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Forwarded from Medical Mnemonics
๐Ÿงฉ Medical Mnemonics


Hypokalemia causes


Eid Mubarak ๐ŸŒบ๐ŸŒ™ ๐ŸŽ†๐Ÿ•‹

โšกEnteric losses ( diarrhea)
โšกInsulin excess, Increase of Glucocorticoid (Cushing's, exogenous steroids, ectopic ACTH), Increased sweat losses
โšกDialysis, Decrease of body temperature (hypothermia)

โšกMagnesium depletion
โšกUrinary losses(Diuretics)
โšกฮฒ-adrenergic activity, Bartter's or Gitelman's syndrome
โšกAlkalosis, Amphotericin B
โšกRTA types I and II
โšกAldosteronism
โšกKetoacidosis

#nephrology
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ยฉMedical Mnemonics
โค25๐Ÿ‘6๐Ÿฅฐ1
Forwarded from Medical Mnemonics
๐Ÿงฉ Medical Mnemonics

Differential diagnosis of elevated serum aminotransferases


๐ŸŒ  โ€Œ๐Ÿ‡ฆโ€Œโ€Œ๐Ÿ‡งโ€Œโ€Œ๐Ÿ‡จโ€Œโ€Œ๐Ÿ‡ฉโ€Œโ€Œ๐Ÿ‡ชโ€Œโ€Œ๐Ÿ‡ซโ€Œโ€Œ๐Ÿ‡ฌโ€Œโ€Œ๐Ÿ‡ญโ€Œโ€Œ๐Ÿ‡ฎโ€Œ


๐Ÿชถ ๐—”utoimmune hepatitis, ๐—”drenal insufficiency, ๐—”norexia nervosa
๐Ÿชถ Hepatitis ๐—•
๐Ÿชถ Hepatitis ๐—–, ๐—–eliac disease
๐Ÿชถ ๐——rugs or toxins
๐Ÿชถ ๐—˜thanol
๐Ÿชถ ๐—™atty liver
๐Ÿชถ ๐—šenetic disorders (Wilson disease, Hemochromatosis, Alpha-1 antitrypsin deficiency)
๐Ÿชถ ๐—›emodynamic disorders (CHF and MI)
๐Ÿชถ ๐—œnfiltration of the liver by malignancy, ๐—œnjury to muscle (strenuous exercise, myopathy)

#gastroenterology
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ยฉMedical Mnemonics
๐Ÿ‘10โค4
Forwarded from Medical Mnemonics
On MCU RESEARCH COLLABORATION, we provide you:

- Language revision
- Scientific Editing
- Journal Selection
- Submitting the manuscript
- Guide to dealing with Journals

๐Ÿ‘Œ And Much More!

๐Ÿชฉ Feel Free to contact admin ๐Ÿ‘‰ @Mohamm_ADs
๐Ÿ‘1
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๐Ÿงฉ Medical Mnemonics

Learn ๐Ÿซง SOAP BUBBLE appearance in Giant cell tumor of bone by #visual_mnemonics.

๐Ÿ’ป Join us in the official Instagram page: Online Medical School

#radiology 
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ยฉMedical Mnemonics
๐Ÿ‘6โค1
Forwarded from EDL Backup Channel
โš  ๐Ÿ”” ๐’๐€๐•๐„ ๐“๐‡๐ˆ๐’ ๐‹๐ˆ๐’๐“ ๐…๐Ž๐‘ ๐€ ๐‘๐€๐ˆ๐๐˜ ๐ƒ๐€๐˜ ! โคต


1. ๐Ÿงฉ ๐— ๐—˜๐——๐—œ๐—–๐—”๐—Ÿ ๐— ๐—ก๐—˜๐— ๐—ข๐—ก๐—œ๐—–๐—ฆ (๐—Ÿ๐—˜๐—”๐—ฅ๐—ก ๐—˜๐—”๐—ฆ๐—œ๐—Ÿ๐—ฌ)

2. ๐—–๐—”๐—ฆ๐—˜ - ๐—•๐—”๐—ฆ๐—˜๐—— ๐— ๐—–๐—ค๐—ฆ ๐Ÿ’ฏ

3. ๐Ÿ‡จ๐Ÿ‡ฆ ๐— ๐—–๐—–๐—ค๐—˜ ๐—ฃ๐—ฅ๐—˜๐—ฃ๐—”๐—ฅ๐—”๐—ง๐—œ๐—ข๐—ก

4. ๐Ÿฉบ ๐—˜๐——๐—Ÿ ๐— ๐—˜๐——๐—œ๐—–๐—ข๐—ฆ (๐— ๐—˜๐——๐—œ๐—–๐—”๐—Ÿ ๐—•๐—ข๐—ข๐—ž๐—ฆ ๐—”๐—ก๐—— ๐—Ÿ๐—œ๐—ก๐—ž๐—ฆ)

5. ๐Ÿ“š ๐—˜๐——๐—Ÿ ๐—ฃ๐—›๐—”๐—ฅ๐— 

6. ๐Ÿ› ๐—ข๐—ก๐—Ÿ๐—œ๐—ก๐—˜ ๐— ๐—˜๐——๐—œ๐—–๐—”๐—Ÿ ๐—ฆ๐—–๐—›๐—ข๐—ข๐—Ÿ

7. ๐—ฅ๐—˜๐—ฆ๐—œ๐——๐—˜๐—ก๐—–๐—ฌ ๐—œ๐—ก ๐—š๐—˜๐—ฅ๐— ๐—”๐—ก๐—ฌ ๐Ÿ‡ฉ๐Ÿ‡ช

8. ๐—ฃ๐—ฅ๐—”๐—–๐—ง๐—œ๐—–๐—˜ ๐—œ๐—ก ๐—”๐—จ๐—ฆ๐—ง๐—ฅ๐—”๐—Ÿ๐—œ๐—” ๐Ÿ‡ฆ๐Ÿ‡บ

9. ๐— ๐—•๐—•๐—ฆ & ๐—ฅ๐—˜๐—ฆ๐—œ๐——๐—˜๐—ก๐—–๐—ฌ ๐—œ๐—ก ๐—œ๐—ง๐—”๐—Ÿ๐—ฌ ๐Ÿ‡ฎ๐Ÿ‡น

10. ๐—ฅ๐—˜๐—ฆ๐—œ๐——๐—˜๐—ก๐—–๐—ฌ ๐—œ๐—ก ๐—จ๐—ž ๐Ÿ‡ฌ๐Ÿ‡ง

11. ๐—ฅ๐—˜๐—ฆ๐—œ๐——๐—˜๐—ก๐—–๐—ฌ ๐—œ๐—ก ๐—จ๐—ฆ ๐Ÿ‡บ๐Ÿ‡ธ

12. ๐—ฅ๐—˜๐—ฆ๐—œ๐——๐—˜๐—ก๐—–๐—ฌ ๐—œ๐—ก ๐—–๐—”๐—ก๐—”๐——๐—” ๐Ÿ‡จ๐Ÿ‡ฆ

13. ๐—™๐—ฅ๐—˜๐—ก๐—–๐—› ๐— ๐—˜๐——๐—œ๐—–๐—”๐—Ÿ ๐—•๐—ข๐—ข๐—ž๐—ฆ ๐Ÿ‡ซ๐Ÿ‡ท

14. ๐—š๐—˜๐—ฅ๐— ๐—”๐—ก ๐— ๐—˜๐——๐—œ๐—–๐—”๐—Ÿ ๐—•๐—ข๐—ข๐—ž๐—ฆ ๐Ÿ‡ฉ๐Ÿ‡ช

15. ๐— ๐—˜๐——๐—œ๐—–๐—”๐—Ÿ ๐—ฅ๐—˜๐—ฆ๐—˜๐—”๐—ฅ๐—–๐—› ๐ŸŽ“

16. ๐Ÿ“ธ ๐——๐—˜๐—ฅ๐— ๐—”๐—ง๐—ข๐—Ÿ๐—ข๐—š๐—ฌ ๐—”๐—ง๐—Ÿ๐—”๐—ฆ

17. ๐—ข๐—˜๐—ง ๐—ฃ๐—ฅ๐—˜๐—ฃ๐—”๐—ฅ๐—”๐—ง๐—œ๐—ข๐—ก ๐Ÿ”ก

18. ๐— ๐—˜๐——๐—œ๐—–๐—”๐—Ÿ ๐—”๐— ๐—”๐—ญ๐—ข๐—ก ๐ŸŒ

19. ๐— ๐—˜๐——๐—œ๐—–๐—–๐—ข๐—จ๐—ก๐—ง - ๐— ๐—˜๐——๐—œ๐—–๐—”๐—Ÿ ๐—”๐—–๐—–๐—ข๐—จ๐—ก๐—ง ๐ŸŒ€

20. ๐Ÿ“ ๐—จ๐—ช๐—ข๐—ฅ๐—Ÿ๐—— ๐—˜๐——๐—จ๐—–๐—”๐—ง๐—œ๐—ข๐—ก๐—”๐—Ÿ ๐—ข๐—•๐—๐—˜๐—–๐—ง๐—œ๐—ฉ๐—˜๐—ฆ
๐Ÿ‘6โค2๐Ÿ‘1๐Ÿ˜1
Forwarded from Medical Mnemonics
- Do you want to publish your paper in the High Impact journal?

- Would you like your work to be seen in the best journals?


๐Ÿ”ท We will proceed with the journal publishing process with our professional team on MCU RESEARCH COLLABORATION

๐Ÿ”ป Feel Free to contact admin ๐Ÿ‘‰ @Mohamm_ADs
โค5
Forwarded from Medical Mnemonics
๐Ÿงฉ Medical Mnemonics


The 4 โ€Œ๐Ÿ‡ฆโ€Œโ€Œ's of Guillain-Barrรฉ
syndrome ๐Ÿ“

โœ– ๐—”cute inflammatory demyelinating
polyradiculopathy
โœ– ๐—”scending paralysis
โœ– ๐—”utonomic neuropathy
โœ– ๐—”lbuminocytologic dissociation (increased albumin in CSF)


#neurology
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ยฉMedical Mnemonics
๐Ÿ‘14โค1
Case-based MCQ pinned Deleted message
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๐Ÿงฉ Medical Mnemonics

Crigler-Najjar and Gilbert have problems with CoNjuGation of bilirubin while Dubin-Johnson and Rotor have a defective DooR for secretion of bilirubin.

#in_a_tweet
#pediatrics
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ยฉMedical Mnemonics
โค6๐Ÿ‘1
Forwarded from Medical Mnemonics
Today, Medical Channels Union (MCU) launches a new group for cardiology enthusiasts;

๐Ÿค“ Cardiology Cases is considering helping medical staff to become highly skilled in cardiology, particularly ECG interpretation.

๐Ÿ”— https://t.me/Cardiology_Cases

๐Ÿค We also welcome cardiologists from around the world to help us as group administrators.

Invite your Friends ๐Ÿ™‹โ€โ™‚ ๐Ÿฆ‹
๐Ÿ‘4โค2๐Ÿฅฐ2
A 40-year-old man is evaluated for palpitations in the emergency department. He has had these symptoms several times over the past year but this episode is worse. He denies any associated chest pain, lightheadedness, or syncope. He denies any history of medical problems and takes no medications. Examination reveals an adult male in no significant distress. Cardiac examination is significant for regular tachycardia with no jugular venous pressure elevation. Lungs are clear without crackles or wheezing. There is no lower extremity edema. ECG shown below. What is the best intervention at this point?


A. Valsalva maneuver
B. Adenosine
C. Metoprolol
D. Digoxin
E. Cardioversion
๐Ÿ‘17โค2๐Ÿ‘Ž1
๐Ÿ‘6
The correct answer is A.

Supraventricular tachycardias (SVT) include paroxysmal, reentry, or preexcitation tachycardias. Reentry SVTs include AV nodal reentry (AVNRT), atrioventricular reentry, or atrial reentry. Reentry circuits require the presence of at least two different conduction pathways with differential refractory times. It is characterized by an abrupt onset and termination of tachycardia, that distinguishes it from sinus tachycardia, which has gradual changes in rate. It is precipitated by a premature atrial or ventricular contraction or hyperadrenergic state. Other triggers include hyperthyroidism and stimulants, including caffeine, drugs, and alcohol. This patient has supraventricular tachycardia (SVT) likely related to atrioventricular nodal reentrant tachycardia (AVNRT) and he has no concerning symptoms.

The ECG shows a regular, fast rhythm with absent P waves and a narrow QRS complex. Unstable patients require immediate synchronized cardioversion. Stable patients, such as the patient above, should first undergo vagal maneuvers. Some common vagal maneuvers include holding your breath and bearing down (Valsalva maneuver), coughing, gagging, and immersing your face in ice-cold water. If vagal maneuvers are unsuccessful, adenosine is used both diagnostically and therapeutically. Adenosine transiently blocks the AV-node and allows the circuit to โ€œreset.โ€


โš Choice B is not correct:

Adenosine is very short acting and can be used if vagal maneuvers fail to terminate the arrhythmia.

โš Choice C is not correct:

Metoprolol would also be considered if the above measures failed.

โš Choice D is not correct:

Digoxin would also inhibit the AV node but has more potential side effects than the other medications and is rarely used for this purpose.

โš Choice E is not correct:

Cardioversion would be reserved for hemodynamic instability including hypotension, heart failure, or angina.

Summarized Points:

This patient has SVT likely related to AVNRT and he has no concerning symptoms. The initial attempts at termination should use vagal maneuvers such as the Valsalva maneuver.
๐Ÿ‘8โค2๐Ÿฅฐ1
โค1
A 36-year-old life-long asthmatic has repeated episodes of asthma exacerbation accompanied by fever and thick brownish sputum that respond to oral prednisone therapy. His leukocyte count is 8,500/ยตL with 15% eosinophils. Chest x-ray reveals bronchiectasis on the left side. Which of the following is the best next step in the management of this patient?

A. Bronchoscopy with bronchoalveolar lavage
B. Immunoglobulin levels to evaluate for immunodeficiency
C. Induced sputum for Pneumocystis jirovecii
D. Purified protein derivative test
E. Aspergillus skin testing
๐Ÿ‘8โค4
This patient with known asthma has recurrent exacerbations with fever and expectoration of dark brown mucus, which is suggestive of allergic bronchopulmonary aspergillosis (ABPA). Patients may experience additional systemic symptoms (e.g., anorexia, malaise, or weight loss) or have hemoptysis. ABPA is caused by hypersensitivity to bronchial colonization by Aspergillus fumigatus. It occurs more commonly in asthmatics and patients with cystic fibrosis. Proteolytic enzymes and mycotoxins induce an intense inflammatory reaction that can eventually cause bronchiectasis and fibrosis. Early diagnosis is important as the onset of bronchiectasis is associated with poor outcomes. Diagnosis is based on clinical and radiographic features and immunologic testing. Immediate cutaneous hypersensitivity to skin prick testing is a characteristic finding. If negative, intradermal reactivity may be tested for confirmation. A negative skin prick test and intradermal reactivity excludes the diagnosis. If positive, immunologic testing is performed. High-resolution CT characteristically shows infiltrates, central bronchiectasis, and mucus-filled bronchi (mucoid impaction). A positive bronchodilator response on pulmonary function tests is only seen in about 50% of patients. Treatment involves a combination of corticosteroids and antifungal agents (e.g., itraconazole).

โš  Choice A is not correct:

Bronchoalveolar lavage specimens may show Aspergillus species. However, the test is more invasive and is usually not indicated.

โš  Choice B is not correct:

Common variable immunodeficiency (CVID) can present with recurrent pulmonary infections and bronchiectasis, but it does not cause recurrent asthma exacerbations responsive to steroids.

โš  Choice C is not correct:

Induced sputum for Pneumocystis jirovecii may be helpful to diagnose Pneumocystis pneumonia in an asthmatic taking high-dose chronic oral steroids. However, this patient was only on intermittent steroids.

โš  Choice D is not correct:

Patients with tuberculosis may also present with chronic cough and constitutional symptoms, but these are progressive (not intermittent) and do not respond to steroids.

Summarized Points:

Allergic bronchopulmonary aspergillosis is a hypersensitivity reaction to Aspergillus that may colonize the airways of patients with asthma and cystic fibrosis. It presents with worsening asthma symptoms, fever, fleeting infiltrates, pleuritic chest pain, and peripheral eosinophilia. The first step in diagnosis is Aspergillus skin testing. If left untreated, the disorder can result in bronchiectasis with a poor prognosis.
๐Ÿ‘16โค3
A 49-year-old female presents to the emergency department with complaint of severe epigastric and right upper quadrant (RUQ) abdominal pain. Her symptoms started 3 days ago but have progressively worsened over the past 12 hours. Her symptoms are worse with meals. She has had two episodes of vomiting in the past 12 hours and is nauseous currently. Past medical history is significant for diabetes, osteoarthritis, and hypertension. Her blood pressure is 146/80 mmHg, pulse is 110/min, respiratory rate is 16/min, temperature is 39 C (102.3 F). On physical examination, there is severe right upper quadrant pain on deep palpation, most pronounced on palpation after deep inspiration. Bowel sounds are diminished. The patient is lying on her side holding an emesis basin. Which of the following is causing this patientโ€™s disorder?

A. Alcoholic liver disease
B. Gallstone obstruction in the cystic duct
C. Obstruction from carcinoma of the head of the pancreas
D. Gallstone obstruction in the common bile duct
E. Pancreatic inflammation
๐Ÿ‘11โค4