Heather Rae, Functional Health Practitioner
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TERRAIN MODEL health coach based in Jalisco Mexico
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We make a tea similar to this recipe, Manuka honey and fresh organic ginger. We toss the ginger in the garden and this morning found a sprouting piece of ginger in the dirt :)
Forwarded from Freedom ideas
Another recipe.
More recipies here: https://t.me/+fmBQBGo20pYwM2Q0
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Join the freedom telegram channel here: https://t.me/covidvaccineexemption
INTRIGUED BY DMSO (DIMETHYL SULFOXIDE) after lectures by Dr Sam Yanuck on fibroblasts, IL1b, NF-kB, TNF, DAMPS, PAMPS, Nrf2, cytokines, neutrophils and a host of inflammation and detoxification compounds.
We are in the mitochondria here, the nuts ‘n bolts of functional genomics (my practice). There are genes (enzymes) involved but this study makes no reference to them (like the genes that code compounds related to glutathione, sulfation detox which will be unique to each person)

“DMSO is an [reactive oxygen species] scavenger resulting in inhibition of NLRP3 inflammasome activation.” It blocks inflammatory interleukins like IL6 and NFkB signaling.

The end of the paper says that DMSO may have negative side effects, as in the mice stank of garlic. There are some possible issues with heart/vascular (possibly having to do with calcium channels) but stinking like garlic (sulphur, sulfoxide) isn’t so bad ;)

https://tahomaclinic.com/Private/Articles3/DMSO/Ahn%202014%20-%20Dimethyl%20sulfoxide%20inhibits%20NLRP3%20inflammasome%20activation.pdf
“Due to its anti-inflammatory and ROS scavenger activities, DMSO is popularly used as a vehicle in drug therapy for various diseases, including dermatological disorders, amyloidosis, gastrointestinal diseases, traumatic brain edema, musculoskeletal disorders, pulmonary adenocarcinoma, rheumatologic disease, schizophrenia, and Alzhimer’s diseases (Santos et al., 2003).”
DMSO Is anti-inflammatory and quite similar in action to Nrf2, the signaling molecule for glutathione.
I painted delicate chairs with ARSENIC (aka “Paris Green”) by Farrow and Ball. This green paint once contained arsenic, thus the name.

“Arsenic as copper acetoarsenite was a pigment in paints, the best known being “Paris green”. Before electricity was used for illumination, hydrogen liberated from coal fires and from gas for lighting combined with arsenic in the Paris green used in wallpaper to form arsine, a toxic gas. A fungus Scopulariopsis breviculis present in damp wallpaper also metabolised the arsenic in Paris green to arsine.” https://pmj.bmj.com/content/79/933/391

How many people are still poisoned by arsenic which causes cells to decay and die. And how many ‘viruses’ are blamed for these illnesses, rather than the real culprits such as arsenic containing water, pesticides?
VACCINE INJURY & YOUR GENETICS (a close up) In the past few years, I've participated in genomic analysis of over 100 case studies of people struggling with complex chronic health conditions. Then came "COVID" I knew the 'SARS COV2 'virus' had never been found in a human being, so it wasn't clear, not at all, what "COVID" meant. When my mentor said, "COVID" or "virus" my brain exploded a little, and I made mental holding place for 'whatever that is." Then came the "COVID injections" and spike proteins, I got really deep into the inflammation (genes/enzymes) of spike proteins and those angiotensin enzymes that kick up histamine and mast cells and RANTES. Now these genomic analyses have shifted to looking at inflammatory NF-kB and TNF (mentioned a couple posts above). "COVID" so goes the narrative, activates inflammatory NF-kB and TNF-a. The ovals in the diagram above are genes/enyzmes. In the next post, I'll list the names of the natural compounds and pharma drugs that suppress TNF-a.
NATURAL SUPPRESSION OF TNF-a black cumin seed oil, milk thistle, boswellia, nicotinamide, ECGC, licorice, luteolin, andrographis, fisetin, resveratrol
PHARMA SUPPRESSION OF TNF-a ivermectin, rapamycin, cannabis/THC, statins/red rice yeast, low dose naltrexone
The thing about "COVID" (whatever that is) is this: if you have other toxins on board, they too will activate these inflammatory compounds, TNF-a and NF-kB. We're talking glyphosate, myotoxins, and excess iron and excess oxalates and glutamate and EMFs. We're talking damaged cells and bacteria coming in to clean up. We're talking about toxins in an injection, toxins which have yet to be fully disclosed to the public. A 'virus' is a convenient cover story for Fauci and Fraudsters, but believing in unicorns simply keeps people from figuring out what's really causing them to be ill and taking the proper steps to right their ships, to find homeostasis and equilibrium in mind and body.
"DMSO reduces the secretion of 13 cytokines important in inflammatory response" including both TNF-a and NF-kB. https://www.researchgate.net/publication/320995707_Dimethyl_Sulfoxide_DMSO_Decreases_Cell_Proliferation_and_TNF-a_IFN-g_and_IL-2_Cytokines_Production_in_Cultures_of_Peripheral_Blood_Lymphocytes "Dimethyl Sulfoxide (DMSO) Decreases Cell Proliferation and TNF-α, IFN-γ, and IL-2 Cytokines Production in Cultures of Peripheral Blood Lymphocytes"
Parking this nugget here for future reference.
What is LONG HAUL COVID and how to treat?
It looks to be a combination of
MAST CELL ACTIVATION SYNDROME,
MACROPHAGE ACTIVATION SYNDROME (excess macrophages) and
ANTI-PHOSPHOLIPID SYNDROME (lipid peroxidation, disruption of phospholipids which looks a lot like LUPUS/AUTOIMMUNITY), FERROPTOSIS (excess intravascular iron) which causes excess GLUTAMATE and blocks CYSTEINE which damages the cell (lipid peroxidation)
A few suggestions .... vitamin E (for ferroptosis), selenium for GPX4. Nrf2 support, NAC/cysteine .. lactoferrin, skullcap.
Watch for varicose/spider veins ... indication of blood dysregulation from the 'vaccine'
FERROPTOSIS "Ferroptosis is a type of programmed cell death dependent on iron and characterized by the accumulation of lipid peroxides, and is genetically and biochemically distinct from other forms of regulated cell death such as apoptosis." https://pubmed.ncbi.nlm.nih.gov/33454595/ The role of iron in the pathogenesis of COVID-19 and possible treatment with lactoferrin and other iron chelators
MARCH 2022 PAPER ON CENTRAL NERVOUS SYSTEM DISORDERS ... just last month, a paper was released describing in detail inflammation and genes (enzymes) associated with neurological disorders. It describes neuro damage occurring in “COVID vaccine” injury and “long haul COVID” ... without stating it. It is the activation of the things I wrote about above: NF-kB, TNF-a, NLRP3, Nrf2, as well as MindMap inflammation markers also above. In a genomic analysis, we can see how well an individual’s enzymes (genes) are designed to manage toxins like injections and identify specific nutritional supports to bring the inflammation to resolution.
https://www.researchgate.net/publication/359568061_Targeting_NLRP3_Inflammasome_With_Nrf2_Inducers_in_Central_Nervous_System_Disorders
NRF2/KEAP1 ... KEAP1 controls the master signaling molecule called Nrf2; when you have variants in KEAP1, you will hold on to Nrf2. Nrf2 signals the body to use glutathione, a master anti-oxidant. When you have variants in both KEAP1 *and* Nrf2, you will have double difficulty releasing the Nrf2 signal to make glutathione. If you have variants in the genes that make and recycle glutathione (like GSR) you should not take glutathione straight up. Nitric oxide and vitamin and toxins also play a role. In short, you will need to be smarter about nutritional support, know your DNA, else you can make yourself worse off.