#STD_in_right_vent_hypertrophy
right ventricular hypertrophy (RVH) causes ST depression and T-wave inversion in the right precordial leads V1-3.
right ventricular hypertrophy (RVH) causes ST depression and T-wave inversion in the right precordial leads V1-3.
#STD_IN_SVT:_
Supraventricular tachycardia (e.g. AVNRT) typically causes widespread horizontal ST depression, most prominent in the left precordial leads (V4-6).
This rate-related ST depression does not necessarily indicate the presence of myocardial ischaemia, provided that it resolves with treatment
Supraventricular tachycardia (e.g. AVNRT) typically causes widespread horizontal ST depression, most prominent in the left precordial leads (V4-6).
This rate-related ST depression does not necessarily indicate the presence of myocardial ischaemia, provided that it resolves with treatment
#ST_depression_in_hypokalemia:_
Hypokalaemia causes widespread downsloping ST depression with T-wave flattening/inversion, prominent U waves and a prolonged QU interval
Hypokalaemia causes widespread downsloping ST depression with T-wave flattening/inversion, prominent U waves and a prolonged QU interval
#Morphology_of_ST_Depression
ST depression can be either upsloping, downsloping, or horizontal.
#Horizontal or #downsloping ST depression ≥ 0.5 mm at the J-point in ≥ 2 contiguous leads indicates myocardial ischaemia (according to the 2007 Task Force Criteria).
#Upsloping ST depression in the precordial leads with prominent De Winter T waves is highly specific for occlusion of the LAD.
Reciprocal change has a morphology that resembles “upside down” ST elevation and is seen in leads electrically opposite to the site of infarction.
#Posterior MI manifests as horizontal ST depression in V1-3 and is associated with upright T waves and tall R waves.
ST depression can be either upsloping, downsloping, or horizontal.
#Horizontal or #downsloping ST depression ≥ 0.5 mm at the J-point in ≥ 2 contiguous leads indicates myocardial ischaemia (according to the 2007 Task Force Criteria).
#Upsloping ST depression in the precordial leads with prominent De Winter T waves is highly specific for occlusion of the LAD.
Reciprocal change has a morphology that resembles “upside down” ST elevation and is seen in leads electrically opposite to the site of infarction.
#Posterior MI manifests as horizontal ST depression in V1-3 and is associated with upright T waves and tall R waves.
#STD_in_digoxin_treatment:_
Treatment with digoxin causes downsloping ST depression with a “sagging” morphology, reminiscent of Salvador Dali’s moustache
Treatment with digoxin causes downsloping ST depression with a “sagging” morphology, reminiscent of Salvador Dali’s moustache
🫀 100 Echo Signs Every Cardiologist Should Know
B-BUMP SIGN
📈 A classic M-mode clue to elevated LV filling pressure
🔍 WHAT IS THE B-BUMP?
The B-bump is an abnormal late-diastolic configuration of the mitral valve seen on M-mode echocardiography.
It appears as a small bump or plateau between the A and C points, with the mitral valve remaining partially open before closure.
⏱️ WHEN DOES IT OCCUR?
⚠️ The B-bump is a LATE-DIASTOLIC phenomenon.
It occurs after the A point and before mitral valve closure (C point).
🔍 WHY DOES IT OCCUR?
⬆️ LV end-diastolic pressure ⬇️ Transmitral pressure gradient during late diastole ➡️ Mitral leaflets remain partially open ➡️ Abnormal late-diastolic leaflet motion ➡️ B-bump
❤️ CLINICAL ASSOCIATIONS
The B-bump has been described in:
🔸 Elevated LVEDP
🔸 Reduced LV compliance
🔸 Severe LV systolic dysfunction
🔸 Ischemic cardiomyopathy
🔸 Dilated cardiomyopathy
💡 ECHO PEARL
📈 B-BUMP = LATE-DIASTOLIC MITRAL LEAFLET ABNORMALITY
B-BUMP SIGN
📈 A classic M-mode clue to elevated LV filling pressure
🔍 WHAT IS THE B-BUMP?
The B-bump is an abnormal late-diastolic configuration of the mitral valve seen on M-mode echocardiography.
It appears as a small bump or plateau between the A and C points, with the mitral valve remaining partially open before closure.
⏱️ WHEN DOES IT OCCUR?
⚠️ The B-bump is a LATE-DIASTOLIC phenomenon.
It occurs after the A point and before mitral valve closure (C point).
🔍 WHY DOES IT OCCUR?
⬆️ LV end-diastolic pressure ⬇️ Transmitral pressure gradient during late diastole ➡️ Mitral leaflets remain partially open ➡️ Abnormal late-diastolic leaflet motion ➡️ B-bump
❤️ CLINICAL ASSOCIATIONS
The B-bump has been described in:
🔸 Elevated LVEDP
🔸 Reduced LV compliance
🔸 Severe LV systolic dysfunction
🔸 Ischemic cardiomyopathy
🔸 Dilated cardiomyopathy
💡 ECHO PEARL
📈 B-BUMP = LATE-DIASTOLIC MITRAL LEAFLET ABNORMALITY
ECG diagnosis: Acute myocardial injury in the setting of complete left bundle branch block (LBBB).
Key findings:
- Sinus rhythm
- Complete LBBB with left-axis deviation
- Concordant ST-segment elevation in the anterior precordial leads
- Concordant ST elevation in a patient with ischemic symptoms is highly concerning for acute coronary occlusion
Key findings:
- Sinus rhythm
- Complete LBBB with left-axis deviation
- Concordant ST-segment elevation in the anterior precordial leads
- Concordant ST elevation in a patient with ischemic symptoms is highly concerning for acute coronary occlusion
This is a high-risk ECG. LBBB alone does not establish acute MI, but concordant ST-segment elevation markedly increases concern for acute myocardial infarction.
Clinical context is critical: severe ongoing ischemic symptoms plus these ECG findings should prompt immediate ACS evaluation and cardiology/reperfusion pathway activation.
Clinical context is critical: severe ongoing ischemic symptoms plus these ECG findings should prompt immediate ACS evaluation and cardiology/reperfusion pathway activation.
🔥1