Forwarded from Internal Medicine By Doha Rawag
๐Causes of pulmonary edema
๐ฅCardiogenic
Acute HF
๐ฅNoncardiogenic
๐ต Drowning
๐ต Acute glomerulonephritis
๐ต Fluid overload
๐ต Aspiration
๐ต Inhalation injury
๐ต Opioid overdose
๐ต Salicylate toxicity
๐ต Neurogenic pulmonary edema
๐ต Allergic reaction
๐ต Adult respiratory distress syndrome
๐ฅCardiogenic
Acute HF
๐ฅNoncardiogenic
๐ต Drowning
๐ต Acute glomerulonephritis
๐ต Fluid overload
๐ต Aspiration
๐ต Inhalation injury
๐ต Opioid overdose
๐ต Salicylate toxicity
๐ต Neurogenic pulmonary edema
๐ต Allergic reaction
๐ต Adult respiratory distress syndrome
Forwarded from Internal Medicine By Doha Rawag
๐Pneumonia
There are 2 types of pneumonia:
๐community-acquired pneumonia ๐ hospital acquired (nosocomial) pneumonia.
Community-acquired pneumonia are either typical or atypical, and occurs in community or within the first 72 hours of hospitalization.
Typical community-acquired pneumonia presents with acute onset of shaking chills then fever, productive cough with thick purulent sputum, pleuritic chest pain, and dyspnea. Signs are tachypnea, tachycardia, and pleural friction rub (if pleural effusion). Chest X rays shows lobar or multilobar consolidation.
Atypical community-acquired pneumonia presents with insidious onset of headache, sore throat, fatigue, myalgia, fever (chills are uncommon), and dry cough (no sputum production). Signs include fever with relative bradycardia. Chest X ray shows diffuse reticulonodular infiltrates with absent or minimal consolidation.
Nosocomial pneumonia occurs during hospitalization after the first 73 hours.
The most common pathogens of typical community-acquired pneumonia are strept. pneumoniae, hemophilus influenza, klebsiella and other enterobacteriaceae, and staph. aureus.
The most common pathogens of atypical community-acquired pneumonia are mycoplasma, chlamydia, coxiella, legionella, and viral pneumonia.
The most common bacterial pathogens of nosocomial pneumonia are escherichia coli, pseudomonas, and staph. aureus.
Studies have shown that if vital signs are entirely normal, the probability of pneumonia in outpatients is less than 1%.
Atypical pneumonia refers to organisms not visible on Gram stain and not culturable on standard blood agar.
Upper respiratory infection is more likely than lower respiratory infection if nasal discharge, sore throat, or ear pain predominates.
After treatment, changes evident on CXR usually lag behind the clinical response (up to 6 weeks).
False-negative chest radiographs occur with neutropenia, dehydration, infection with pneumocystis carinii, and early disease (less than 24 hours).
Legionella pneumonia is common in organ transplant recipients, patients with renal failure, patients with chronic lung disease, and smokers; presents with GI symptoms and hyponatremia. Urinary antigen assay for legionella is very sensitive, and the antigen persists in urine for weeks even after starting the treatment.
There are 2 types of pneumonia:
๐community-acquired pneumonia ๐ hospital acquired (nosocomial) pneumonia.
Community-acquired pneumonia are either typical or atypical, and occurs in community or within the first 72 hours of hospitalization.
Typical community-acquired pneumonia presents with acute onset of shaking chills then fever, productive cough with thick purulent sputum, pleuritic chest pain, and dyspnea. Signs are tachypnea, tachycardia, and pleural friction rub (if pleural effusion). Chest X rays shows lobar or multilobar consolidation.
Atypical community-acquired pneumonia presents with insidious onset of headache, sore throat, fatigue, myalgia, fever (chills are uncommon), and dry cough (no sputum production). Signs include fever with relative bradycardia. Chest X ray shows diffuse reticulonodular infiltrates with absent or minimal consolidation.
Nosocomial pneumonia occurs during hospitalization after the first 73 hours.
The most common pathogens of typical community-acquired pneumonia are strept. pneumoniae, hemophilus influenza, klebsiella and other enterobacteriaceae, and staph. aureus.
The most common pathogens of atypical community-acquired pneumonia are mycoplasma, chlamydia, coxiella, legionella, and viral pneumonia.
The most common bacterial pathogens of nosocomial pneumonia are escherichia coli, pseudomonas, and staph. aureus.
Studies have shown that if vital signs are entirely normal, the probability of pneumonia in outpatients is less than 1%.
Atypical pneumonia refers to organisms not visible on Gram stain and not culturable on standard blood agar.
Upper respiratory infection is more likely than lower respiratory infection if nasal discharge, sore throat, or ear pain predominates.
After treatment, changes evident on CXR usually lag behind the clinical response (up to 6 weeks).
False-negative chest radiographs occur with neutropenia, dehydration, infection with pneumocystis carinii, and early disease (less than 24 hours).
Legionella pneumonia is common in organ transplant recipients, patients with renal failure, patients with chronic lung disease, and smokers; presents with GI symptoms and hyponatremia. Urinary antigen assay for legionella is very sensitive, and the antigen persists in urine for weeks even after starting the treatment.
Forwarded from Internal Medicine By Doha Rawag
๐Complications of Pneumonia
โฝ๏ธRespiratory failure, Type 1 respiratory failure is relatively common
โฝ๏ธHypotension
โฝ๏ธAtrial fibrillation
โฝ๏ธPleural effusion
โฝ๏ธEmpyema
โฝ๏ธLung abscess
โฝ๏ธPericarditis and Myocarditis
โฝ๏ธJaundice
โฝ๏ธSepticaemia
โฝ๏ธRespiratory failure, Type 1 respiratory failure is relatively common
โฝ๏ธHypotension
โฝ๏ธAtrial fibrillation
โฝ๏ธPleural effusion
โฝ๏ธEmpyema
โฝ๏ธLung abscess
โฝ๏ธPericarditis and Myocarditis
โฝ๏ธJaundice
โฝ๏ธSepticaemia
โค1
Forwarded from Internal Medicine By Doha Rawag
๐ด Sleep Apnea
โช patients often present with daytime sleepiness, nighttime snoring with apnea, nocturnal awakenings, diffculties in their jobs, and automobile accidents due to falling asleep at the wheel.
โชSpecifc symptoms include paroxysmal nocturnal dyspnea, morning headaches, cardiac arrhythmias, truncal obesity, pulmonary hypertension, nocturnal enuresis, peripheral edema, hypertension, and an elevated hematocrit on laboratory examination.
โชThree patterns of sleep apnea have been described
๐จcentral apnea,
๐จobstructive apnea
๐จmixed apnea.
โซIn central apnea๐ there is #impaired central nervous system control of respiratory effort.
โซConversely, in obstructive apnea๐ the #upper airway becomes #transiently_obstructed, causing airfow to stop despite continuing efforts of the respiratory muscles.
โชAffected patients experience apneic periods, which can occur #between 40 and 100 times per hour.
โชDuring prolonged periods of apnea, the Po2 can #fall to values as low as 20 to 25 mm Hg with oxygen saturation <50%.
โชSustained hypoxemia of this type leads to #arrhythmias, including sinus bradycardia, sinus arrest, long asystoles, frequent atrial premature beats, and ventricular arrhythmias.
โช Pulmonary hypertension may develop with secondary right ventricular hypertrophy.
๐ฅAn effective therapy for many patients with sleep apnea is continuous positive airway pressure (#CPAP), applied #during_sleep via mask or nasal prongs.
โชPatients treated with CPAP demonstrate #improved neuropsychiatric function and reduced daytime somnolence. Nocturnal desaturation, pulmonary hypertension, and right-sided heart failure fndings can all #improve with this technique.
โชMore severe forms of sleep apnea may require
๐palatal_corrective_surgery or, in the most diffcult cases,
๐tracheostomy.
โช patients often present with daytime sleepiness, nighttime snoring with apnea, nocturnal awakenings, diffculties in their jobs, and automobile accidents due to falling asleep at the wheel.
โชSpecifc symptoms include paroxysmal nocturnal dyspnea, morning headaches, cardiac arrhythmias, truncal obesity, pulmonary hypertension, nocturnal enuresis, peripheral edema, hypertension, and an elevated hematocrit on laboratory examination.
โชThree patterns of sleep apnea have been described
๐จcentral apnea,
๐จobstructive apnea
๐จmixed apnea.
โซIn central apnea๐ there is #impaired central nervous system control of respiratory effort.
โซConversely, in obstructive apnea๐ the #upper airway becomes #transiently_obstructed, causing airfow to stop despite continuing efforts of the respiratory muscles.
โชAffected patients experience apneic periods, which can occur #between 40 and 100 times per hour.
โชDuring prolonged periods of apnea, the Po2 can #fall to values as low as 20 to 25 mm Hg with oxygen saturation <50%.
โชSustained hypoxemia of this type leads to #arrhythmias, including sinus bradycardia, sinus arrest, long asystoles, frequent atrial premature beats, and ventricular arrhythmias.
โช Pulmonary hypertension may develop with secondary right ventricular hypertrophy.
๐ฅAn effective therapy for many patients with sleep apnea is continuous positive airway pressure (#CPAP), applied #during_sleep via mask or nasal prongs.
โชPatients treated with CPAP demonstrate #improved neuropsychiatric function and reduced daytime somnolence. Nocturnal desaturation, pulmonary hypertension, and right-sided heart failure fndings can all #improve with this technique.
โชMore severe forms of sleep apnea may require
๐palatal_corrective_surgery or, in the most diffcult cases,
๐tracheostomy.
โค1
Forwarded from Internal Medicine By Doha Rawag
๐ชRemember causes of difficult to control asthma:
1โข non compliant on medications
2โข poor MDI technique (always ask how you do cope with MDI).
3โข CSS (remember upper and lower respiratory symptoms with mononeuritis multiplex).
4โข ABPA (constitutional, hemoptysis, immunosuppressed).
5โข EAA (EPisodic )
6โข GERD
7โข NSAIDs, drugs lower oesophageal sphincter tone
8โข drugs worsen bronchoconstriction (adenosine, ticagrelor, beta blocker)
9โข smoking, occupational hazards, and persistent exposure to allergens.
1โข non compliant on medications
2โข poor MDI technique (always ask how you do cope with MDI).
3โข CSS (remember upper and lower respiratory symptoms with mononeuritis multiplex).
4โข ABPA (constitutional, hemoptysis, immunosuppressed).
5โข EAA (EPisodic )
6โข GERD
7โข NSAIDs, drugs lower oesophageal sphincter tone
8โข drugs worsen bronchoconstriction (adenosine, ticagrelor, beta blocker)
9โข smoking, occupational hazards, and persistent exposure to allergens.
โค1
#Bedside_Notes
CNS / ุฏ.ูุงุทู ุฉ ุงููุนู ู
Q1_ What is the Definition of normal speech ?
๐
Pt should be understanding you and speak clear for you to understand
NO Aphysia
NO Dysartheria
NO Dysphonia
Q2_ What are the causes of dysphonia?
๐
๐ธ๏ธNon neurological
1_ After prolonged speech
2_ URTI
3_ Mass compression from outside
4_ Hypothyroidism
5_ Post inhaler use
6_ Post intubation
๐ธ๏ธNeurological
Recurrent laryngeal N damage
Q3_ Why you cannot use irritating substance for testing the Olfactory N ?
๐
Bc it will stimulate the 5th cranial N.
CNS / ุฏ.ูุงุทู ุฉ ุงููุนู ู
Q1_ What is the Definition of normal speech ?
๐
Pt should be understanding you and speak clear for you to understand
NO Aphysia
NO Dysartheria
NO Dysphonia
Q2_ What are the causes of dysphonia?
๐
๐ธ๏ธNon neurological
1_ After prolonged speech
2_ URTI
3_ Mass compression from outside
4_ Hypothyroidism
5_ Post inhaler use
6_ Post intubation
๐ธ๏ธNeurological
Recurrent laryngeal N damage
Q3_ Why you cannot use irritating substance for testing the Olfactory N ?
๐
Bc it will stimulate the 5th cranial N.
โค2
#Bedside_Notes
CNS / ุฏ.ูุงุทู ุฉ ุงููุนู ู
#Glabbelar_reflex
ุชุจุน ุงู 5th cranial N examination
ููู ููุฏุงุฑ ุ
ุชุถุฑุจ ุถุฑุจุฉ ุฎูููุฉ ุนุงูุฌุจูุฉ (ุจุงููุงู ุฑ ุงู ู ู ุบูุฑู) / ุงู response ุงูุทุจูุนู ูุตูุฑ Eye Blinking ู ุฑุฉ ูุญุฏุฉ ุงู 2
ูู ุงุณุชู ุฑ ุงูุซุฑ ู ู ููู ู ุนูุงูุง / Exaggerated.
CNS / ุฏ.ูุงุทู ุฉ ุงููุนู ู
#Glabbelar_reflex
ุชุจุน ุงู 5th cranial N examination
ููู ููุฏุงุฑ ุ
ุชุถุฑุจ ุถุฑุจุฉ ุฎูููุฉ ุนุงูุฌุจูุฉ (ุจุงููุงู ุฑ ุงู ู ู ุบูุฑู) / ุงู response ุงูุทุจูุนู ูุตูุฑ Eye Blinking ู ุฑุฉ ูุญุฏุฉ ุงู 2
ูู ุงุณุชู ุฑ ุงูุซุฑ ู ู ููู ู ุนูุงูุง / Exaggerated.
โค2
Glabbelar reflex.
๐1
#Bedside_Notes
CNS /. ุฏ.ุงูุตุฏูู ุนุจูุฏ
๐ธ๏ธHow to test for congugate eye movement?
๐
ุชุญุท ุงูุฏูู ุงูุฒูุฒ ูุฏุงู ุนููู ุงูุจูุดูุช ุนูู ู ุณุงูุฉ ูุต ู ุชุฑ
ุจุญูุซ ูุญุฏุฉ ุชููู ู ูุชูุญุฉ ๐ ู ุงูุชุงููุฉ ุชููู ู ุณูุฑุฉโ
ู ุชููู ููุจูุดูุช ู ุฑุฉ ูุดูู ูููุฏ ุงูู ูุชูุญุฉ ๐ ุ ู ู ุฑุฉ ูุดูู ูููุฏ ุงูู ุณูุฑุฉ โ
ู ุชุฑุงูุจ ุญุฑูุฉ ุงูุนููู ูู ูุชุญุฑููุง ู ุน ุจุนุถ ู ุฃู ูุฑูู ุชู ุงู ุงู ูุง.
CNS /. ุฏ.ุงูุตุฏูู ุนุจูุฏ
๐ธ๏ธHow to test for congugate eye movement?
๐
ุชุญุท ุงูุฏูู ุงูุฒูุฒ ูุฏุงู ุนููู ุงูุจูุดูุช ุนูู ู ุณุงูุฉ ูุต ู ุชุฑ
ุจุญูุซ ูุญุฏุฉ ุชููู ู ูุชูุญุฉ ๐ ู ุงูุชุงููุฉ ุชููู ู ุณูุฑุฉโ
ู ุชููู ููุจูุดูุช ู ุฑุฉ ูุดูู ูููุฏ ุงูู ูุชูุญุฉ ๐ ุ ู ู ุฑุฉ ูุดูู ูููุฏ ุงูู ุณูุฑุฉ โ
ู ุชุฑุงูุจ ุญุฑูุฉ ุงูุนููู ูู ูุชุญุฑููุง ู ุน ุจุนุถ ู ุฃู ูุฑูู ุชู ุงู ุงู ูุง.
โค2
How to examine pt with parkinson ๐
โฝ ุชุจุฏุง ุจุงูFace. ๐ง
1_ ุงุฏูุฑูู test for eye movement ๐
โฌ ๏ธ( ุงููู ุนูุฏู parkinson ูุจุฏุง ุนูุฏูู impaired eye movement ุฎุงุตุฉ ุงู up gaze ุจุณุจุจ ุงู Bradykynesia )
2_ ุชูููู ูุจุชุณู ๐
โฌ ๏ธ (ุทุจุนุงู ุงููู ุนูุฏู parkinson ู ุง ููุฏุฑุด ูุญุฑู ุนุถูุงุช ูุฌูู Mask face ุจุณุจุจ ุงู bradykynesia )
3_ ุชูููู ูููู ุฌู ูุฉ ( ู ุชูุงู ุจุณู ุงููู ุงูุฑุญู ู ุงูุฑุญูู ) ู ุชูููู ู ุฑุฉ ูููููุง ุจุตูุช ุนุงูู ู ู ุฑุฉ ูููููุง ุจุตูุช ูุงุชู๐ฃ
โฌ ๏ธ( ุงููู ุนูุฏู parkinson ู ุง ููุฏุฑุด ูุนูู ู ููุทู ุงูุตูุช ู ุชุนู monotonous speech ุจุณุจุจ ุงู Bradykynesia )
โฝุจุนุฏูู ุงู Hand ๐
1_ ุชุดูู ูู ุนูุฏู Tremor ุงู ูุง
โฌ ๏ธ(ุงููู ุนูุฏู parkinson ูุจุฏุง ุนูุฏู Resting or static tremor )
2_ ุชุนุทูู ูุฑูุฉ ู ุจูุฑู ู ุชูููู ููุชุจ ๐
โฌ ๏ธ( ุงููู ุนูุฏูparkinson ุงูุฎุท ู ุชุนู ูุจุฏุง ุตุบูุฑ Microgaphia )
3_ ุงุฏูุฑ test for ms tone
โฌ ๏ธ ( ุงููู ุนูุฏู parkinson ุญูููู ุนูุฏู Hypertonia / Rigidity )
โฝ ุจุนุฏูู ุงู lower limb
1_ ุงุฏูุฑ Test for ms tone
โฌ ๏ธ ( ุงููู ุนูุฏู parkinson ุญูููู ุนูุฏู Hypertonia / Rigidity )
2_ ุชููู ุงูู ุฑูุถ ู ุชูููู ูู ุดู assess the gait ุ ุชุฎููู ูู ุดู ู ุณุงูุฉ ู ุจุนุฏูู ุชูููู ูุบูุฑ ู ุณุงุฑ ุงูู ุดู ุ ูุชููุช ูู ูู ุงู ูุณุงุฑ ู ุจุนุฏูู ุชูููู ูููู .๐ถโโ๏ธ
โฌ ๏ธ (pt with parkinson have Shuffling gait , difficult to start , difficult to stop , difficult to turns around / Due to Bradykynesia )
โฝ ุชุจุฏุง ุจุงูFace. ๐ง
1_ ุงุฏูุฑูู test for eye movement ๐
โฌ ๏ธ( ุงููู ุนูุฏู parkinson ูุจุฏุง ุนูุฏูู impaired eye movement ุฎุงุตุฉ ุงู up gaze ุจุณุจุจ ุงู Bradykynesia )
2_ ุชูููู ูุจุชุณู ๐
โฌ ๏ธ (ุทุจุนุงู ุงููู ุนูุฏู parkinson ู ุง ููุฏุฑุด ูุญุฑู ุนุถูุงุช ูุฌูู Mask face ุจุณุจุจ ุงู bradykynesia )
3_ ุชูููู ูููู ุฌู ูุฉ ( ู ุชูุงู ุจุณู ุงููู ุงูุฑุญู ู ุงูุฑุญูู ) ู ุชูููู ู ุฑุฉ ูููููุง ุจุตูุช ุนุงูู ู ู ุฑุฉ ูููููุง ุจุตูุช ูุงุชู๐ฃ
โฌ ๏ธ( ุงููู ุนูุฏู parkinson ู ุง ููุฏุฑุด ูุนูู ู ููุทู ุงูุตูุช ู ุชุนู monotonous speech ุจุณุจุจ ุงู Bradykynesia )
โฝุจุนุฏูู ุงู Hand ๐
1_ ุชุดูู ูู ุนูุฏู Tremor ุงู ูุง
โฌ ๏ธ(ุงููู ุนูุฏู parkinson ูุจุฏุง ุนูุฏู Resting or static tremor )
2_ ุชุนุทูู ูุฑูุฉ ู ุจูุฑู ู ุชูููู ููุชุจ ๐
โฌ ๏ธ( ุงููู ุนูุฏูparkinson ุงูุฎุท ู ุชุนู ูุจุฏุง ุตุบูุฑ Microgaphia )
3_ ุงุฏูุฑ test for ms tone
โฌ ๏ธ ( ุงููู ุนูุฏู parkinson ุญูููู ุนูุฏู Hypertonia / Rigidity )
โฝ ุจุนุฏูู ุงู lower limb
1_ ุงุฏูุฑ Test for ms tone
โฌ ๏ธ ( ุงููู ุนูุฏู parkinson ุญูููู ุนูุฏู Hypertonia / Rigidity )
2_ ุชููู ุงูู ุฑูุถ ู ุชูููู ูู ุดู assess the gait ุ ุชุฎููู ูู ุดู ู ุณุงูุฉ ู ุจุนุฏูู ุชูููู ูุบูุฑ ู ุณุงุฑ ุงูู ุดู ุ ูุชููุช ูู ูู ุงู ูุณุงุฑ ู ุจุนุฏูู ุชูููู ูููู .๐ถโโ๏ธ
โฌ ๏ธ (pt with parkinson have Shuffling gait , difficult to start , difficult to stop , difficult to turns around / Due to Bradykynesia )
โค3
#CNS๐ง
#bedside_notes๐
ุฏ.ุฌู ุงู ุงูุจุงุฑููู
How to examine mental status of pt ?
ุดูู ุฅุณู ูุ
ุดูู ุงูููุช ุชูุง ( ุตุจุญ ุ ุนุดูุฉ ู ูุง ููู ) ุ
ููู ุงูุช ุชูุง ุ
**ูุฑุถุงู ุนุฑู ู ูุงูู ุงูู ูู ุงูู ุณุชุดูู ุ ุชุณุฃูู ๐
ุนูุงุด ุฌูุช ููู ุณุชุดูู ุ
ู ู ุงููู ุฌุงุจู ููู ุณุชุดูู ุ
**ูู ููู ู ุนุงู Relative ุชุณุฃูู ๐
ู ู ูุฐุง ููู ู ุนุงู ุ
**(ูุฑุถุงู ู ุนุงู ููุฏู ) ุชุณุฃูู ๐
ููุฏู ูุฏุงุด ุนู ุฑู ุ ู ูุงููุฏ ูู ุ!
ุชูุฑูู ุณุงุนุฉ ู ูุง ุชููููู ู ูุง ุจูุฑู ู ุชุณุฃูู ูุฐุง ุดูู ุ
ุชูููู ูุนุฏ ุจุงูุนูุณ (ุนุดุฑุฉ / ุชุณุนุฉ .....)
ุชุณุฃูู ุนูู ุชุงุฑูุฎ ุญุงุฌุฉ ูุฏูู ุฉ (ู ุชูุงู ุ ุนู ู ู ูุญู ุนูู ุงูุบุงุฑุฉ ุ ูู ุงู ุณูุฉ ูุงูุช )
NB:- 1st 3 Questions to assess the orientation for time , place and person .
ุจุงูุชูููู .๐๐
#bedside_notes๐
ุฏ.ุฌู ุงู ุงูุจุงุฑููู
How to examine mental status of pt ?
ุดูู ุฅุณู ูุ
ุดูู ุงูููุช ุชูุง ( ุตุจุญ ุ ุนุดูุฉ ู ูุง ููู ) ุ
ููู ุงูุช ุชูุง ุ
**ูุฑุถุงู ุนุฑู ู ูุงูู ุงูู ูู ุงูู ุณุชุดูู ุ ุชุณุฃูู ๐
ุนูุงุด ุฌูุช ููู ุณุชุดูู ุ
ู ู ุงููู ุฌุงุจู ููู ุณุชุดูู ุ
**ูู ููู ู ุนุงู Relative ุชุณุฃูู ๐
ู ู ูุฐุง ููู ู ุนุงู ุ
**(ูุฑุถุงู ู ุนุงู ููุฏู ) ุชุณุฃูู ๐
ููุฏู ูุฏุงุด ุนู ุฑู ุ ู ูุงููุฏ ูู ุ!
ุชูุฑูู ุณุงุนุฉ ู ูุง ุชููููู ู ูุง ุจูุฑู ู ุชุณุฃูู ูุฐุง ุดูู ุ
ุชูููู ูุนุฏ ุจุงูุนูุณ (ุนุดุฑุฉ / ุชุณุนุฉ .....)
ุชุณุฃูู ุนูู ุชุงุฑูุฎ ุญุงุฌุฉ ูุฏูู ุฉ (ู ุชูุงู ุ ุนู ู ู ูุญู ุนูู ุงูุบุงุฑุฉ ุ ูู ุงู ุณูุฉ ูุงูุช )
NB:- 1st 3 Questions to assess the orientation for time , place and person .
ุจุงูุชูููู .๐๐
โค3
๐Mental status exam
ู ูู ุฉ ุฌุฏุงู ูู ุงู MCQs ุฏูู ุง ูุจูุง ุงู exception.
ู ูู ุฉ ุฌุฏุงู ูู ุงู MCQs ุฏูู ุง ูุจูุง ุงู exception.
๐Dx of Muliple sclerosis (MS)
MS is diagnosed on the basis of clinical findings and supporting evidence from ancillary tests
โ ๏ธMagnetic resonance imaging: The imaging procedure of choice for confirming MS monitoring disease progression in the CNS
Showing hyperintensity on T2-weighted images.
typical locations for MS lesions include the periventricular white matter, brainstem
โ ๏ธVisual Evoked potentials: Used to identify subclinical lesions; results are not specific for MS
โ ๏ธLumber puncture:
evaluated for oligoclonal bands and intrathecal immunoglobulin G (IgG) production.
ุฑูุฒูุง ุนูู ุงู oligomonoclonal antibiotics ู ูู ุฉ ููุจุง ูู ุงู MCQs ๐ฉบ
MS is diagnosed on the basis of clinical findings and supporting evidence from ancillary tests
โ ๏ธMagnetic resonance imaging: The imaging procedure of choice for confirming MS monitoring disease progression in the CNS
Showing hyperintensity on T2-weighted images.
typical locations for MS lesions include the periventricular white matter, brainstem
โ ๏ธVisual Evoked potentials: Used to identify subclinical lesions; results are not specific for MS
โ ๏ธLumber puncture:
evaluated for oligoclonal bands and intrathecal immunoglobulin G (IgG) production.
ุฑูุฒูุง ุนูู ุงู oligomonoclonal antibiotics ู ูู ุฉ ููุจุง ูู ุงู MCQs ๐ฉบ
โค5
Internal Medicine By Doha Rawag pinned ยซุงูุณูุงู
ุนูููู
ูุฑุญู
ุฉ ุงููู ูุจุฑูุงุชู ุฏูุงุชุฑุฉ ุจุฎุตูุต ููุฑุณ ุงููุญุต ุงูุณุฑูุฑู ุชุทุจูู Clinical Medical Examination courseุ ุฎุฏูุช ุงูุนุฏุฏ ุงููุงูู ุญุงููุง ุณุฌููุง ู
ุนุงู 4 ู
ุฌู
ูุนุงุช ู ุงูุนุฏุฏ ู
ุญุฏูุฏ 5 ุฏูุงุชุฑุฉ ูู ูู ู
ุฌู
ูุนุฉ ูุงู ุงูุบุฑุถ ู
ู ุงูููุฑุณ ูู ุฏูุชูุฑ ูุงุฎุฏ ุญูู ูู ุงูุชุทุจูู ู ูุดูู ู ูุณู
ุน findingsโฆยป