HEALTH CARE 2.0 ... WHEN YOU'RE TRULY READY TO RE(GAIN) OWNERSHIP OF YOUR MIND, YOUR BODY, AND YOUR HEALTH

Detox’ing the toxins in the COVID injections requires not just glutathione but sulfates. Know Your DNA ... know how well you make, use and recycle glutathione and sulfates. This article describes why sulfates are so important, how to make more of them and what genes (enzymes) are involved (like SUOX, SULT). https://www.eonutrition.co.uk/post/sulfate-i-the-basics

You can test yourself for levels of glyphosate ... here is one of many reasons to keep glyphosate out of your body especially if you have variants in the SUOX (sulfite to sulfate) enzyme.
“Sulfite oxidase is responsible for oxidising sulfite into sulfate, the form that the body can utilise. It is important to note that this enzyme requires the trace element molybdenum to function. People who are insufficient in molybdenum (for whatever reason) may experience negative reactions when they consume sulfites or excess sulfur-containing foods. Glyphosate is a known chelator of molybdenum, and therefore can contribute to sulfite oxidase dysfunction. Furthermore, heavy metals such aluminium and mercury can also interfere with the enzyme's function.”

GAS BLOAT DIARRHEA & SIBO ... It may be caused by bacteria in the gut making hydrogen sulfide when you don’t make enough sulfates elsewhere ....

New Study Shows Omicron Replicates 70 Times Faster Than Delta in Airways, but Lung Infection Appears Less Severe

🇭🇰📝🦠@COVID19Up: The Omicron variant of SARS-CoV-2 replicates 70 times faster in human airways than Delta, but infection in the lungs appears to be less severe compared to the original virus strain, according to a new study published this week by researchers at the University of Hong Kong’s Faculty of Medicine. The study is under peer review.

Dr. Michael Chan Chi-wai and a team of researchers found that the variant replicates much faster in the bronchus, which connects the windpipe to the lungs, 24 hours after infection. Yet it reproduces more than 10 times slower in the actual human lung tissue, they said.

Omicron’s rapid replication in the airway may explain why it transmits faster than previous variants of the virus, but lower infection in the lungs may indicate that it causes less severe disease, according to the study.

“It is important to note that the severity of disease in humans is not determined only by virus replication but also by the host immune response to the infection,” Chan said in a statement.

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What is the risk of Autoimmunity to ACE2 from Vaccination?

Angiotensin-converting enzyme 2 (ACE2) is expressed in the lung, heart and kidney, free serum ACE2 also circulates in the blood. The SARS-CoV-2 spike protein has a very high affinity for the ACE2 receptor. During infection it has been theorized that because of this, the spike-ACE2 complex is taken up by immune cells and presented to the immune system. Autoantibodies against ACE2 (aa-ACE2) have been detected in patients that have recovered from SARS-CoV-2 infection. Additionally, ACE2 cleaves angiotensin II resulting in decreased activation of the AT1 receptor and decreased immune system activation. Meaning if aa-ACE2s reduce ACE2 activity, this could trigger a proinflammatory state. This could partially explain lingering symptoms after SARS-CoV-2 infection and vaccination.

Does this occur in vaccination against SARS-CoV-2? (How if at all has this been safeguarded against?)

Does the breastmilk of vaccinated mothers contain autoantibodies against ACE2?

During exertion a vaccinee will be increasing the exposure of any circulating aa-ACE2 to the ACE2 expressed on heart tissue due to increased circulation. What is the concentration of autoantibodies against ACE2 in vaccinated athletes who have collapsed?

Due to selection pressure created by the leaky vaccine, variants have become more infectious, in part due to mutations that increase affinity for ACE2. What impact will vaccination with an updated spike from a more infectious variant have on the creation of autoantibodies against ACE2?

🫀Autoimmunity to ACE2 as a possible cause of tissue inflammation in COVID-19 | Medical Hypotheses (2020)
🫀IgM autoantibodies recognizing ACE2 are associated with severe COVID-19 | medRxiv (2020)
🫀Development of ACE2 autoantibodies after SARS-CoV-2 infection | PLOS ONE (2021)
🫀COVID-19—A Theory of Autoimmunity Against ACE-2 Explained | Front. Immunol. (2021)

Almost two years into the fraud. People on pharma meds (ACE inhibitors) have been primed to be harmed by the proteins in the injections.

Self-Organized Criticality Theory of Autoimmunity | PLOS One (2009)

Repeated immunization with antigen causes systemic autoimmunity in mice otherwise not
prone to spontaneous autoimmune diseases.

This study concludes that systemic autoimmunity appears to be the inevitable consequence of over-stimulating the host’s immune system by repeated immunization with antigen.

With ridiculous talk of repeated mass vaccination and "boosters" with one-size-fits-all dosing, this work from 2009 predicts that if this goes ahead we will see a historic spike in autoimmune disorders in the years to come.