Forwarded from James Lyons-Weiler, PhD
A little light reading as I being to prepare course material for The Biology of Immunology @ IPAK-EDU. This in addition to the tens of thousands of papers on Immunology. Here's the working syllabus. Reserve your seat today!
https://ipak-edu.org/old-home-2/?store-page=The-Biology-of-the-Immune-System-p412473268
IPAK-EDU
Syllabus – The Biology of the Immune System
Spring 2022 (January-June)
Instructor: James Lyons-Weiler, PhD
Live class meetings: Day TBA @ 1PM ET via Zoom
If you miss a class, don’t worry. Lecture videos will be available after each class.
Course description: In a series of live online presentations and discussions, students will learn the fundamentals of the biology of the immune system and become well-informed and primed for discussions in the public sphere.There will be recommended readings, additional recommended videos, recommended books.There are no quizzes or exams. Students are encouraged to think of questions during each lecture to be ready to facilitate the live discussion after each lecture.
Lecture Schedule Topics (15 or 16 lectures in total, tentative, subject to change)
1 Background and Course Overview
2 History of Immunology
3 First Defenses
4 Innate Immunity Overview
5 Adaptive Immunity Overview (Adaptive Immunity is Recombination Defense)
6 Gastroimmunity
7 Immune System and Development (Immunohomeostasis)
8 Autoimmunity (Processes, Nervous System, Cardiovascular System, Metabolism)
9 Anaphylaxis, Mast Cell Disease
10 Asthma is an Autoimmune Condition
11 Immunology and Cancer
12 Immunology and Degenerative Diseases
13 Microglia and Your Brain
14 Pathogen/Host Interactions
15 Immunotoxins
16 SARS-COV-2, COVID-19 and Human Immunity
https://ipak-edu.org/old-home-2/?store-page=The-Biology-of-the-Immune-System-p412473268
IPAK-EDU
Syllabus – The Biology of the Immune System
Spring 2022 (January-June)
Instructor: James Lyons-Weiler, PhD
Live class meetings: Day TBA @ 1PM ET via Zoom
If you miss a class, don’t worry. Lecture videos will be available after each class.
Course description: In a series of live online presentations and discussions, students will learn the fundamentals of the biology of the immune system and become well-informed and primed for discussions in the public sphere.There will be recommended readings, additional recommended videos, recommended books.There are no quizzes or exams. Students are encouraged to think of questions during each lecture to be ready to facilitate the live discussion after each lecture.
Lecture Schedule Topics (15 or 16 lectures in total, tentative, subject to change)
1 Background and Course Overview
2 History of Immunology
3 First Defenses
4 Innate Immunity Overview
5 Adaptive Immunity Overview (Adaptive Immunity is Recombination Defense)
6 Gastroimmunity
7 Immune System and Development (Immunohomeostasis)
8 Autoimmunity (Processes, Nervous System, Cardiovascular System, Metabolism)
9 Anaphylaxis, Mast Cell Disease
10 Asthma is an Autoimmune Condition
11 Immunology and Cancer
12 Immunology and Degenerative Diseases
13 Microglia and Your Brain
14 Pathogen/Host Interactions
15 Immunotoxins
16 SARS-COV-2, COVID-19 and Human Immunity
Why do people believe “it” is real? David Martin gets into it .... https://brandnewtube.com/watch/dr-david-martin-the-endgame-of-the-elite-jab-tyranny-amp-war-on-humanity_sdkpGaYlr6cS9BG.html
Forwarded from Heather Rae, Functional Health Practitioner
Forwarded from Heather Rae, Functional Health Practitioner
Forwarded from Heather Rae, Functional Health Practitioner
Forwarded from Heather Rae, Functional Health Practitioner
HEALTH CARE 2.0 ... WHEN YOU'RE TRULY READY TO RE(GAIN) OWNERSHIP OF YOUR MIND, YOUR BODY, AND YOUR HEALTH
Detox’ing the toxins in the COVID injections requires not just glutathione but sulfates. Know Your DNA ... know how well you make, use and recycle glutathione and sulfates. This article describes why sulfates are so important, how to make more of them and what genes (enzymes) are involved (like SUOX, SULT). https://www.eonutrition.co.uk/post/sulfate-i-the-basics
EONUTRITION
Sulfate I: The Basics
Overview and key points: • Sulfur is an element which the body uses for sulfation reactions. • Sulfur can be acquired in many forms such as: Inorganic sulfate, sulfite and hydrogen sulfide found in various foods and sources of water …
Forwarded from Heather Rae, Functional Health Practitioner
You can test yourself for levels of glyphosate ... here is one of many reasons to keep glyphosate out of your body especially if you have variants in the SUOX (sulfite to sulfate) enzyme.
“Sulfite oxidase is responsible for oxidising sulfite into sulfate, the form that the body can utilise. It is important to note that this enzyme requires the trace element molybdenum to function. People who are insufficient in molybdenum (for whatever reason) may experience negative reactions when they consume sulfites or excess sulfur-containing foods. Glyphosate is a known chelator of molybdenum, and therefore can contribute to sulfite oxidase dysfunction. Furthermore, heavy metals such aluminium and mercury can also interfere with the enzyme's function.”
“Sulfite oxidase is responsible for oxidising sulfite into sulfate, the form that the body can utilise. It is important to note that this enzyme requires the trace element molybdenum to function. People who are insufficient in molybdenum (for whatever reason) may experience negative reactions when they consume sulfites or excess sulfur-containing foods. Glyphosate is a known chelator of molybdenum, and therefore can contribute to sulfite oxidase dysfunction. Furthermore, heavy metals such aluminium and mercury can also interfere with the enzyme's function.”
GAS BLOAT DIARRHEA & SIBO ... It may be caused by bacteria in the gut making hydrogen sulfide when you don’t make enough sulfates elsewhere ....
The obfuscation is in the words .... “We performed whole-genome sequencing of available remnant mid-turbinate nasal, nasopharyngeal and/or oropharyngeal (OP) swab samples collected from 1,373 PCR-positive COVID-19”. The PCR IS NOT A TEST. It can be used to identify genetic sequences in a sample. The “COVID” PCR does not look for SARS-COV2 sequence. So what on earth does a “positive PCR test” even mean? Statements about “whole gene sequencing” need to be defined, verified, authenticated.
Forwarded from COVID-19 Up
New Study Shows Omicron Replicates 70 Times Faster Than Delta in Airways, but Lung Infection Appears Less Severe
🇭🇰📝🦠@COVID19Up: The Omicron variant of SARS-CoV-2 replicates 70 times faster in human airways than Delta, but infection in the lungs appears to be less severe compared to the original virus strain, according to a new study published this week by researchers at the University of Hong Kong’s Faculty of Medicine. The study is under peer review.
Dr. Michael Chan Chi-wai and a team of researchers found that the variant replicates much faster in the bronchus, which connects the windpipe to the lungs, 24 hours after infection. Yet it reproduces more than 10 times slower in the actual human lung tissue, they said.
Omicron’s rapid replication in the airway may explain why it transmits faster than previous variants of the virus, but lower infection in the lungs may indicate that it causes less severe disease, according to the study.
“It is important to note that the severity of disease in humans is not determined only by virus replication but also by the host immune response to the infection,” Chan said in a statement.
Join our 12k member chat:
🗣@COVID1984Chat
Follow our news channel:
📡@COVID19Up
🇭🇰📝🦠@COVID19Up: The Omicron variant of SARS-CoV-2 replicates 70 times faster in human airways than Delta, but infection in the lungs appears to be less severe compared to the original virus strain, according to a new study published this week by researchers at the University of Hong Kong’s Faculty of Medicine. The study is under peer review.
Dr. Michael Chan Chi-wai and a team of researchers found that the variant replicates much faster in the bronchus, which connects the windpipe to the lungs, 24 hours after infection. Yet it reproduces more than 10 times slower in the actual human lung tissue, they said.
Omicron’s rapid replication in the airway may explain why it transmits faster than previous variants of the virus, but lower infection in the lungs may indicate that it causes less severe disease, according to the study.
“It is important to note that the severity of disease in humans is not determined only by virus replication but also by the host immune response to the infection,” Chan said in a statement.
Join our 12k member chat:
🗣@COVID1984Chat
Follow our news channel:
📡@COVID19Up
In my practice, genomic analysis, I can see where someone has variants in the angiotensin/inflammatory pathway that leads (via mast cell, histamine, interleukins) to “cytokine storm”. KNOW YOUR DNA, nutrient and toxic levels ... because each one of us is uniquely and divinely designed to manage fungi, bacteria, and toxins.
Forwarded from LeakyVax
What is the risk of Autoimmunity to ACE2 from Vaccination?
Angiotensin-converting enzyme 2 (ACE2) is expressed in the lung, heart and kidney, free serum ACE2 also circulates in the blood. The SARS-CoV-2 spike protein has a very high affinity for the ACE2 receptor. During infection it has been theorized that because of this, the spike-ACE2 complex is taken up by immune cells and presented to the immune system. Autoantibodies against ACE2 (aa-ACE2) have been detected in patients that have recovered from SARS-CoV-2 infection. Additionally, ACE2 cleaves angiotensin II resulting in decreased activation of the AT1 receptor and decreased immune system activation. Meaning if aa-ACE2s reduce ACE2 activity, this could trigger a proinflammatory state. This could partially explain lingering symptoms after SARS-CoV-2 infection and vaccination.
Does this occur in vaccination against SARS-CoV-2? (How if at all has this been safeguarded against?)
Does the breastmilk of vaccinated mothers contain autoantibodies against ACE2?
During exertion a vaccinee will be increasing the exposure of any circulating aa-ACE2 to the ACE2 expressed on heart tissue due to increased circulation. What is the concentration of autoantibodies against ACE2 in vaccinated athletes who have collapsed?
Due to selection pressure created by the leaky vaccine, variants have become more infectious, in part due to mutations that increase affinity for ACE2. What impact will vaccination with an updated spike from a more infectious variant have on the creation of autoantibodies against ACE2?
🫀Autoimmunity to ACE2 as a possible cause of tissue inflammation in COVID-19 | Medical Hypotheses (2020)
🫀IgM autoantibodies recognizing ACE2 are associated with severe COVID-19 | medRxiv (2020)
🫀Development of ACE2 autoantibodies after SARS-CoV-2 infection | PLOS ONE (2021)
🫀COVID-19—A Theory of Autoimmunity Against ACE-2 Explained | Front. Immunol. (2021)
Angiotensin-converting enzyme 2 (ACE2) is expressed in the lung, heart and kidney, free serum ACE2 also circulates in the blood. The SARS-CoV-2 spike protein has a very high affinity for the ACE2 receptor. During infection it has been theorized that because of this, the spike-ACE2 complex is taken up by immune cells and presented to the immune system. Autoantibodies against ACE2 (aa-ACE2) have been detected in patients that have recovered from SARS-CoV-2 infection. Additionally, ACE2 cleaves angiotensin II resulting in decreased activation of the AT1 receptor and decreased immune system activation. Meaning if aa-ACE2s reduce ACE2 activity, this could trigger a proinflammatory state. This could partially explain lingering symptoms after SARS-CoV-2 infection and vaccination.
Does this occur in vaccination against SARS-CoV-2? (How if at all has this been safeguarded against?)
Does the breastmilk of vaccinated mothers contain autoantibodies against ACE2?
During exertion a vaccinee will be increasing the exposure of any circulating aa-ACE2 to the ACE2 expressed on heart tissue due to increased circulation. What is the concentration of autoantibodies against ACE2 in vaccinated athletes who have collapsed?
Due to selection pressure created by the leaky vaccine, variants have become more infectious, in part due to mutations that increase affinity for ACE2. What impact will vaccination with an updated spike from a more infectious variant have on the creation of autoantibodies against ACE2?
🫀Autoimmunity to ACE2 as a possible cause of tissue inflammation in COVID-19 | Medical Hypotheses (2020)
🫀IgM autoantibodies recognizing ACE2 are associated with severe COVID-19 | medRxiv (2020)
🫀Development of ACE2 autoantibodies after SARS-CoV-2 infection | PLOS ONE (2021)
🫀COVID-19—A Theory of Autoimmunity Against ACE-2 Explained | Front. Immunol. (2021)