุฏ ุนูููู ๐
Spirometry , peak flow meter
(ุนูู ุฎุงุทุฑ ุงูุงุฒู ุง )
HRCT
(ุนูู ุฎุงุทุฑ ูู ุนูุฏู ุงู ููุฒูู ุง )
CBC
Sputum culture, blood culture, urine culture
(ุนูู ุฎุงุทุฑ ุงููููู ูููุง )
ุงูู ูู ุงูุฎูุงุตุฉ ุจุชุณู ู ุงูุงููุฒุชูููุดู ุนูู ุญุณุจ ุงู DD
..........................................................................
๐ธ๏ธFine crepitation
~ูุณุฃูู ุจุนุฏูุง ุนุงู DD
1_ILD
2_early pneumonia
3_pulmonary edema as complications of HF
~ ู ูุณุฃูู ุจุนุฏูุง ุนุงู investigations ุ
ููุณ ุงูุดู
ABG
PFT
CXR
ู ุชุฒูุฏ ุนูููู ๐
CBC
ESR , CRP
CTD screening / ANA , Antids
HRCT
BAl
Biopsy
( ุนูู ุฎุงุทุฑ ุงู ILD )
Echo
(ุนูู ุฎุงุทุฑ pulmonary edema )
Sputum culture , blood culture
(ุนูู ุฎุงุทุฑ ุงููููู ูููุง )
...........................................................................
๐ธ๏ธCourse crepitation
~ูุณุฃูู ุจุนุฏูุง ุนุงู DD ุ
1_Bronchiactasis
2_chronic bronchitis
3_late pneumonia
~ ู ูุณุฃูู ุจุนุฏูุง ุนุงู investigations ุ
ููุณ ุงูุดู
ABG , pusle oxymetry
PFT
CXR
ู ุชุฒูุฏ ุนูููู ๐
HRCT
Sputum microbiology
Serology
Sacharin test
Ig level
(ุนูู ุฎุงุทุฑ ุงู Bronchiactasis )
CBC
Sputum culture, blood culture
(ุนูู ุฎุงุทุฑ ุงู ูููู ูููุง )
ุญุงุฌุฉ ุชุงููุฉ ูู ุงูุฑูุณุจูุฑุงุชูุฑู ุ ู ุฑุงุช ุงูููุงูุฏููู ุชููู๐
Decreased air entry
Dullness at lower zones
Chest tube under water seal
~ูุณุฃูู ุจุนุฏูุง ุนูู ุงู DD of dulness ุ
1_Pleural effusion
2_Collapse
3_Consolidation
~ู ูุณุฃูู ุจุนุฏูุง ุนูู ุงู most likely Diagnosis ู ุงููู ูู plural effusion.
~ู ูุณุฃูู ุนูู ุงู causes ุ
โก๏ธA_General
CHF
Nephrotic syndrome
Liver cell failure
โก๏ธB_local
Pneumonia
TB
CTD / SLE , RA
Malignancies
Pulmonary infarction
~ุจุนุฏูุง ูุณุฃูู ุนูู ุงู investigations ุ
CXR
CT scan
US ู ุฑุงุช ุชููุฏูู ุจุณ
ABG , pulse oxymetry
Plurocentesis /ุจุงุด ูุญุฏุฏ ุงูููุน exudative ุงู transudative
ู ุนูู ุฃุณุงุณ ุงูุชุญููู ูุฐุง ุจูุจุนุช ุชุญุงููู ุชุงููุฉ ุจุงุด ูุฒุจุท ุงู cause ุ
ูู ูุงู transudative
ุจูุจุนุช
ECG
Echo
Urine for RE
U/Cr/E
LFT
Abdominal US
ูู ูุงู ุงูุณุจุจ exudative
ููุณ ุงูุดู ุจุชุจุนุช ุชุญุงููู ุจุงุด ุชุณุชุจุนุฏ ูู ุงูุงุณุจุงุจ
CBC
Sputum culture, blood culture
Quantiferon test
ESR , CRP
ANA , Anti ds
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