🔴Exercise 1
1)are:
a-boy 5 years old
b- recurrent bacterial infection
c- normal Antibodies & B cell & T cell
d- deficient in NADPH oxchroni
2) chronic granulomatous disease (CGD)
3) H2O2 produce by organism in some time they produce catalase which destroy H2O2 but strept .Pneumoni doesn't pruduce catalase so it killed by H2O2 >> no infection
4)X-linked disease so it affects male mor than female
🔴Exercis 2 :
1)are :
a-3 weaks of age
b-swelling ,redeness around umblical cord without pus formation
c-no WBC around the inflamation
d-normal Ig & B cell & T cell count and function
2)Leukocyte adhesion deficiency (LAD)
3)due to defect in adhesiin mulecules integrin (CD18)
4) a : LAD
●impair emigration of WBC
●No abscess
●no defect in phagicytosis
b: CGD
●no impair in emigration
●Abscess is present
●defect in phagocytosis
1)are:
a-boy 5 years old
b- recurrent bacterial infection
c- normal Antibodies & B cell & T cell
d- deficient in NADPH oxchroni
2) chronic granulomatous disease (CGD)
3) H2O2 produce by organism in some time they produce catalase which destroy H2O2 but strept .Pneumoni doesn't pruduce catalase so it killed by H2O2 >> no infection
4)X-linked disease so it affects male mor than female
🔴Exercis 2 :
1)are :
a-3 weaks of age
b-swelling ,redeness around umblical cord without pus formation
c-no WBC around the inflamation
d-normal Ig & B cell & T cell count and function
2)Leukocyte adhesion deficiency (LAD)
3)due to defect in adhesiin mulecules integrin (CD18)
4) a : LAD
●impair emigration of WBC
●No abscess
●no defect in phagicytosis
b: CGD
●no impair in emigration
●Abscess is present
●defect in phagocytosis
🔴Exercise 4 :
1) are :
a- 6 days
b- anomalies
c- hypocalciemia
d- no CD3 T cell & no IgG
2) Digeorges syndrome (no thymus ,no parathyroid gland )
3)attenuated vaccine is T cell dependent antigen >>in T cell >> no B cell activation >>no Ab production
4)because no class switching due to no cytokine from T cell
🔴Exercise 5:
1)are :
a- 9 months
b- recurrent infection
c- T cell normal
d- No B cell & no Ig class
2)because maternal IgG that crossed placenta give him passive immunity till 9 months
3)X-Linked infantile Bruton's agammaglubulinaemia
1) are :
a- 6 days
b- anomalies
c- hypocalciemia
d- no CD3 T cell & no IgG
2) Digeorges syndrome (no thymus ,no parathyroid gland )
3)attenuated vaccine is T cell dependent antigen >>in T cell >> no B cell activation >>no Ab production
4)because no class switching due to no cytokine from T cell
🔴Exercise 5:
1)are :
a- 9 months
b- recurrent infection
c- T cell normal
d- No B cell & no Ig class
2)because maternal IgG that crossed placenta give him passive immunity till 9 months
3)X-Linked infantile Bruton's agammaglubulinaemia
🔴Exercise 6 :
1)are :
a- 6 months age
b- opportunistic infection
c-low lymphocytes &low CD4
b- Low antibodies &absence DR
2) because maturation of CD4 T cell require interaction with MHC II molecules on thymic epithelial cell
3) Becouse B cell proliferation & differentiation require cytokine from CD4 T cell
4)yes , the rejection will be by CD8 T cell
1)are :
a- 6 months age
b- opportunistic infection
c-low lymphocytes &low CD4
b- Low antibodies &absence DR
2) because maturation of CD4 T cell require interaction with MHC II molecules on thymic epithelial cell
3) Becouse B cell proliferation & differentiation require cytokine from CD4 T cell
4)yes , the rejection will be by CD8 T cell
السلاااام عليكم ⚇
هولاء المواضيع والمحاضرات اللي اخذناهااا من بداية ترم ثاني ....
🔵Pathology 🌟
1-Cardiovascular system د. امين عقبة
2-Respiratory system د. امين عقبة
3-GIT د. عبدالله الذبحاني
4-Female genetal tract د. منية .....
🔵 Pharmacology 🌟
🔴 blood د. اسماء
●Cuagulation disorder
●Dislipidaemia
●NSAID
● chemotherapy( anticancer ) د. عدنان
🔴Endocrine ☚د. عدنان
●Thyroid gland disease
●Gonad
●Pituitary Gland disorder
●Diabetes melitus
●Adrenal gland disorder
●Parathyroid Gland Disorder
🔴Antibiotics د. اسماء
●Quinolonse
● Protein synthesis inhibitors
🔴CNS د. عدنان
● Psychosis
●Depression
●Epilepsy
●Parkinson Disease
●Opoid analgisic &Drug of abuse
●Muscle relaxant
●Hypnotic &Sedative &Alcohl abuse
🔸General &local anesthesia د. اسماء
🔴Infectious Diseases د. عدنان
●T.B & Leprosy & Brucellosis
🔵virology🌟
1-General virology
2- Replication of viruses
3-Labiratory Diagnosis
4-Herpes simplex viruses (1&2)
5-Varicella zoster virus
6-Epstein Barr virus & Cytomegalovirus
7-smallpox virus
8-Adenovirus &Papilloma virus
9-Parvovirus B19
10-Hepatitis viruses (HAV&HEV)
11-Hepatitis B virus
12-Hepatitis viruses (HCV& HGV)
13-Coxsackieviruses
14 Rhinovirus & Poliovirus
15-Reoviruse & Caliciviruse & Asterovirus
16-AIDS
17- Orthomyxovirus ( Infleunza )
18- Paramyxoviruses ( Parainfleunza & Respiratory Syncytial & Measles & Mumps & Rubella)
19-Rabies virus
20-Slow & Oncogenic viruses &Dengue fever
🔵Bacteriology🌟 بالاضافة لتكملة مواضيع ترم اول
1- Mycoplasma
2- Normal flora
3- host parasite relationships
4- Epidemiology
5-Respiratory diseases
6-Diarrhoeal Diseases
7-UTI
8-PUO
9-STD
🔵Immunology 🌟
1-Immunity to mimicroto
2-Hypersensitivity Reaction Types
3-Tumor Immunology
4-Transplantation immunology
5-Tolerance &Autoimmune disease
6-Complement system
🔵Parasitology🌟
1-Trematoda ( Hepatic & Blood flukes)
2- Cestoda كامل
3- Nematoda لين صفحة 63 بكتاب الشيخ
4-Dracunculus medinensis
🔵Community medicine🌟
🔴الدكتورة امة اللطيف :-
●Introduction to Researches
●Health research and Study types
🔴 الدكتورة بثينة :-
● Observational study
🔴 الدكتورة نجيبة:-
●Case Control study
● Data collection techniques
🔴 الدكتور خالد الجنداري وبائيات ( Epidemiology ):
🔵علم النفس - السلوكيات🌟
●مقدمة عن السلوك وعلاقة بالطب
●انواع الشخصيات
#Group_3💙
هولاء المواضيع والمحاضرات اللي اخذناهااا من بداية ترم ثاني ....
🔵Pathology 🌟
1-Cardiovascular system د. امين عقبة
2-Respiratory system د. امين عقبة
3-GIT د. عبدالله الذبحاني
4-Female genetal tract د. منية .....
🔵 Pharmacology 🌟
🔴 blood د. اسماء
●Cuagulation disorder
●Dislipidaemia
●NSAID
● chemotherapy( anticancer ) د. عدنان
🔴Endocrine ☚د. عدنان
●Thyroid gland disease
●Gonad
●Pituitary Gland disorder
●Diabetes melitus
●Adrenal gland disorder
●Parathyroid Gland Disorder
🔴Antibiotics د. اسماء
●Quinolonse
● Protein synthesis inhibitors
🔴CNS د. عدنان
● Psychosis
●Depression
●Epilepsy
●Parkinson Disease
●Opoid analgisic &Drug of abuse
●Muscle relaxant
●Hypnotic &Sedative &Alcohl abuse
🔸General &local anesthesia د. اسماء
🔴Infectious Diseases د. عدنان
●T.B & Leprosy & Brucellosis
🔵virology🌟
1-General virology
2- Replication of viruses
3-Labiratory Diagnosis
4-Herpes simplex viruses (1&2)
5-Varicella zoster virus
6-Epstein Barr virus & Cytomegalovirus
7-smallpox virus
8-Adenovirus &Papilloma virus
9-Parvovirus B19
10-Hepatitis viruses (HAV&HEV)
11-Hepatitis B virus
12-Hepatitis viruses (HCV& HGV)
13-Coxsackieviruses
14 Rhinovirus & Poliovirus
15-Reoviruse & Caliciviruse & Asterovirus
16-AIDS
17- Orthomyxovirus ( Infleunza )
18- Paramyxoviruses ( Parainfleunza & Respiratory Syncytial & Measles & Mumps & Rubella)
19-Rabies virus
20-Slow & Oncogenic viruses &Dengue fever
🔵Bacteriology🌟 بالاضافة لتكملة مواضيع ترم اول
1- Mycoplasma
2- Normal flora
3- host parasite relationships
4- Epidemiology
5-Respiratory diseases
6-Diarrhoeal Diseases
7-UTI
8-PUO
9-STD
🔵Immunology 🌟
1-Immunity to mimicroto
2-Hypersensitivity Reaction Types
3-Tumor Immunology
4-Transplantation immunology
5-Tolerance &Autoimmune disease
6-Complement system
🔵Parasitology🌟
1-Trematoda ( Hepatic & Blood flukes)
2- Cestoda كامل
3- Nematoda لين صفحة 63 بكتاب الشيخ
4-Dracunculus medinensis
🔵Community medicine🌟
🔴الدكتورة امة اللطيف :-
●Introduction to Researches
●Health research and Study types
🔴 الدكتورة بثينة :-
● Observational study
🔴 الدكتورة نجيبة:-
●Case Control study
● Data collection techniques
🔴 الدكتور خالد الجنداري وبائيات ( Epidemiology ):
🔵علم النفس - السلوكيات🌟
●مقدمة عن السلوك وعلاقة بالطب
●انواع الشخصيات
#Group_3💙
🔴ACETAMINOPHEN
✍MOA/ inhibits prostaglandin synthesis in the CNS also act as a partial inhibitor of COX 1,2&3.
✍Uses / is the analgesic/antipyretic of first choice for pregnancy, lactating women, peptic ulcer and children with viral infections or chickenpox.
👉lake anti-inflammatory effects
👉Acetaminophen should be
avoided in patients with severe hepatic impairment.
👉Patients with hepatic disease, viral hepatitis, or a history of alcoholism are at higher risk of acetaminophen induced hepatotoxicity.
⚡️Side effect it may cause Tolerance.
👉 N-acetylcysteine, which contains sulfhydryl groups to which the toxic metabolite can bind, is an antidote in cases of overdose.
✍MOA/ inhibits prostaglandin synthesis in the CNS also act as a partial inhibitor of COX 1,2&3.
✍Uses / is the analgesic/antipyretic of first choice for pregnancy, lactating women, peptic ulcer and children with viral infections or chickenpox.
👉lake anti-inflammatory effects
👉Acetaminophen should be
avoided in patients with severe hepatic impairment.
👉Patients with hepatic disease, viral hepatitis, or a history of alcoholism are at higher risk of acetaminophen induced hepatotoxicity.
⚡️Side effect it may cause Tolerance.
👉 N-acetylcysteine, which contains sulfhydryl groups to which the toxic metabolite can bind, is an antidote in cases of overdose.
✍There are three drugs of NSAID Safe in peptic ulcer pateints:-
1)Paracetamol
2)Ibuprofen
3)Meloxicam&celecoxib.
1)Paracetamol
2)Ibuprofen
3)Meloxicam&celecoxib.
سلااام الله عليكم
بكرة معانا معمل فارما بيكون بعد المجموعة الثانية
سيتم توفير كل الادوية للكل
وانتم عليكم الارانب
#وشكرا
بكرة معانا معمل فارما بيكون بعد المجموعة الثانية
سيتم توفير كل الادوية للكل
وانتم عليكم الارانب
#وشكرا
💥Ketorolac is notable as a drug used mainly as a systemic analgesic, not as an anti-inflammatory.
💥It is the only NSAID available in a parenteral formulation.
💥has greater analgesic effectiveness.
💥It is the only NSAID available in a parenteral formulation.
💥has greater analgesic effectiveness.
🐰Nonselective NSAIDs reduce polyp formation in patients with primary familial adenomatous polyposis.
🐰Long-term use of NSAIDs reduces the risk of colon cancer.
🐰Long-term use of NSAIDs reduces the risk of colon cancer.
🤒The major difference between the mechanisms of action of aspirin and other NSAIDs is that aspirin (but not its active metabolite, salicylate) acetylates and thereby irreversibly inhibits cyclooxygenase, whereas the inhibition produced by other NSAIDs is reversible.
🤕The irreversible action of aspirin results in a longer duration of its antiplatelet effect and is the basis for its use as an antiplatelet drug.
🤕The irreversible action of aspirin results in a longer duration of its antiplatelet effect and is the basis for its use as an antiplatelet drug.
😔Aspirin or salicylate toxicity lead to death by respiratory failure.
👽Acetaminophen (Paracetamol) toxicity lead to death by liver failure.
👽Acetaminophen (Paracetamol) toxicity lead to death by liver failure.
💥Celecoxib is a COX-2-selective inhibitor. Although the COX-2 inhibitors have the advantage over nonselective NSAIDs of reduced gastrointestinal toxicity, clinical data suggest that they are more likely to cause arterial thrombotic events.
☄A history of myocardial infarction would be a compelling reason to avoid a COX-2 inhibitor.
☄A history of myocardial infarction would be a compelling reason to avoid a COX-2 inhibitor.
💥Aspirin an irreversible COX inhibitor, is particularly effective. Because platelets lack the machinery for synthesis of new protein, inhibition by aspirin persists for several days until new platelets are formed.
Other NSAIDs, which cause a less persistent antiplatelet effect (hours), are not used as antiplatelet drugs and, in fact, can interfere with the antiplatelet effect of aspirin when used in combination with aspirin.
Other NSAIDs, which cause a less persistent antiplatelet effect (hours), are not used as antiplatelet drugs and, in fact, can interfere with the antiplatelet effect of aspirin when used in combination with aspirin.