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May 3, 2013
my list of peer reviewed vaccine research
• 57 Comments
This list is just a thrown together list and is pretty helter skelter..but, there are a lot of links to lead you down the research path if you are searching. There are are so many, many, many more out there that haven’t made it to this list. They sit and wait for me to find them..i better get to looking.. May our truth digging be successful!
Hypothesis: conjugate vaccines may predispose children to autism spectrum disorders.
the potential effects of conjugate vaccines on neural development merit close examination. Conjugate vaccines fundamentally change the manner in which the immune systems of infants and young children function by deviating their immune responses to the targeted carbohydrate antigens from a state of hypo-responsiveness to a robust B2 B cell mediated response. This period of hypo-responsiveness to carbohydrate antigens coincides with the intense myelination process in infants and young children, and conjugate vaccines may have disrupted evolutionary forces that favored early brain development over the need to protect infants and young children from capsular bacteria.
http://www.ncbi.nlm.nih.gov/pubmed/21993250
Serological association of measles virus and human herpesvirus-6 with brain autoantibodies in autism.
Considering an autoimmunity and autism connection, brain autoantibodies to myelin basic protein (anti-MBP) and neuron-axon filament protein (anti-NAFP) have been found in autistic children. In this current study, we examined associations between virus serology and autoantibody by simultaneous analysis of measles virus antibody (measles-IgG), human herpesvirus-6 antibody (HHV-6-IgG), anti-MBP, and anti-NAFP. We found that measles-IgG and HHV-6-IgG titers were moderately higher in autistic children but they did not significantly differ from normal controls. Moreover, we found that a vast majority of virus serology-positive autistic sera was also positive for brain autoantibody: (i) 90% of measles-IgG-positive autistic sera was also positive for anti-MBP; (ii) 73% of measles-IgG-positive autistic sera was also positive for anti-NAFP; (iii) 84% of HHV-6-IgG-positive autistic sera was also positive for anti-MBP; and (iv) 72% of HHV-6-IgG-positive autistic sera was also positive for anti-NAFP. This study is the first to report an association between virus serology and brain autoantibody in autism; it supports the hypothesis that a virus-induced autoimmune response may play a causal role in autism.
http://www.ncbi.nlm.nih.gov/pubmed/9756729
Effectiveness of pertussis vaccines for adolescents and adults: case-control study
The adjusted estimate of effectiveness of Tdap vaccination against pertussis was 53.0.
http://www.bmj.com/content/347/bmj.f4249
Neurologic Adverse Events Following Vaccination (Progress in Health Sciences Vol. 2(1) 2012•pp 129-141.)
“Conclusions: Despite the assurances of the necessity and safety of vaccinations, there are more and more questions and doubts, which both physicians and parents are waiting to be clarified… It seems that it would be worthwhile to apply the precautionary principle – the ethical principle (from 1988) according to which if there is a probable, although poorly known, risk of adverse effects of new technology, it is better not to implement it rather than risk uncertain but potentially very harmful consequences.”
http://progress.umb.edu.pl/sites/progress.umb.edu.pl/files/129-141.pdf
Abnormal measles-mumps-rubella antibodies and CNS autoimmunity in children with autism.

“Thus the MMR antibody in autistic sera detected measles HA protein, which is unique to the measles subunit of the vaccine. Furthermore, over 90% of MMR antibody-positive autistic sera were also positive for MBP autoantibodies, suggesting a strong association between MMR and CNS autoimmunity in autism. Stemming from this evidence, we suggest that an inappropriate antibody response to MMR, specifically the measles component thereof, might be related to pathogenesis of autism.”
http://www.ncbi.nlm.nih.gov/pubmed/12145534
 Influenza: marketing vaccine by marketing disease
Closer examination of influenza vaccine policies shows that although proponents employ the rhetoric of science, the studies underlying the policy are often of low quality, and do not substantiate officials’ claims. The vaccine might be less beneficial and less safe than has been claimed, and the threat of influenza appears overstated.
http://www.bmj.com/content/346/bmj.f3037
An unmasking phenomenon in an observational post-licensure safety study of adolescent girls and young women.
Our recent experience in a post-licensure safety study of autoimmune conditions following the quadrivalent human papillomavirus vaccine in 189,629 girls and young women ages 9-26 years led us to question the adequacy of the exclusion of Day 0 events to prevent the erroneous association of prevalent conditions with vaccination. Of the 18 confirmed cases of Graves’ disease diagnosed in days 1-60 following vaccination, only 6 cases appeared to be truly new onset. Among the remaining 12 cases, 2 cases had abnormal thyroid stimulating hormone or thyroxine labs drawn prior to or on Day 0 but had no documented pre-existing symptoms. The other 10 cases had mention of symptoms of hyperthyroidism referencing a period prior to first HPV-4 dose. This ‘unmasking’ phenomenon, due to health care visits that include vaccination and new workups of preexisting symptoms, may not be adequately controlled through the exclusion of Day 0 events.
http://www.ncbi.nlm.nih.gov/m/pubmed/22580356/
 
How aluminum, an intracellular ROS generator promotes hepatic and neurological diseases: the metabolic tale
Metal pollutants are a global health risk due to their ability to contribute to a variety of diseases. Aluminum (Al), a ubiquitous environmental contaminant is implicated in anemia, osteomalacia, hepatic disorder, and neurological disorder. In this review, we outline how this intracellular generator of reactive oxygen species (ROS) triggers a metabolic shift towards lipogenesis in astrocytes and hepatocytes. This Al-evoked phenomenon is coupled to diminished mitochondrial activity, anerobiosis, and the channeling of α-ketoacids towards anti-oxidant defense. The resulting metabolic reconfiguration leads to fat accumulation and a reduction in ATP synthesis, characteristics that are common to numerous medical disorders. Hence, the ability of Al toxicity to create an oxidative environment promotes dysfunctional metabolic processes in astrocytes and hepatocytes. These molecular events triggered by Al-induced ROS production are the potential mediators of brain and liver disorders.”
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Waning of Maternal Antibodies Against Measles, Mumps, Rubella, and Varicella in Communities With Contrasting Vaccination Coverage
Conclusions: Children of mothers vaccinated against measles and, possibly, rubella have lower concentrations of maternal antibodies and lose protection by maternal antibodies at an earlier age than children of mothers in communities that oppose vaccination. This increases the risk of disease transmission in highly vaccinated populations.
http://jid.oxfordjournals.org/content/early/2013/04/29/infdis.jit143.full
“vaccine injury is so rare..don’t worry about it!” who has heard this?

“The Health Resources and Services Administration (HRSA) is publishing this notice of petitions received under the National Vaccine Injury Compensation Program.. A pet…ition may be filed with respect to injuries, disabilities, illnesses, conditions, and deaths resulting from vaccines described in the Vaccine Injury Table… Set forth below is a list of petitions received by HRSA on * March 13, 2013, through April 30, 2013.*”
[take note of #7 and #17..]
1. Tory J. and Sarah E. Moody on behalf of Victorya E. Moody, Bedford, Indiana, Court of Federal Claims No: 13-0190V.
2. Pamela Jean Peguess, Memphis, Tennessee, Court of Federal Claims No: 13-0191V.
3. Eileen Goeschel, Sarasota, Florida, Court of Federal Claims No: 13-0199V.
4. Kearsten Demczuk, Park Ridge, Illinois, Court of Federal Claims No: 13-0205V. 5. Howard Reddy and Hanan Tarabay on behalf of Andrew Howard Reddy, Pensacola, Florida, Court of Federal Claims No: 13-0208V.
6. Mona Marie Troup, Everett, Washington, Court of Federal Claims No: 13-0209V.
7. Angel Blackstone on behalf of S.B., Deceased, Trenton, New Jersey, Court of Federal Claims No: 13-0213V.
8. Isidra Durwin, Sarasota, Florida, Court of Federal Claims No: 13-0214V.
9. Nancy and Sandro Giannetta on behalf of A.M.G., Sarasota, Florida, Court of Federal Claims No: 13-0215V.
10. Kimberly Pedersen, West Allis, Wisconsin, Court of Federal Claims No: 13-0216V.
11. Charles and Jeannie Maikish on behalf of S.M., Nyack, New York,Court of Federal Claims No: 13-0217V.
12. Ina Scanlon, Muncie, Indiana, Court of Federal Claims No: 13-0219V.
13. David Stachlewitz on behalf of H.G.S., Glendale, Arizona, Court of Federal Claims No: 13-0220V.
14. Mary E. Thompson, Brookport, Illinois, Court of Federal Claims No: 13-0222V.
15. Matthew Gorski, Wynnewood, Pennsylvania, Court of Federal Claims No: 13-0224V.
16. Woodrow Coffey, Jr., Irvine, California, Court of Federal Claims No: 13-0225V. 17. Stephen Warren on behalf of Taylor Warren, Deceased, New York, New York, Court of Federal Claims No: 13-0226V.
18. Robert Wiggins, Nashville, North Carolina, Court of Federal Claims No: 13-0228V.
19. Peggy Kalmeyer, Depew, New York, Court of Federal Claims No: 13-0230V.
20. Rosemary and Wayne Trezza on behalf of P.T., West Orange, New Jersey, Court of Federal Claims No: 13-0231V.
21. Jane Tomassetti, Woodbury, Minnesota, Court of Federal Claims No: 13-0234V.
22. Everett Johnson, Sr., Ashland, Kentucky, Court of Federal Claims No: 13-0235V.
23. Edwin W. Fockler, Sarasota, Florida, Court of Federal Claims No: 13-0237V. 24. James Cox, Las Cruces, New Mexico, Court of Federal Claims No: 13-0238V. 25. Chanel and Paul A. Monroe on behalf of Angelina Monroe, Las Vegas, Nevada, Court of Federal Claims No: 13-0239V.
26. Noteel Koss, Houston, Texas, Court of Federal Claims No: 13-0240V.
27. Tamika M. Kratzer on behalf of Ian M. Kratzer, Sacramento, California, Court of Federal Claims No: 13-0243V.
28. Rosalie Peck, Boston, Massachusetts, Court of Federal Claims No: 13-0249V. 29. Shannon Keller, Sacramento, California, Court of Federal Claims No: 13-0250V.
30. Edwina Bradshaw, North Myrtle Beach, North Carolina, Court of Federal Claims No: 13-0252V.
31. William and Brenda Lehann Rodriguez on behalf of C.R., Clayton, Georgia, Court of Federal Claims No: 13-0253V.
32. Corrine K. Ibana, Kamuela, Hawaii, Court of Federal Claims No: 13-0257V. 33. Lorel Cubano, San Juan, Puerto Rico, Court of Federal Claims No: 13-0259V. 34. Brittany and Davey Lambert on behalf of Noah Lambert, Memphis, Tennessee, Court of Federal Claims No: 13-0265V.
35. Scott and Caroline VanScoy on behalf of Alyssa VanScoy, Simi Valley, California, Court of Federal Claims No: 13-0266V.
36. Jane Sprecher, Reading, Pennsylvania, Court of Federal Claims No: 13-0271V.
37. Georgia Murdock, Silver Spring, Maryland, Court of Federal Claims No: 13-0273V.
38. Willie Andre Simmons, Augusta, Georgia, Court of Federal Claims No: 13-0274V.
39. Jung Park, M.D., New York, New York, Court of Federal Claims No: 13-0275V. 40.

Allison and Steven Council on behalf of Adam Council, Plainfield, Illinois, Court of Federal Claims No: 13-0276V.
41. Maryann Giordano, Lindenhurst, New York, Court of Federal Claims No: 13-0277V.
42. Laura A. Jones, Greensboro, North Carolina, Court of Federal Claims No: 13-0279V.
43. David D. Griffin, Afghanistan, Court of Federal Claims No: 13-0280V.
44. James Demoski, Endicott, New York, Court of Federal Claims No: 13-0286V. 45. Christina N. Steinat, Seattle, Washington, Court of Federal Claims No: 13-0287V.
46. Jessica L. Stone, Baraboo, Wisconsin, Court of Federal Claims No: 13-0289V. 47. Holly Rhew, Wichita, Kansas, Court of Federal Claims No: 13-0293V.
48. Janet DeYear, Dallas, Texas, Court of Federal Claims No: 13-0299V.
49. Cynthia Adkins, Sarasota, Florida, Court of Federal Claims No: 13-0295V.
50. Saurabh V. and Archana Amin on behalf of Sheaa Amin, Linwood, New Jersey, Court of Federal Claims No: 13-0300V.
51. Juliet and Mohamed Edoo on behalf of Justin Edoo, Miami, Florida, Court of Federal Claims No: 13-0302V.
52. James Boyer, Boston, Massachusetts, Court of Federal Claims No: 13-0303V.
*these are from March 13, 2013 – April 30, 2013. 48 days. what is the true number that these 52 petitions represent? how many don’t file claims? think about it..its scary. I wish we could see more about these petitions..more about the injury caused.It is impossible for a parent to make a solid risk/benefit analysis when it comes to vaccinations.. I don’t care what anyone may say.. vaccine injury is downplayed and pushed aside, disease rates and risks are over exaggerated and blasted throughout the media via mass scare campaigns (remember those 8 measly cases of the measles in Wales during the month of march 2013?) ..and natural and safe preventative measures and treatments are suppressed. How are we supposed to make an informed medical decision when it comes to our children being injected with almost 50 doses of 16 vaccines before the age of 6?
https://www.federalregister.gov/articles/2013/05/24/2013-12347/national-vaccine-injury-compensation-program-list-of-petitions-received?utm_content=next&utm_medium=PrevNext&utm_source=Article
“In 1990, infants received a total of 15 vaccine doses prior to their first year of life: 3 DPT injections (9 vaccine doses), 3 polio, and 3 Hib vaccines—5 vaccine doses at 2, 4, and 6 months of age. By 2007, the CDC recommended 26 vaccine doses for infants: 3 DTaP, 3 polio, 3 Hib, 3 hepatitis B, 3 pneumococcal, 3 rotavirus, and 2 influenza vaccines. While each childhood vaccine has individually undergone clinical trials to assess safety, studies have not been conducted to determine the safety (or efficacy) of combining vaccines during a single physician visit as recommended by CDC guidelines. For example, 2-, 4-, and 6-month-old infants are expected to receive vaccines for polio, hepatitis B, diphtheria, tetanus, pertussis, rotavirus, Haemophilus influenzae type B, and pneumococcal, all during a single well-baby visit—even though this combination of 8 vaccine doses was never tested in clinical trials.
An article written by Guess, representing a vaccine manufacturer, claimed that it is “impractical to conduct preapproval studies of all combinations [of vaccines] in clinical practice.”1 However, a recent study by Miller and Goldman found that among the developed nations, infant mortality increased with an increase in the number of vaccine doses.2 Similar associations have also been found with respect to other serious adverse outcomes. Delong reported that the higher the proportion of children receiving recommended vaccinations, the higher the prevalence of autism or speech and language impairment.3 A CDC report on mixed exposures to chemical substances and other stressors, including prescribed pharmaceuticals, found that they may produce “increased or unexpected deleterious health effects.” In addition, “exposures to mixed stressors can produce health consequences that are additive, synergistic, antagonistic, or can potentiate the response expected from individual component exposures.

”4 Administering six, seven, or eight vaccine doses to an infant during a single physician visit may certainly be more convenient for parents—rather than making additional trips to the doctor’s office—but evidence of a positive association between infant adverse reactions and the number of vaccine doses administered confirms that vaccine safety must remain the highest priority”
http://het.sagepub.com/content/31/10/1012.full
“Maternal transfer of mercury to the developing embryo/fetus: is there a safe level?”
“This study focused on standardized embryonic and fetal Hg exposures via primary exposure to the pregnant mother of two common Hg sources (dietary fish and parenteral vaccines). Data demonstrated that Hg exposures, particularly during the first trimester of pregnancy, at well-established dose/weight ratios produced severe damage to humans including death. . In light of research suggestive of a mercuric risk factor for childhood conditions such as tic disorders, cerebral palsy, and autism, it is essential that Hg advisories account for secondary prenatal human exposures.”
http://www.tandfonline.com/doi/full/10.1080/02772248.2012.724574
Empirical Data Confirm Autism Symptoms Related to Aluminum and Acetaminophen Exposure
“Autism is a condition characterized by impaired cognitive and social skills, associated with compromised immune function. The incidence is alarmingly on the rise, and environmental factors are increasingly suspected to play a role. This paper investigates word frequency patterns in the U.S. CDC Vaccine Adverse Events Reporting System (VAERS) database. Our results provide strong evidence supporting a link between autism and the aluminum in vaccines. A literature review showing toxicity of aluminum in human physiology offers further support. Mentions of autism in VAERS increased steadily at the end of the last century, during a period when mercury was being phased out, while aluminum adjuvant burden was being increased. Using standard log-likelihood ratio techniques, we identify several signs and symptoms that are significantly more prevalent in vaccine reports after 2000, including cellulitis, seizure, depression, fatigue, pain and death, which are also significantly associated with aluminum-containing vaccines. We propose that children with the autism diagnosis are especially vulnerable to toxic metals such as aluminum and mercury due to insufficient serum sulfate and glutathione. A strong correlation between autism and the MMR (Measles, Mumps, Rubella) vaccine is also observed, which may be partially explained via an increased sensitivity to acetaminophen administered to control fever.”
http://www.mdpi.com/1099-4300/14/11/2227
full text: http://groups.csail.mit.edu/sls/publications/2012/entropy-14-02227.pdf
Acetaminophen use after measles-mumps-rubella vaccination was SIGNIFICANTLY associated with autistic disorder when considering children 5 years of age or less, after limiting cases to children with regression in development and when considering only children who had post-vaccination sequelae adjusting for age, gender, mother’s ethnicity, and the presence of illness concurrent with measles-mumps-rubella vaccination. Ibuprofen use after measles-mumps-rubella vaccination was not associated with autistic disorder. This preliminary study found that acetaminophen use after measles-mumps-rubella vaccination was associated with autistic disorder.
http://www.ncbi.nlm.nih.gov/pubmed/18445737
A 1% increase in vaccination was associated with an additional 680 children having AUT or SLI. Neither parental behavior nor access to care affected the results, since vaccination proportions were not significantly related (statistically) to any other disability or to the number of pediatricians in a U.S. state. The results suggest that although mercury has been removed from many vaccines, other culprits may link vaccines to autism. Further study into the relationship between vaccines and autism is warranted”
http://www.ncbi.nlm.nih.gov/pubmed/21623535

Ten to 12 days post 18 month vaccination, the relative incidence was 1.25 (95%, 1.17–1.33) which represented at least one excess event for every 730 children vaccinated. The primary reason for increased events was statistically significant elevations in emergency room visits following all vaccinations. There were non-significant increases in hospital admissions. There were an additional 20 febrile seizures for every 100,000 vaccinated at 12 months.
Conclusions
There are significantly elevated risks of primarily emergency room visits approximately one to two weeks following 12 and 18 month vaccination. Future studies should examine whether these events could be predicted or prevented
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3236196/
“Administration of thimerosal to infant rats increases overflow of glutamate and aspartate in the prefrontal cortex: protective role of dehydroepiandrosterone sulfate.  “
In summary, the present study documents that exposure of infant rats to THIM perturbs the balance between excitatory and inhibitory amino acids in the brain, shifting it toward excessive neuroexcitation. Despite of intrinsic limitations, present findings have important clinical implications, as they provide a plausible mechanism, whereby THIM exerts neurotoxic effects in the brain. It is likely that this mercurial—still present in pediatric vaccines in many countries—causes a similar disturbance of excitatory and inhibitory neurotransmitters in the brains of human infants, leading to neurotoxicity, encephalopaties, and in consequence to neurodevelopmental disorders, including autism..*On the whole, the current study provides further empirical evidence that exposure to THIM leads to neurotoxic changes in the developing brain, arguing for urgent and permanent removal of this preservative from all vaccines for children (and adults) since effective, less toxic and less costly alternatives are available. The stubborn insistence of some vaccine manufacturers and health agencies on continuation of use of this proven neurotoxin in vaccines is testimony of their disregard for both the health of young generations and for the environment.*
http://www.ncbi.nlm.nih.gov/pubmed/22015977
“Thus vaccination DOES NOT account for the impressive declines in mortality seen in the first half of the century”
http://pediatrics.aappublications.org/content/106/6/1307.abstract
Thimerosal, an organomercurial added as a preservative to some vaccines, is a suspected iatrogenic factor, possibly contributing to paediatric neurodevelopmental disorders including autism. We examined the effects of early postnatal administration of thimerosal (four i.m. injections, 12 or 240 μg THIM-Hg/kg, on postnatal days 7, 9, 11 and 15) on brain pathology in Wistar rats. Numerous neuropathological changes were observed in young adult rats which were treated postnatally with thimerosal. They included: ischaemic degeneration of neurons and “dark” neurons in the prefrontal and temporal cortex, the hippocampus and the cerebellum, pathological changes of the blood vessels in the temporal cortex, diminished synaptophysin reaction in the hippocampus, atrophy of astroglia in the hippocampus and cerebellum, and positive caspase-3 reaction in Bergmann astroglia. These findings document neurotoxic effects of thimerosal, at doses equivalent to those used in infant vaccines or higher, in developing rat brain, suggesting likely involvement of this mercurial in neurodevelopmental disorders.
http://www.ncbi.nlm.nih.gov/pubmed/21225508
and it’s the unvaccinated that are spreading pertussis?
“Despite widespread vaccination, whooping cough incidence is on the rise worldwide, making it the only vaccine-preventable disease associated with increasing deaths in the United States. Although this disease is most often attributed to Bordetella pertussis infection, it is also caused by the closely related pathogen, B. parapertussis. However, B. pertussis has remained the center of attention, whereas B. parapertussis has been greatly overlooked in the development of whooping cough vaccines.

“Furthermore, while India has been polio-free for a year, there has been a huge increase in non-polio acute flaccid paralysis (NPAFP). In 2011, there were an extra 47,500 new cases of NPAFP. Clinically indistinguishable from polio paralysis but twice as deadly, the incidence of NPAFP was directly proportional to doses of oral polio received. Though this data was collected within the polio surveillance system, it was not investigated.”
http://www.ncbi.nlm.nih.gov/pubmed/22591873
Detection of fecal shedding of rotavirus vaccine in infants following their first dose of pentavalent rotavirus vaccine. (and how they blame everything on kids that are not vaccinated is beyond me! These vaccines are helping to keep diseases in circulation..)
Studies on rotavirus vaccine shedding and its potential transmission within households including immunocompromised individuals are needed to better define the potential risks and benefits of vaccination. We examined fecal shedding of pentavalent rotavirus vaccine (RV5) for 9 days following the first dose of vaccine in infants between 6 and 12 weeks of age. Rotavirus antigen was detected by enzyme immunoassay (EIA), and vaccine-type rotavirus was identified by nucleotide sequencing based on genetic relatedness to the RV5 VP6 gene. Stool from 22 (21.4%) of 103 children contained rotavirus antigen-positive specimens on ≥ 1 post-vaccination days. Rotavirus antigen was detected as early as post-vaccination day 3 and as late as day 9, with peak numbers of shedding on post-vaccination days 6 through 8. Vaccine-type rotavirus was detected in all 50 antigen-positive specimens and 8 of 8 antigen-negative specimens. Nine (75%) of 12 EIA-positive and 1 EIA-negative samples tested culture-positive for vaccine-type rotavirus. Fecal shedding of rotavirus vaccine virus after the first dose of RV5 occurred over a wide range of post-vaccination days not previously studied. These findings will help better define the potential for horizontal transmission of vaccine virus among immunocompromised household contacts of vaccinated infants for future studies
http://www.ncbi.nlm.nih.gov/pubmed/21477676
“Effectiveness of trivalent inactivated influenza vaccine in influenza-related hospitalization in children: a case-control study.”
“Using the Cochran-Mantel-Haenszel test for asthma status stratification, there was a significant association between hospitalization in asthmatic subjects and TIV (p = 0.001). TIV did not provide any protection against hospitalization in pediatric subjects, especially children with asthma. On the contrary, we found a threefold increased risk of hospitalization in subjects who did get the TIV vaccine. This may be a reflection not only of vaccine effectiveness but also the population of children who are more likely to get the vaccine.”
http://www.ncbi.nlm.nih.gov/pubmed/22525386
The odds of having a history of asthma was twice as great among vaccinated subjects than among unvaccinated subjects (adjusted odds ratio, 2.00; 95% confidence interval, 0.59 to 6.74). The odds of having had any allergy-related respiratory symptom in the past 12 months was 63% greater among vaccinated subjects than unvaccinated subjects (adjusted odds ratio, 1.63; 95% confidence interval, 1.05 to 2.54). The associations between vaccination and subsequent allergies and symptoms were greatest among children aged 5 through 10 years.
DTP or tetanus vaccination appears to increase the risk of allergies and related respiratory symptoms in children and adolescents. Although it is unlikely that these results are entirely because of any sources of bias, the small number of unvaccinated subjects and the study design limit our ability to make firm causal inferences about the true magnitude of effect.
http://www.ncbi.nlm.nih.gov/pubmed/10714532
Four to 12 days post 12 month vaccination, children had a 1.33 (1.29–1.38) increased relative incidence of the combined endpoint compared to the control period, or at least one event during the risk interval for every 168 children vaccinated.

vaccination led to a 40-fold enhancement of B. parapertussis colonization in the lungs of mice.. these data suggest that the vaccine may be contributing to the observed rise in whooping cough incidence over the last decade by promoting B. parapertussis infection.”
http://www.cidd.psu.edu/research/synopses/acellular-vaccine-enhancement-b.-parapertussis
Despite widespread childhood vaccination against Bordetella pertussis, disease remains prevalent. It has been suggested that acellular vaccine may be less effective than previously believed. During a large outbreak, we examined the incidence of pertussis and effectiveness of vaccination in a well-vaccinated, well-defined community.. Our data suggests that the current schedule of acellular pertussis vaccine doses is insufficient to prevent outbreaks of pertussis.
http://cid.oxfordjournals.org/content/early/2012/03/13/cid.cis287.short
In the last 3 decades, there has been an unexplained increase in the prevalence of asthma and hay fever.
OBJECTIVE:
We sought to determine whether there is an association between childhood vaccination and atopic diseases, and we assessed the self-reported prevalence of atopic diseases in a population that included a large number of families not vaccinating their children.
RESULTS:
The data included 515 never vaccinated, 423 partially vaccinated, and 239 completely vaccinated children. In multiple regression analyses there were significant ( P < .0005) and dose-dependent negative relationships between vaccination refusal and self-reported asthma or hay fever only in children with no family history of the condition and, for asthma, in children with no exposure to antibiotics during infancy. Vaccination refusal was also significantly ( P < .005) and negatively associated with self-reported eczema and current wheeze. A sensitivity analysis indicated that substantial biases would be required to overturn the observed associations.
CONCLUSION:
Parents who refuse vaccinations reported less asthma and allergies in their unvaccinated children. Although this relationship was independent of measured confounders, it could be due to differences in other unmeasured lifestyle factors or systematic bias. Further research is needed to verify these results and investigate which exposures are driving the associations between vaccination refusal and allergic disease..
http://www.ncbi.nlm.nih.gov/pubmed/15805992
“Unvaccinated children tended to be white, to have a mother who was married and had a college degree, to live in a household with an annual income exceeding $75,000 dollars, and to have parents who expressed concerns regarding the safety of vaccines and indicated that medical doctors have little influence over vaccination decisions for their children.”
http://www.ncbi.nlm.nih.gov/pubmed/15231927
Although persons often use vaccination and immunization interchangeably in reference to active immunization, the terms are not synonomous because the administration of an immunobiologic cannot be automatically equated with the development of adequate immunity.
http://wonder.cdc.gov/wonder/prevguid/p0000348/p0000348.asp#head002000000000000
“Virus-induced autoimmunity may play a causal role in autism. To examine the etiologic link of viruses in this brain disorder, we conducted a serologic study of measles virus, mumps virus, and rubella virus. Viral antibodies were measured by enzyme-linked immunosorbent assay in the serum of autistic children, normal children, and siblings of autistic children. The level of measles antibody, but not mumps or rubella antibodies, was significantly higher in autistic children as compared with normal children (P = 0.003) or siblings of autistic children (P <or= 0.0001). Furthermore, immunoblotting of measles vaccine virus revealed that the antibody was directed against a protein of approximately 74 kd molecular weight. The antibody to this antigen was found in 83% of autistic children but not in normal children or siblings of autistic children.

Thus autistic children have a hyperimmune response to measles virus, which in the absence of a wild type of measles infection might be a sign of an abnormal immune reaction to the vaccine strain or virus reactivation.”
http://www.ncbi.nlm.nih.gov/pubmed/12849883
We do not vaccinate against yellow fever in the US but this still is of importance because it shows that things like this can and HAVE happened.
“However, in 2001, the vaccine was found to cause a serious, frequently fatal, multisystemic illness, called yellow fever vaccine–associated viscerotropic disease (YEL-AVD), which resembles the illness it was designed to prevent (1–3). ”
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3310656/
Effectiveness of Influenza Vaccine in the Community-Dwelling Elderly
“Most high-risk medical conditions that were measured were more prevalent among vaccinated than among unvaccinated persons.”
http://jama.jamanetwork.com/article.aspx?articleid=189023
” The aim of this study was to compare the number of inactivated-influenza vaccine–related spontaneous abortion and stillbirth (SB) reports in the Vaccine Adverse Event Reporting System (VAERS) database during three consecutive flu seasons beginning 2008/2009 and assess the relative fetal death reports associated with the two-vaccine 2009/2010 season. The VAERS database was searched for reports of fetal demise following administration of the influenza vaccine/vaccines to pregnant women.. reporting bias was too low to explain the magnitude increase in fetal-demise reporting rates in the VAERS database relative to the reported annual trends. Thus, a synergistic fetal toxicity likely resulted from the administration of both the pandemic (A-H1N1) and seasonal influenza vaccines during the 2009/2010 season.”
http://het.sagepub.com/content/early/2012/09/12/0960327112455067.abstract
Hepatitis B vaccine might be followed by various rheumatic conditions and might trigger the onset of underlying inflammatory or autoimmune rheumatic diseases.. Further epidemiological studies are needed to establish whether hepatitis B vaccination is associated or not with an incidence of rheumatic disorders higher than normal. A few cases of onset or reactivation of SLE after vaccination against hepatitis B have been described. The onset of symptoms occurred within 5 days–1 month after the immunization. Two patients had a lupus nephritis (associated in one with fever and arthralgia), one patient had pericarditis, one had thrombocytopenic purpura.. We observed four patients with myalgia and polyarthralgia, and, in three of them, fatigue following hepatitis B vaccination. These manifestations can be connected to the chronic fatigue syndrome. A few years ago, an independent working group agreed that there was no evidence of a cause–effect relationship between hepatitis B vaccine and chronic fatigue syndrome [37]. However, the number of patients followed up may have been too small to detect a slight increase in the relative risk.
Various other conditions following hepatitis B vaccination have been described. They include erythema nodosum and polyarthritis [21], erythema nodosum with arthralgia and Takayasu’s arteritis [38], vasculitis [39–41], polyarthritis associated with hypercalcaemia and lytic bone lesions [29].
http://rheumatology.oxfordjournals.org/content/38/10/978.long
“Autoimmunity to the central nervous system (CNS), especially to myelin basic protein (MBP), may play a causal role in autism, a neurodevelopmental disorder. Because many autistic children harbor elevated levels of measles antibodies, we conducted a serological study of measles-mumps-rubella (MMR) and MBP autoantibodies. Using serum samples of 125 autistic children and 92 control children, antibodies were assayed by ELISA or immunoblotting methods. ELISA analysis showed a significant increase in the level of MMR antibodies in autistic children. Immunoblotting analysis revealed the presence of an unusual MMR antibody in 75 of 125 (60%) autistic sera but not in control sera. This antibody specifically detected a protein of 73-75 kD of MMR.

This protein band, as analyzed with monoclonal anti bodies, was immunopositive for measles hemagglutinin (HA) protein but not for measles nucleoprotein and rubella or mumps viral proteins. Thus the MMR antibody in autistic sera detected measles HA protein, which is unique to the measles subunit of the vaccine. Furthermore, over 90% of MMR antibody-positive autistic sera were also positive for MBP autoantibodies, suggesting a strong association between MMR and CNS autoimmunity in autism. Stemming from this evidence, we suggest that an inappropriate antibody response to MMR, specifically the measles component thereof, might be related to pathogenesis of autism.”
http://www.ncbi.nlm.nih.gov/pubmed/12145534
http://vran.org/wp-content/documents/VRAN-Abnormal%20Measles-Mumps-Rubella-Antibodies-CNS-Autoimmunity-Children-Autism-Singh-Lin-Newell-Nelson.pdf
“Autoimmune hazards of hepatitis B vaccine”
“According to Hippocratic tradition, the safety level of a preventive medicine must be very high, as it is aimed at protecting people against diseases that they may not contract. This paper points out that information on the safety of hepatitis B vaccine (HBV) is biased as compared to classical requirements of evidence-based medicine (EBM), as exemplified by a documented selectivity in the presentation or even publication of available clinical or epidemiological data. Then, a review is made of data suggesting that HBV is remarkable by the frequency, the severity and the variety of its complications, some of them probably related to a mechanism of molecular mimicry leading to demyelinating diseases, and the others reproducing the spectrum of non-hepatic manifestations of natural hepatitis B. To be explained, this unusual spectrum of toxicity requires additional investigations based upon complete release of available data.
-More research is necessary and there is a need that the scientific community exerts pressure on health authorities to obtain that all existing data become available for peer-reviewed debate.
-There is an impressive convergence of data given credibility to a potential of this vaccine to induce severe and irreversible central demyelinating disorders.
-A number of clinical or epidemiological data on the safety hepatitis B vaccine (HBV) have not been published and do not seem to be.
-Modern vaccine research and development does not comply with basic requirements of evidence based medicine (EBM).”
http://www.ncbi.nlm.nih.gov/pubmed/15722255
FULL TEXT http://sanevax.org/wp-content/uploads/2011/02/autoimmune-hazards-hepB-vaccine1.pdf
“We find that ethylmercury not only inhibits mitochondrial respiration leading to a drop in the steady state membrane potential, but also concurrent with these phenomena increases the formation of superoxide, hydrogen peroxide, and Fenton/Haber-Weiss generated hydroxyl radical. These oxidants increase the levels of cellular aldehyde/ketones. Additionally, we find a five-fold increase in the levels of oxidant damaged mitochondrial DNA bases and increases in the levels of mtDNA nicks and blunt-ended breaks.. These mitochondria appear to have undergone a permeability transition, an observation supported by the five-fold increase in Caspase-3 activity observed after Thimerosal treatment.”
*the next time someone says that ethylmercury is ok for children ask them to read this article.
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3395253/
The main route of Al excretion is the urine; therefore, subjects with kidney malfunction or immature kidney, such as nephropathy patients or neonates, might experience toxic accumulation of Al in the body [12]. Infant formula is the primary food source for bottle-fed neonates. The study of Yuan et al reviewed several other studies and reported that most commercial infant formulas contained higher Al (6.5 μM to 87 μM) than human breast milk (0.2 μM to 1.7 μM) [12]. Infants display rapid growth and their brain-blood-barrier, detoxification system (liver), and excretory system (kidney) are not well-developed [13,14].

Aluminum can cross the blood-brain barrier and accumulate in glial and neural cells [15]. Thus, high intake of Al-containing formula might cause accumulation of Al in the neonatal brain, interfering with appropriate development.
In previous studies, exposure to excess dietary Al during gestation and lactation periods had no toxic effects on the mother, but resulted in persistent neurobehavioral deficits in the pups, such as defects in the sensory motor reflexes, locomotor activity, learning capability, and cognitive behavior [16,17]. These behavioral studies, therefore, suggested that Al exposure might cause developmental changes in neonatal brain. Until recently, a marker with which to effectively detect neonatal brain development was lacking. The group’s previous study with Al treatment in neonatal rat hippocampal neurons at concentrations of 37 μM and 74 μM for 14 days significantly reduced NMDAR (N-methyl-D-aspartate receptor) expression which was used as a marker of brain development. This suggested that Al exposure might influence the development of hippocampal neurons in neonatal rats.
http://www.jbiomedsci.com/content/19/1/51
The future of measles in highly immunized population. A modeling approach
However, despite short-term success in eliminating the disease, long-range projections demonstrate that the proportion of susceptibles in the year 2050 may be greater than in the prevaccine era. Present vaccine technology and public health policy must be altered to deal with this eventuality.
http://www.ncbi.nlm.nih.gov/pubmed/6741921
Summary
In conclusion, by apparently prioritizing vaccination policy over vaccine safety, the JCVI, the DH
and the Committee on Safety of Medicines (CSM) may have shown a disregard for the safety of
children. Through selective data reporting, the JCVI in conjunction with the DH, has promulgated
information relating to vaccine safety that may be inaccurate and potentially misleading, thereby
making it impossible for the parents to make a fully informed consent regarding vaccination.
Furthermore, by 1) apparently misleading patients about the true risks of adverse reactions as to
gain their consent for the administration of the treatment and 2) seemingly siding with vaccine
manufacturers rather than public health interests, the JCVI and the CSM appear to have signally
failed their fiduciary duty to protect individuals from vaccines of questionable safety. If these
provisional conclusions are indeed correct, then the information presented here may help us in
understanding the UK government’s and the JCVI’s official position on vaccine damage, that is, one
of persistent denial.
http://www.ecomed.org.uk/wp-content/uploads/2011/09/3-tomljenovic.pdf
“One way forward that appears to be favoured by most in the medical establishment is to continue to add more and more vaccines indiscriminately to the immunisation schedule in ever larger combinations. Just to question this policy is to be accused of putting children’s lives at risk and of being “anti-vaccine”. I have been called “anti-vaccine” even though I actually run a children’s immunisation clinic!
The government can;t bear any suggestion of lack of safety of vaccines. They will not even discuss it. I think they have a policy of suppression of any discussion on safety. This was said by a leading vaccine expert with the Cochrane Collaboration, a widely respected international not-for-profit and independent organisation, dedicated to making up-to-date and accurate information about the effects of health care readily available worldwide.
I would advocate another way forward: a more cautious approach incorporating honesty about the true benefits and risks of vaccination to enable parents to make a genuinely informed choice. I would like to see an environment in which parents are able to have a rational discussion without bullying, patronising, condescension and being accused of putting their child at risk.”
“Vaccine, Atopy and Allergy: Problems and Solutions”
http://www.ecomed.org.uk/wp-content/uploads/2011/09/2-halvorsen.pdf